Dariusz Pytel

3.1k total citations · 1 hit paper
50 papers, 2.3k citations indexed

About

Dariusz Pytel is a scholar working on Molecular Biology, Cell Biology and Epidemiology. According to data from OpenAlex, Dariusz Pytel has authored 50 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Molecular Biology, 23 papers in Cell Biology and 11 papers in Epidemiology. Recurrent topics in Dariusz Pytel's work include Endoplasmic Reticulum Stress and Disease (23 papers), Autophagy in Disease and Therapy (11 papers) and DNA Repair Mechanisms (7 papers). Dariusz Pytel is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (23 papers), Autophagy in Disease and Therapy (11 papers) and DNA Repair Mechanisms (7 papers). Dariusz Pytel collaborates with scholars based in United States, Poland and France. Dariusz Pytel's co-authors include J. Alan Diehl, Ireneusz Majsterek, Wioletta Rozpędek‐Kamińska, Bartosz Mucha, Ekaterina Bobrovnikova-Marjon, Adam Wawrzynkiewicz, Natalia Siwecka, Constantinos Koumenis, Tomasz Śliwiński and Tomasz Popławski and has published in prestigious journals such as Molecular Cell, Molecular and Cellular Biology and Cancer Research.

In The Last Decade

Dariusz Pytel

50 papers receiving 2.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The Role of the PERK/eIF2α/ATF4/CHOP Signaling Pathway in Tumor Progression During Endoplasmic Reticulum Stress

2016 645 citations Wioletta Rozpędek‐Kamińska, Dariusz Pytel et al. Current Molecular Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Dariusz Pytel United States	22	1.2k	1.1k	679	240	223	50	2.3k
David G. Breckenridge United States	21	1.7k 1.4×	654 0.6×	682 1.0×	252 1.1×	148 0.7×	40	2.5k
Che Xu China	10	1.3k 1.1×	1.1k 1.0×	693 1.0×	449 1.9×	366 1.6×	14	2.7k
Fawzia Bardag‐Gorce United States	31	1.5k 1.2×	745 0.7×	734 1.1×	151 0.6×	227 1.0×	93	2.6k
Renata Sano United States	11	1.2k 1.0×	1.2k 1.0×	755 1.1×	194 0.8×	97 0.4×	14	2.4k
Karen D. McCullough United States	8	1.3k 1.1×	1.3k 1.1×	678 1.0×	237 1.0×	191 0.9×	11	2.4k
Sandra Healy Ireland	12	974 0.8×	890 0.8×	525 0.8×	146 0.6×	110 0.5×	20	1.8k
Shotaro Nakajima Japan	25	783 0.7×	593 0.5×	455 0.7×	542 2.3×	213 1.0×	72	2.0k
Shun-ichiro Iemura Japan	6	2.0k 1.6×	376 0.3×	887 1.3×	186 0.8×	238 1.1×	7	2.6k
Norihiko Furuya Japan	18	1.7k 1.4×	762 0.7×	2.5k 3.7×	179 0.7×	372 1.7×	27	3.5k
Olivier Geneste France	22	1.7k 1.4×	372 0.3×	894 1.3×	178 0.7×	435 2.0×	35	2.7k

Countries citing papers authored by Dariusz Pytel

Since Specialization

Citations

This map shows the geographic impact of Dariusz Pytel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dariusz Pytel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dariusz Pytel more than expected).

Fields of papers citing papers by Dariusz Pytel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dariusz Pytel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dariusz Pytel. The network helps show where Dariusz Pytel may publish in the future.

Co-authorship network of co-authors of Dariusz Pytel

This figure shows the co-authorship network connecting the top 25 collaborators of Dariusz Pytel. A scholar is included among the top collaborators of Dariusz Pytel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dariusz Pytel. Dariusz Pytel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pytel, Dariusz & Jody Fromm Longo. (2025). The Proteostasis Network in Proteinopathies. American Journal Of Pathology. 195(11). 1998–2014. 1 indexed citations

Rozpędek‐Kamińska, Wioletta, et al.. (2024). Evaluation of the LDN-0060609 PERK Inhibitor as a Selective Treatment for Primary Open-Angle Glaucoma: An In Vitro Study on Human Retinal Astrocytes. International Journal of Molecular Sciences. 25(2). 728–728. 1 indexed citations

Anderson, Mark, Vera Holzmayer, Barbara J. Harris, et al.. (2023). The diversification of SARS-CoV-2 Omicron variants and evaluation of their detection with molecular and rapid antigen assays. Journal of Clinical Virology. 166. 105532–105532. 1 indexed citations

Siwecka, Natalia, Wioletta Rozpędek‐Kamińska, Adam Wawrzynkiewicz, et al.. (2021). The Structure, Activation and Signaling of IRE1 and Its Role in Determining Cell Fate. Biomedicines. 9(2). 156–156. 117 indexed citations

Rozpędek‐Kamińska, Wioletta, Grzegorz Galita, Natalia Siwecka, et al.. (2021). The Potential Role of Small-Molecule PERK Inhibitor LDN-0060609 in Primary Open-Angle Glaucoma Treatment. International Journal of Molecular Sciences. 22(9). 4494–4494. 9 indexed citations

