Tom Verfaillie

3.8k total citations · 2 hit papers
16 papers, 3.1k citations indexed

About

Tom Verfaillie is a scholar working on Epidemiology, Cell Biology and Molecular Biology. According to data from OpenAlex, Tom Verfaillie has authored 16 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Epidemiology, 10 papers in Cell Biology and 9 papers in Molecular Biology. Recurrent topics in Tom Verfaillie's work include Autophagy in Disease and Therapy (11 papers), Endoplasmic Reticulum Stress and Disease (9 papers) and Heme Oxygenase-1 and Carbon Monoxide (3 papers). Tom Verfaillie is often cited by papers focused on Autophagy in Disease and Therapy (11 papers), Endoplasmic Reticulum Stress and Disease (9 papers) and Heme Oxygenase-1 and Carbon Monoxide (3 papers). Tom Verfaillie collaborates with scholars based in Belgium, Ireland and United States. Tom Verfaillie's co-authors include Patrizia Agostinis, Abhishek D. Garg, Noemí Rubio, Alexander R. van Vliet, Jacques Piette, Peter Vandenabeele, Dmitri V. Krysko, Afshin Samali, Chantal Mathieu and Gabriela B. Ferreira and has published in prestigious journals such as The EMBO Journal, Molecular Cell and Biochemical and Biophysical Research Communications.

In The Last Decade

Tom Verfaillie

16 papers receiving 3.1k citations

Hit Papers

PERK is required at the ER-mitochondrial contact sites to... 2012 2026 2016 2021 2012 2012 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tom Verfaillie Belgium 14 1.5k 1.2k 1.0k 675 450 16 3.1k
Yu Jiang United States 45 3.6k 2.4× 1.0k 0.9× 702 0.7× 430 0.6× 184 0.4× 119 5.1k
Katherine L. Cook United States 30 1.3k 0.9× 543 0.5× 606 0.6× 281 0.4× 110 0.2× 83 2.5k
Penny E. Lovat United Kingdom 34 1.7k 1.1× 708 0.6× 740 0.7× 399 0.6× 68 0.2× 88 2.9k
Hee Gu Lee South Korea 37 2.0k 1.3× 317 0.3× 454 0.4× 854 1.3× 184 0.4× 140 3.8k
Paul Chang United States 25 1.6k 1.0× 521 0.4× 208 0.2× 536 0.8× 376 0.8× 72 3.3k
Angel L. Armesilla United Kingdom 30 1.6k 1.1× 315 0.3× 366 0.4× 286 0.4× 188 0.4× 50 2.8k
Mauricio J. Reginato United States 35 4.2k 2.7× 734 0.6× 647 0.6× 1.0k 1.5× 283 0.6× 63 5.7k
Florian J. Bock Austria 16 1.8k 1.2× 208 0.2× 359 0.3× 478 0.7× 168 0.4× 21 2.7k
Zhengzhi Zou China 29 2.0k 1.3× 260 0.2× 372 0.4× 749 1.1× 349 0.8× 53 3.5k
Mojgan Djavaheri‐Mergny France 28 1.8k 1.2× 396 0.3× 1.4k 1.4× 345 0.5× 63 0.1× 59 3.3k

Countries citing papers authored by Tom Verfaillie

Since Specialization
Citations

This map shows the geographic impact of Tom Verfaillie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tom Verfaillie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tom Verfaillie more than expected).

Fields of papers citing papers by Tom Verfaillie

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tom Verfaillie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tom Verfaillie. The network helps show where Tom Verfaillie may publish in the future.

