Tohru Kataoka
- Aging top 1%
- Cell Biology top 0.5%
- Cellular transport and secretion 10
- Molecular Biology top 0.5%
- Protein Kinase Regulation and GTPase Signaling 68
- Fungal and yeast genetics research 28
- Melanoma and MAPK Pathways 11
- PI3K/AKT/mTOR signaling in cancer 10
- Cell death mechanisms and regulation 10
- Immunology and Allergy top 2%
- Immunology top 5%
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- Enzyme Structure and Function 13
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- Monoclonal and Polyclonal Antibodies Research 9
- Co-authors
- Michael WiglerScott PowersJames R. BroachDaniel BroekTakaya SatohTasuku HonjoOttavio FasanoKen‐ichi Kariya
- Cited by
- AgingCell BiologyMolecular Biology
- Partner nations
- JapanUnited StatesEgypt
In The Last Decade
Tohru Kataoka
121 papers receiving 7.9k citations
Hit Papers
Peers
Comparison fields: 5 of 122
- Aging 232
- Cell Biology 1.7k
- Molecular Biology 6.7k
- Immunology and Allergy 308
- Immunology 763
Countries citing papers authored by Tohru Kataoka
This map shows the geographic impact of Tohru Kataoka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tohru Kataoka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tohru Kataoka more than expected).
Fields of papers citing papers by Tohru Kataoka
This network shows the impact of papers produced by Tohru Kataoka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tohru Kataoka. The network helps show where Tohru Kataoka may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Tohru Kataoka, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2021 | 5 | |
| 3 | 2017 | 2 | |
| 4 | 2016 | 10 | |
| 5 | 2011 | 85 | |
| 6 | 2009 | 32 | |
| 7 | 2008 | 73 | |
| 8 | 2007 | 43 | |
| 9 | 2006 | 25 | |
| 10 | Direct activation of fission yeast adenylyl cyclase by heterotrimeric G protein gpa2. | 2004 | 7 |
| 11 | 2004 | 46 | |
| 12 | 2004 | 39 | |
| 13 | 2004 | 21 | |
| 14 | 2003 | 55 | |
| 15 | 2002 | 29 | |
| 16 | 1999 | 73 | |
| 17 | 1997 | 48 | |
| 18 | 1996 | 60 | |
| 19 | 1992 | 2 | |
| 20 | 1990 | 212 |
About Tohru Kataoka
Tohru Kataoka is a scholar working on Aging, Molecular Biology and Immunology and Allergy, having authored 122 papers that have together received 8.2k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (68 papers), Fungal and yeast genetics research (28 papers), Enzyme Structure and Function (13 papers), Melanoma and MAPK Pathways (11 papers), PI3K/AKT/mTOR signaling in cancer (10 papers), Cell death mechanisms and regulation (10 papers), Cellular transport and secretion (10 papers) and Monoclonal and Polyclonal Antibodies Research (9 papers). The work is most often cited by research in Aging (232 citations), Cell Biology (1.7k citations) and Molecular Biology (6.7k citations). Tohru Kataoka has collaborated with scholars based in Japan, United States and Egypt. Frequent co-authors include Michael Wigler, Scott Powers, James R. Broach, Daniel Broek, Takaya Satoh, Tasuku Honjo, Ottavio Fasano, Ken‐ichi Kariya, Scott J. Cameron and Yuriko Yamawaki‐Kataoka. Their work appears in journals such as Science, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.