Atsu Aiba
- Molecular Biology top 2%
- Cellular and Molecular Neuroscience top 0.5%
- Cognitive Neuroscience top 2%
- Neurology top 1%
- Cell Biology top 2%
- Co-authors
- Masanobu KanoKouichi HashimotoKazuki NakaoMasahiko WatanabeMotoya KatsukiTakashi MaejimaTakayuki YoshidaYoshiro Inoue
- Topics
- Neuroscience and Neuropharmacology Research (38 papers)Neurogenesis and neuroplasticity mechanisms (16 papers)Neuroinflammation and Neurodegeneration Mechanisms (14 papers)
- Partner nations
- JapanUnited StatesUnited Kingdom
In The Last Decade
Atsu Aiba
124 papers receiving 6.1k citations
Hit Papers
Peers
Comparison fields: 5 of 126
- Molecular Biology 3.3k
- Cellular and Molecular Neuroscience 3.1k
- Cognitive Neuroscience 894
- Neurology 843
- Cell Biology 741
Countries citing papers authored by Atsu Aiba
This map shows the geographic impact of Atsu Aiba's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Atsu Aiba with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Atsu Aiba more than expected).
Fields of papers citing papers by Atsu Aiba
This network shows the impact of papers produced by Atsu Aiba. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Atsu Aiba. The network helps show where Atsu Aiba may publish in the future.
Co-authorship network of co-authors of Atsu Aiba
This figure shows the co-authorship network connecting the top 25 collaborators of Atsu Aiba. A scholar is included among the top collaborators of Atsu Aiba based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Atsu Aiba. Atsu Aiba is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 2 | |
| 4 | 4 | |
| 5 | 4 | |
| 6 | 41 | |
| 7 | 4 | |
| 8 | 28 | |
| 9 | 42 | |
| 10 | 47 | |
| 11 | 8 | |
| 12 | 8 | |
| 13 | 4 | |
| 14 | 41 | |
| 15 | 59 | |
| 16 | 14 | |
| 17 | 40 | |
| 18 | ADDITIVE CONTROL BY METABOTROPIC GLUTAMATE RECEPTORS 1 AND 5 IN BLADDER AFFERENT MECHANOSENSORY TRANSMISSION | 2 |
| 19 | 44 | |
| 20 | 259 |
About Atsu Aiba
Atsu Aiba is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Neurology, having authored 126 papers that have together received 6.2k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (38 papers), Neurogenesis and neuroplasticity mechanisms (16 papers) and Neuroinflammation and Neurodegeneration Mechanisms (14 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (3.1k citations), Developmental Neuroscience (568 citations) and Neurology (843 citations). Atsu Aiba has collaborated with scholars based in Japan, United States and United Kingdom. Frequent co-authors include Masanobu Kano, Kouichi Hashimoto, Kazuki Nakao, Masahiko Watanabe, Motoya Katsuki, Takashi Maejima, Takayuki Yoshida, Yoshiro Inoue, Susumu Tonegawa and Hidetoshi Kassai. Their work appears in journals such as Science, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.