Co-authorship network of co-authors of Tom Verfaillie

This figure shows the co-authorship network connecting the top 25 collaborators of Tom Verfaillie. A scholar is included among the top collaborators of Tom Verfaillie based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tom Verfaillie. Tom Verfaillie is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Vliet, Alexander R. van, Francesca Giordano, Sarah Gerlo, et al.. (2017). The ER Stress Sensor PERK Coordinates ER-Plasma Membrane Contact Site Formation through Interaction with Filamin-A and F-Actin Remodeling. Molecular Cell. 65(5). 885–899.e6. 167 indexed citations
2.
Eygen, Sofie Van, Dmitri V. Krysko, Peter Vandenabeele, et al.. (2014). BNIP3 supports melanoma cell migration and vasculogenic mimicry by orchestrating the actin cytoskeleton. Cell Death and Disease. 5(3). e1127–e1127. 108 indexed citations
3.
Vliet, Alexander R. van, Tom Verfaillie, & Patrizia Agostinis. (2014). New functions of mitochondria associated membranes in cellular signaling. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1843(10). 2253–2262. 310 indexed citations
5.
Garg, Abhishek D., Aleksandra M. Dudek, Gabriela B. Ferreira, et al.. (2013). ROS-induced autophagy in cancer cells assists in evasion from determinants of immunogenic cell death. Autophagy. 9(9). 1292–1307. 249 indexed citations
6.
Verfaillie, Tom, Alexander R. van Vliet, Abhishek D. Garg, et al.. (2013). Pro-apoptotic signaling induced by photo-oxidative ER stress is amplified by Noxa, not Bim. Biochemical and Biophysical Research Communications. 438(3). 500–506. 40 indexed citations
7.
Maes, Hannelore, Miguel A. Martín-Acebes, Tom Verfaillie, & Patrizia Agostinis. (2013). Dynamic interplay between autophagic flux and Akt during melanoma progression in vitro. Experimental Dermatology. 23(2). 101–106. 21 indexed citations
8.
Verfaillie, Tom, Noemí Rubio, Abhishek D. Garg, et al.. (2012). PERK IS REQUIRED AT THE ER-TO-MITOCHONDRIA CONTACT SITES TO CONVEY APOPTOSIS FOLLOWING ROS-MEDIATED ER STRESS. Open Repository and Bibliography (University of Liège). 1 indexed citations
9.
Verfaillie, Tom, Noemí Rubio, Abhishek D. Garg, et al.. (2012). PERK is required at the ER-mitochondrial contact sites to convey apoptosis after ROS-based ER stress. Cell Death and Differentiation. 19(11). 1880–1891. 693 indexed citations breakdown →
10.
Garg, Abhishek D., Dmitri V. Krysko, Tom Verfaillie, et al.. (2012). A novel pathway combining calreticulin exposure and ATP secretion in immunogenic cancer cell death. The EMBO Journal. 31(5). 1062–1079. 651 indexed citations breakdown →
11.
Gupta, Sanjeev, Danielle E. Read, Ayswaria Deepti, et al.. (2012). Perk-dependent repression of miR-106b-25 cluster is required for ER stress-induced apoptosis. Cell Death and Disease. 3(6). e333–e333. 82 indexed citations
12.
Dewaele, Michael, Tom Verfaillie, Wim Martinet, & Patrizia Agostinis. (2010). Death and Survival Signals in Photodynamic Therapy. Methods in molecular biology. 635. 7–33. 19 indexed citations
13.
Verfaillie, Tom, Abhishek D. Garg, & Patrizia Agostinis. (2010). Targeting ER stress induced apoptosis and inflammation in cancer. Cancer Letters. 332(2). 249–264. 338 indexed citations
14.
Dewaele, Michael, Wim Martinet, Noemí Rubio, et al.. (2010). Autophagy pathways activated in response to PDT contribute to cell resistance against ROS damage. Journal of Cellular and Molecular Medicine. 15(6). 1402–1414. 109 indexed citations
15.
Garg, Abhishek D., et al.. (2010). Hypericin-PDT treatment of cancer cells leads to surface exposure/extracellular release of DAMPs and activates human immature dendritic cells. 4(2). 93–94. 3 indexed citations
16.
Verfaillie, Tom, María Salazar‐Roa, Guillermo Velasco, & Patrizia Agostinis. (2010). Linking ER Stress to Autophagy: Potential Implications for Cancer Therapy. International Journal of Cell Biology. 2010. 1–19. 293 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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