Tomasz Cierpicki
- Molecular Biology top 2%
- Hematology top 2%
- Organic Chemistry top 5%
- Oncology top 5%
- Immunology top 10%
- Co-authors
- Jolanta GrembeckaJacek OtlewskiJohn H. BushwellerTrupta PurohitMarcelo J. MuraiDaniel KrowarschPaweł KafarskiShihan He
- Topics
- Protein Degradation and Inhibitors (31 papers)Ubiquitin and proteasome pathways (18 papers)Acute Myeloid Leukemia Research (17 papers)
- Journals
- New England Journal of MedicineProceedings of the National Academy of SciencesJournal of the American Chemical Society
- Partner nations
- United StatesPolandUnited Kingdom
In The Last Decade
Tomasz Cierpicki
110 papers receiving 4.7k citations
Peers
Comparison fields: 5 of 133
- Molecular Biology 3.5k
- Hematology 622
- Organic Chemistry 589
- Oncology 567
- Immunology 399
Countries citing papers authored by Tomasz Cierpicki
This map shows the geographic impact of Tomasz Cierpicki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomasz Cierpicki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomasz Cierpicki more than expected).
Fields of papers citing papers by Tomasz Cierpicki
This network shows the impact of papers produced by Tomasz Cierpicki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomasz Cierpicki. The network helps show where Tomasz Cierpicki may publish in the future.
Co-authorship network of co-authors of Tomasz Cierpicki
This figure shows the co-authorship network connecting the top 25 collaborators of Tomasz Cierpicki. A scholar is included among the top collaborators of Tomasz Cierpicki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomasz Cierpicki. Tomasz Cierpicki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 8 | |
| 3 | 1 | |
| 4 | 4 | |
| 5 | 2 | |
| 6 | 33 | |
| 7 | 10 | |
| 8 | 18 | |
| 9 | 26 | |
| 10 | 36 | |
| 11 | 12 | |
| 12 | 48 | |
| 13 | 158 | |
| 14 | 42 | |
| 15 | 2 | |
| 16 | 152 | |
| 17 | 45 | |
| 18 | 24 | |
| 19 | 145 | |
| 20 | 108 |
About Tomasz Cierpicki
Tomasz Cierpicki is a scholar working on Hematology, Molecular Biology and Cell Biology, having authored 113 papers that have together received 4.7k indexed citations. Recurring topics across this work include Protein Degradation and Inhibitors (31 papers), Ubiquitin and proteasome pathways (18 papers) and Acute Myeloid Leukemia Research (17 papers). The work is most often cited by research in Hematology (622 citations), Molecular Biology (3.5k citations) and Cell Biology (390 citations). Tomasz Cierpicki has collaborated with scholars based in United States, Poland and United Kingdom. Frequent co-authors include Jolanta Grembecka, Jacek Otlewski, John H. Bushweller, Trupta Purohit, Marcelo J. Murai, Daniel Krowarsch, Paweł Kafarski, Shihan He, Artur Mucha and Filip Jeleń. Their work appears in journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of the American Chemical Society.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.