Rozpędek‐Kamińska, Wioletta, Natalia Siwecka, Adam Wawrzynkiewicz, et al.. (2020). The PERK-Dependent Molecular Mechanisms as a Novel Therapeutic Target for Neurodegenerative Diseases. International Journal of Molecular Sciences. 21(6). 2108–2108. 61 indexed citations

Popławski, Tomasz, Dariusz Pytel, Jarosław Dziadek, & Ireneusz Majsterek. (2019). Interplay between Redox Signaling, Oxidative Stress, and Unfolded Protein Response (UPR) in Pathogenesis of Human Diseases. Oxidative Medicine and Cellular Longevity. 2019. 1–2. 14 indexed citations

Siwecka, Natalia, Wioletta Rozpędek‐Kamińska, Dariusz Pytel, et al.. (2019). Dual role of Endoplasmic Reticulum Stress-Mediated Unfolded Protein Response Signaling Pathway in Carcinogenesis. International Journal of Molecular Sciences. 20(18). 4354–4354. 119 indexed citations

Rozpędek‐Kamińska, Wioletta, et al.. (2019). Potencjalne zastosowanie terapeutyczne inhibitorów PERK. Postępy Biochemii. 65(2). 118–127. 2 indexed citations

10.

Kowalczyk, Tomasz, Przemysław Sitarek, Ewa Skała, et al.. (2019). Induction of apoptosis by in vitro and in vivo plant extracts derived from Menyanthes trifoliata L. in human cancer cells. Cytotechnology. 71(1). 165–180. 41 indexed citations

11.

Sitarek, Przemysław, Patrícia Rijo, Catarina García, et al.. (2017). Antibacterial, Anti‐Inflammatory, Antioxidant, and Antiproliferative Properties of Essential Oils from Hairy and Normal Roots of Leonurus sibiricus L. and Their Chemical Composition. Oxidative Medicine and Cellular Longevity. 2017(1). 7384061–7384061. 74 indexed citations

12.

Rozpędek‐Kamińska, Wioletta, et al.. (2017). Molecular Basis of Human Diseases and Targeted Therapy Based on Small-Molecule Inhibitors of ER Stress-Induced Signaling Pathways. Current Molecular Medicine. 17(2). 118–132. 20 indexed citations

13.

Skała, Ewa, Patrícia Rijo, Catarina García, et al.. (2016). The Essential Oils of Rhaponticum carthamoides Hairy Roots and Roots of Soil‐Grown Plants: Chemical Composition and Antimicrobial, Anti‐Inflammatory, and Antioxidant Activities. Oxidative Medicine and Cellular Longevity. 2016(1). 8505384–8505384. 17 indexed citations

14.

Pytel, Dariusz, Yan Gao, Yuliya V. Katlinskaya, et al.. (2016). PERK Is a Haploinsufficient Tumor Suppressor: Gene Dose Determines Tumor-Suppressive Versus Tumor Promoting Properties of PERK in Melanoma. PLoS Genetics. 12(12). e1006518–e1006518. 39 indexed citations

15.

Markiewicz, Łukasz, Dariusz Pytel, Bartosz Mucha, et al.. (2015). Altered Expression Levels of MMP1, MMP9, MMP12, TIMP1, and IL-1βas a Risk Factor for the Elevated IOP and Optic Nerve Head Damage in the Primary Open-Angle Glaucoma Patients. BioMed Research International. 2015. 1–8. 43 indexed citations

16.

Chitnis, Nilesh, Dariusz Pytel, Ekaterina Bobrovnikova-Marjon, et al.. (2012). miR-211 Is a Prosurvival MicroRNA that Regulates chop Expression in a PERK-Dependent Manner. Molecular Cell. 48(3). 353–364. 177 indexed citations

17.

Słupianek, Artur, Rafal Falinski, Paweł Znojek, et al.. (2012). BCR-ABL1 kinase inhibits uracil DNA glycosylase UNG2 to enhance oxidative DNA damage and stimulate genomic instability. Leukemia. 27(3). 629–634. 31 indexed citations

18.

Słupianek, Artur, Tomasz Popławski, Stanisław K. Jóźwiakowski, et al.. (2010). BCR/ABL Stimulates WRN to Promote Survival and Genomic Instability. Cancer Research. 71(3). 842–851. 39 indexed citations

19.

Bobrovnikova-Marjon, Ekaterina, Christina Grigoriadou, Dariusz Pytel, et al.. (2010). PERK promotes cancer cell proliferation and tumor growth by limiting oxidative DNA damage. Oncogene. 29(27). 3881–3895. 241 indexed citations

20.

Pytel, Dariusz, et al.. (2006). Comparative study of DNA damage, cell cycle and apoptosis in human K562 and CCRF-CEM leukemia cells: Role of BCR/ABL in therapeutic resistance. Comparative Biochemistry and Physiology Part C Toxicology & Pharmacology. 144(1). 85–92. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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