Terje Johansen
Impact in
- Physiology top 0.02%
- Calcium signaling and nucleotide metabolism
- Epidemiology top 0.01%
- Autophagy in Disease and Therapy
Papers in
-
- Protein Kinase Regulation and GTPase Signaling 16
- Ubiquitin and proteasome pathways 16
- Epidemiology 100
- Autophagy in Disease and Therapy 100
- Co-authors
- Trond Lamark (58 shared papers)Aud Øvervatn (18 shared papers)Geir Bjørkøy (21 shared papers)Andreas Brech (15 shared papers)Heidi Outzen (4 shared papers)Jack‐Ansgar Bruun (16 shared papers)Serhiy Pankiv (7 shared papers)Vladimir Kirkin (8 shared papers)
- Journals
- Autophagy (24 papers)Journal of Biological Chemistry (23 papers)The Journal of Cell Biology (10 papers)The EMBO Journal (7 papers)Journal of Cell Science (6 papers)
- Partner nations
- NorwayUnited StatesUnited Kingdom
In The Last Decade
Terje Johansen
168 papers receiving 30.0k citations
Terje Johansen's Hit Papers
Peers
Comparison fields: 5 of 160
- Physiology 2.7k
- Epidemiology 18.4k
- Cell Biology 6.8k
- Geriatrics and Gerontology 1.0k
- Parasitology 1.5k
Countries citing papers authored by Terje Johansen
This map shows the geographic impact of Terje Johansen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Terje Johansen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Terje Johansen more than expected).
Fields of papers citing papers by Terje Johansen
This network shows the impact of papers produced by Terje Johansen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Terje Johansen. The network helps show where Terje Johansen may publish in the future.
Co-authors
The 25 scholars most cited alongside Terje Johansen, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 169 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | p62/SQSTM1 Binds Directly to Atg8/LC3 to Facilitate Degradation of Ubiquitinated Protein Aggregates by Autophagy Hit paper breakdown → | 2007 | 3704 |
| 2 | p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death Hit paper breakdown → | 2005 | 2693 |
| 3 | Selective autophagy mediated by autophagic adapter proteins Hit paper breakdown → | 2011 | 1373 |
| 4 | p62/SQSTM1 Is a Target Gene for Transcription Factor NRF2 and Creates a Positive Feedback Loop by Inducing Antioxidant Response Element-driven Gene Transcription Hit paper breakdown → | 2010 | 1185 |
| 5 | Chapter 12 Monitoring Autophagic Degradation of p62/SQSTM1 Hit paper breakdown → | 2009 | 958 |
| 6 | A Role for NBR1 in Autophagosomal Degradation of Ubiquitinated Substrates Hit paper breakdown → | 2009 | 908 |
| 7 | Autophagy in healthy aging and disease Hit paper breakdown → | 2021 | 798 |
| 8 | Interactions between Autophagy Receptors and Ubiquitin-like Proteins Form the Molecular Basis for Selective Autophagy Hit paper breakdown → | 2014 | 794 |
| 9 | The LIR motif – crucial for selective autophagy Hit paper breakdown → | 2013 | 666 |
| 10 | Regulation of selective autophagy: the p62/SQSTM1 paradigm Hit paper breakdown → | 2017 | 561 |
| 11 | TBK-1 Promotes Autophagy-Mediated Antimicrobial Defense by Controlling Autophagosome Maturation Hit paper breakdown → | 2012 | 502 |
| 12 | FYCO1 is a Rab7 effector that binds to LC3 and PI3P to mediate microtubule plus end–directed vesicle transport Hit paper breakdown → | 2010 | 500 |
| 13 | Autophagy mediates degradation of nuclear lamina Hit paper breakdown → | 2015 | 484 |
| 14 | Selective Autophagy: ATG8 Family Proteins, LIR Motifs and Cargo Receptors Hit paper breakdown → | 2019 | 482 |
| 15 | 2009 | 450 | |
| 16 | 1991 | 400 | |
| 17 | 2012 | 373 | |
| 18 | 2009 | 367 | |
| 19 | Microenvironmental autophagy promotes tumour growth Hit paper breakdown → | 2017 | 363 |
| 20 | SIRT1 is downregulated by autophagy in senescence and ageing Hit paper breakdown → | 2020 | 357 |
About Terje Johansen
Terje Johansen is a scholar working on Molecular Biology, Epidemiology, Cell Biology, Oncology and Immunology, having authored 169 papers that have together received 30.2k indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (100 papers), Endoplasmic Reticulum Stress and Disease (35 papers), Cellular transport and secretion (20 papers), Protein Kinase Regulation and GTPase Signaling (16 papers), Ubiquitin and proteasome pathways (16 papers), Toxoplasma gondii Research Studies (13 papers), Mosquito-borne diseases and control (12 papers) and Calcium signaling and nucleotide metabolism (11 papers). The work is most often cited by research in Physiology (2.7k citations), Epidemiology (18.4k citations), Cell Biology (6.8k citations), Geriatrics and Gerontology (1.0k citations) and Parasitology (1.5k citations). Terje Johansen has collaborated with scholars based in Norway, United States and United Kingdom. Frequent co-authors include Trond Lamark, Aud Øvervatn, Geir Bjørkøy, Andreas Brech, Heidi Outzen, Jack‐Ansgar Bruun, Serhiy Pankiv, Vladimir Kirkin, Harald Stenmark and Maria Perander. Their work appears in journals such as Autophagy, Journal of Biological Chemistry, The Journal of Cell Biology, The EMBO Journal and Journal of Cell Science.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.