Veena Singla

5.1k citations
24 papers · 3.9k indexed · 3 hit papers · h-index 15
Topics
Toxic Organic Pollutants Impact (8 papers)Effects and risks of endocrine disrupting chemicals (5 papers)Genetic and Kidney Cyst Diseases (5 papers)

In The Last Decade

Veena Singla

23 papers receiving 3.8k citations

Hit Papers

Vertebrate Smoothened functions at the primary cilium20052026201220192005200620192505007501000

Peers

Veena Singla
Comparison fields: 5 of 155
  • Molecular Biology 2.4k
  • Genetics 2.1k
  • Health, Toxicology and Mutagenesis 630
  • Cell Biology 588
  • Pollution 189
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Veena Singla relative to Nathan Mise Japan Nathan Mise's profile →
Citations per field
00.5×12×
Nathan Mise · 1×
Citations per year

Countries citing papers authored by Veena Singla

Since Specialization
Citations

This map shows the geographic impact of Veena Singla's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Veena Singla with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Veena Singla more than expected).

Fields of papers citing papers by Veena Singla

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Veena Singla. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Veena Singla. The network helps show where Veena Singla may publish in the future.

Co-authorship network of co-authors of Veena Singla

This figure shows the co-authorship network connecting the top 25 collaborators of Veena Singla. A scholar is included among the top collaborators of Veena Singla based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Veena Singla. Veena Singla is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 81
3 1
4 9
5 1
6 14
7 7
8 28
9 12
10 205
11 1
12 36
13 25
14 196
15 33
16 23
17 425
18 97
19
The Primary Cilium as the Cell's Antenna: Signaling at a Sensory Organellebreakdown →
875
20
Vertebrate Smoothened functions at the primary ciliumbreakdown →
1147

About Veena Singla

Veena Singla is a scholar working on Chemical Health and Safety, Health, Toxicology and Mutagenesis and Safety, Risk, Reliability and Quality, having authored 24 papers that have together received 3.9k indexed citations. Recurring topics across this work include Toxic Organic Pollutants Impact (8 papers), Effects and risks of endocrine disrupting chemicals (5 papers) and Genetic and Kidney Cyst Diseases (5 papers). The work is most often cited by research in Genetics (2.1k citations), Health, Toxicology and Mutagenesis (630 citations) and Cell Biology (588 citations). Veena Singla has collaborated with scholars based in United States, Canada and Australia. Frequent co-authors include Jeremy F. Reiter, Kevin C. Corbit, Andrew R. Norman, Didier Y. R. Stainier, Pia Aanstad, Amy E. Shyer, William E. Dowdle, Miriam L. Diamond, Arlene Blum and Allison L. Phillips. Their work appears in journals such as Nature, Science and Environmental Science & Technology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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