William E. Dowdle

2.9k total citations
17 papers, 1.5k citations indexed

About

William E. Dowdle is a scholar working on Molecular Biology, Genetics and Oncology. According to data from OpenAlex, William E. Dowdle has authored 17 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 8 papers in Genetics and 4 papers in Oncology. Recurrent topics in William E. Dowdle's work include Genetic Syndromes and Imprinting (7 papers), Hedgehog Signaling Pathway Studies (6 papers) and Genetic and Kidney Cyst Diseases (5 papers). William E. Dowdle is often cited by papers focused on Genetic Syndromes and Imprinting (7 papers), Hedgehog Signaling Pathway Studies (6 papers) and Genetic and Kidney Cyst Diseases (5 papers). William E. Dowdle collaborates with scholars based in United States, Switzerland and United Kingdom. William E. Dowdle's co-authors include Jeremy F. Reiter, Kevin C. Corbit, Amy E. Shyer, Veena Singla, John Blenis, Sejeong Shin, Sang-Oh Yoon, Leon O. Murphy, Jeffrey P. MacKeigan and Joseph W. Lewcock and has published in prestigious journals such as Nature Communications, Neuron and Blood.

In The Last Decade

William E. Dowdle

17 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
William E. Dowdle United States	12	1.1k	578	252	224	135	17	1.5k
Pumin Zhang United States	20	1.1k 1.0×	259 0.4×	413 1.6×	257 1.1×	98 0.7×	29	1.5k
Vincent W. Keng Hong Kong	27	1.4k 1.3×	395 0.7×	159 0.6×	195 0.9×	173 1.3×	60	2.0k
Jimmy K. Stauffer United States	19	787 0.7×	318 0.6×	224 0.9×	350 1.6×	86 0.6×	31	1.4k
Josephine C. Dorsman Netherlands	23	1.4k 1.3×	393 0.7×	101 0.4×	425 1.9×	54 0.4×	59	2.0k
M. Grazia Cotticelli United States	13	1.6k 1.5×	1.0k 1.7×	113 0.4×	410 1.8×	117 0.9×	19	2.1k
Kathleen H. Holt United States	17	1.5k 1.3×	249 0.4×	284 1.1×	217 1.0×	46 0.3×	18	1.7k
Mónica Álvarez‐Fernández Spain	22	1.4k 1.2×	185 0.3×	537 2.1×	529 2.4×	75 0.6×	30	1.9k
Ann M. Pace United States	6	977 0.9×	174 0.3×	192 0.8×	269 1.2×	167 1.2×	6	1.8k
Marie Vandromme France	28	1.8k 1.6×	279 0.5×	223 0.9×	216 1.0×	40 0.3×	33	2.0k
Ziqiang Yuan United States	23	970 0.9×	140 0.2×	96 0.4×	392 1.8×	137 1.0×	44	1.5k

Countries citing papers authored by William E. Dowdle

Since Specialization

Citations

This map shows the geographic impact of William E. Dowdle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William E. Dowdle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William E. Dowdle more than expected).

Fields of papers citing papers by William E. Dowdle

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William E. Dowdle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William E. Dowdle. The network helps show where William E. Dowdle may publish in the future.

Co-authorship network of co-authors of William E. Dowdle

This figure shows the co-authorship network connecting the top 25 collaborators of William E. Dowdle. A scholar is included among the top collaborators of William E. Dowdle based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William E. Dowdle. William E. Dowdle is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

1.

Hu, Xiaomeng, Kathy White, Eléonore Tham, et al.. (2023). Hypoimmune anti-CD19 chimeric antigen receptor T cells provide lasting tumor control in fully immunocompetent allogeneic humanized mice. Nature Communications. 14(1). 2020–2020. 57 indexed citations

2.

Hu, Xiaomeng, Kathy White, Chi Young, et al.. (2023). Human hypoimmune primary pancreatic islets avoid rejection and autoimmunity and alleviate diabetes in allogeneic humanized mice. Science Translational Medicine. 15(691). eadg5794–eadg5794. 55 indexed citations

3.

Hu, Xiaomeng, Mo A. Dao, Kathy White, et al.. (2021). Engineered Hypoimmune Allogeneic CAR T Cells Exhibit Innate and Adaptive Immune Evasion Even after Sensitization in Humanized Mice and Retain Potent Anti-Tumor Activity. Blood. 138(Supplement 1). 1690–1690. 3 indexed citations

4.

Hu, Xiaomeng, Mo A. Dao, Kathy White, et al.. (2021). Abstract LB144: Overexpression of CD47 protects hypoimmune CAR T cells from innate immune cell killing. Cancer Research. 81(13_Supplement). LB144–LB144. 5 indexed citations

5.

Naguib, Adam, Thomas Sandmann, Fei Yi, et al.. (2019). SUPT4H1 Depletion Leads to a Global Reduction in RNA. Cell Reports. 26(1). 45–53.e4. 13 indexed citations

6.

Fang, Mark Y., Sebastian Markmiller, Anthony Q. Vu, et al.. (2019). Small-Molecule Modulation of TDP-43 Recruitment to Stress Granules Prevents Persistent TDP-43 Accumulation in ALS/FTD. Neuron. 103(5). 802–819.e11. 198 indexed citations

7.

Goodwin, Jonathan M., William E. Dowdle, Zuncai Wang, et al.. (2017). Autophagy-Independent Lysosomal Targeting Regulated by ULK1/2-FIP200 and ATG9. Cell Reports. 20(10). 2341–2356. 140 indexed citations

8.

Honda, Ayako, Edmund Harrington, Iván Cornella‐Taracido, et al.. (2015). Potent, Selective, and Orally Bioavailable Inhibitors of VPS34 Provide Chemical Tools to Modulate Autophagy in Vivo. ACS Medicinal Chemistry Letters. 7(1). 72–76. 40 indexed citations

9.

Saqui–Salces, Milena, William E. Dowdle, Jeremy F. Reiter, & Juanita L. Merchant. (2012). A high‐fat diet regulates gastrin and acid secretion through primary cilia. The FASEB Journal. 26(8). 3127–3139. 45 indexed citations

10.

García-Gonzalo, Francesc R., et al.. (2012). A transition zone complex of ciliopathy proteins regulates ciliary composition. Europe PMC (PubMed Central). 1(S1). 1 indexed citations

11.

Dowdle, William E., Jon F. Robinson, Ma Salomé Sirerol-Piquer, et al.. (2011). Disruption of a Ciliary B9 Protein Complex Causes Meckel Syndrome. The American Journal of Human Genetics. 89(1). 94–110. 109 indexed citations

12.

Dowdle, William E., Jon F. Robinson, Ma Salomé Sirerol-Piquer, et al.. (2011). Disruption of a Ciliary B9 Protein Complex Causes Meckel Syndrome. The American Journal of Human Genetics. 89(4). 589–589. 6 indexed citations

13.

Croyle, Mandy J., Jonathan M. Lehman, Amber K. O’Connor, et al.. (2011). Role of epidermal primary cilia in the homeostasis of skin and hair follicles. Development. 138(9). 1675–1685. 56 indexed citations

14.

Croyle, Mandy J., Jonathan M. Lehman, Amber K. O’Connor, et al.. (2011). Role of epidermal primary cilia in the homeostasis of skin and hair follicles. Journal of Cell Science. 124(9). e1–e1. 5 indexed citations

15.

Shin, Sejeong, et al.. (2010). ERK2 but Not ERK1 Induces Epithelial-to-Mesenchymal Transformation via DEF Motif-Dependent Signaling Events. Molecular Cell. 38(1). 114–127. 243 indexed citations

16.

Corbit, Kevin C., et al.. (2007). Kif3a constrains β-catenin-dependent Wnt signalling through dual ciliary and non-ciliary mechanisms. Nature Cell Biology. 10(1). 70–76. 425 indexed citations

17.

Dowdle, William E., et al.. (2005). Spatially Separate Docking Sites on ERK2 Regulate Distinct Signaling Events In Vivo. Current Biology. 15(14). 1319–1324. 91 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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