Thomas Y. Shih

5.9k total citations · 1 hit paper
61 papers, 5.1k citations indexed

About

Thomas Y. Shih is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Thomas Y. Shih has authored 61 papers receiving a total of 5.1k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Molecular Biology, 27 papers in Oncology and 27 papers in Genetics. Recurrent topics in Thomas Y. Shih's work include Virus-based gene therapy research (27 papers), RNA Interference and Gene Delivery (17 papers) and Cancer-related Molecular Pathways (16 papers). Thomas Y. Shih is often cited by papers focused on Virus-based gene therapy research (27 papers), RNA Interference and Gene Delivery (17 papers) and Cancer-related Molecular Pathways (16 papers). Thomas Y. Shih collaborates with scholars based in United States, Japan and Israel. Thomas Y. Shih's co-authors include Edward M. Scolnick, Howard A. Young, Maureen O. Weeks, Ronald Ellis, Douglas R. Lowy, Alex G. Papageorge, Gerald D. Fasman, Linda S. Ulsh, Deborah DeFeo and James Bonner and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Thomas Y. Shih

61 papers receiving 4.2k citations

Hit Papers

The p21 src genes of Harvey and Kirsten sarcoma viruses o... 1981 2026 1996 2011 1981 200 400 600

Peers

Thomas Y. Shih
Walter Eckhart United States
Mark O. J. Olson United States
Lawrence A. Chasin United States
Roberto Weinmann United States
Daniel J. Donoghue United States
Marianne Oskarsson United States
Klaus Bister Austria
H. Busch United States
Walter Eckhart United States
Thomas Y. Shih
Citations per year, relative to Thomas Y. Shih Thomas Y. Shih (= 1×) peers Walter Eckhart

Countries citing papers authored by Thomas Y. Shih

Since Specialization
Citations

This map shows the geographic impact of Thomas Y. Shih's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Y. Shih with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Y. Shih more than expected).

Fields of papers citing papers by Thomas Y. Shih

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Y. Shih. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Y. Shih. The network helps show where Thomas Y. Shih may publish in the future.

Co-authorship network of co-authors of Thomas Y. Shih

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Y. Shih. A scholar is included among the top collaborators of Thomas Y. Shih based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Y. Shih. Thomas Y. Shih is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ogiso, Yoshifumi, et al.. (1993). Resistance of NIH3T3 Cells to v-fes Transformation Induced by a Dominant Negative H-ras Mutant. Experimental Cell Research. 208(2). 415–421. 8 indexed citations
2.
Ogiso, Yoshifumi, Yu‐Wen Hwang, Thomas Y. Shih, & Noboru Kuzumaki. (1993). Biological activity of a K-ras mutant that contains the 12R/59T/116Y mutations. Cancer Letters. 75(1). 19–26. 3 indexed citations
3.
Boylan, John F., Thomas Y. Shih, Paul B. Fisher, & Stephen G. Zimmer. (1992). Induction and progression of the transformed phenotype in cloned rat embryo fibroblast cells: Studies employing type 5 adenovirus and wild‐type and mutant Ha‐ras oncogenes. Molecular Carcinogenesis. 5(2). 118–128. 8 indexed citations
4.
Lu, Youyong, Donald G. Blair, Shraga Segal, Thomas Y. Shih, & David J. Clanton. (1991). Tumorigenicity, metastasis and suppression of MHC class-I expression in murine fibroblasts transformed by mutantv-ras deficient in GTP binding. International Journal of Cancer. 47(S6). 45–53. 11 indexed citations
5.
Hattori, Seisuke, David J. Clanton, Takaya Satoh, et al.. (1987). Neutralizing Monoclonal Antibody Against Ras Oncogene Product p21 Which Impairs Guanine Nucleotide Exchange. Molecular and Cellular Biology. 7(5). 1999–2002. 17 indexed citations
6.
Hoshino, Masato, David J. Clanton, Thomas Y. Shih, Masao Kawakita, & Seisuke Hattori. (1987). Interaction of ras Oncogene Product p21 with Guanine Nucleotides1. The Journal of Biochemistry. 102(3). 503–511. 30 indexed citations
7.
Clanton, David J., Youyong Lu, Donald G. Blair, & Thomas Y. Shih. (1987). Structural significance of the GTP-binding domain of ras p21 studied by site-directed mutagenesis.. Molecular and Cellular Biology. 7(9). 3092–3097. 48 indexed citations
8.
Darfler, Frederick J., Thomas Y. Shih, & Michael C. Lin. (1986). Revertants of Ha-MuSV-transformed MDCK cells express reduced levels of p21 and possess a more normal phenotype. Experimental Cell Research. 162(2). 335–346. 11 indexed citations
9.
Beckner, Suzanne K., Seisuke Hattori, & Thomas Y. Shih. (1985). The ras oncogene product p21 is not a regulatory component of adenylate cyclase. Nature. 317(6032). 71–72. 179 indexed citations
10.
Ulsh, Linda S. & Thomas Y. Shih. (1984). Metabolic turnover of human c-rasH p21 protein of EJ bladder carcinoma and its normal cellular and viral homologs.. Molecular and Cellular Biology. 4(8). 1647–1652. 52 indexed citations
11.
Dhar, Ravi, Ronald Ellis, Thomas Y. Shih, et al.. (1982). Nucleotide Sequence of the p21 Transforming Protein of Harvey Murine Sarcoma Virus. Science. 217(4563). 934–936. 244 indexed citations
12.
Shih, Thomas Y., Maureen O. Weeks, Howard A. Young, & Edward M. Scolnick. (1979). Identification of a sarcoma virus-coded phosphoprotein in nonproducer cells transformed by Kirsten or Harvey murine sarcoma virus. Virology. 96(1). 64–79. 234 indexed citations
13.
Scolnick, Edward M., Alex G. Papageorge, & Thomas Y. Shih. (1979). Guanine nucleotide-binding activity as an assay for src protein of rat-derived murine sarcoma viruses. Proceedings of the National Academy of Sciences. 76(10). 5355–5359. 255 indexed citations
14.
Shih, Thomas Y., D. R. Williams, Maureen O. Weeks, et al.. (1978). Comparison of the genomic organization of Kirsten and Harvey sarcoma viruses. Journal of Virology. 27(1). 45–55. 87 indexed citations
15.
Chien, Yueh‐hsiu, Inder M. Verma, Thomas Y. Shih, Edward M. Scolnick, & Norman Davidson. (1978). Heteroduplex analysis of the sequence relations between the RNAs of mink cell focus-inducing and murine leukemia viruses. Journal of Virology. 28(1). 352–360. 76 indexed citations
16.
Young, Howard A., Thomas Y. Shih, E M Scolnick, & Wade P. Parks. (1977). Steroid induction of mouse mammary tumor virus: effect upon synthesis and degradation of viral RNA. Journal of Virology. 21(1). 139–146. 109 indexed citations
17.
Shih, Thomas Y. & Malcolm A. Martin. (1974). Chemical linkage of nucleic acids to neutral and phosphorylated cellulose powders and isolation of specific sequences by affinity chromatography. Biochemistry. 13(16). 3411–3418. 34 indexed citations
18.
Shih, Thomas Y. & Malcolm A. Martin. (1973). A General Method of Gene Isolation. Proceedings of the National Academy of Sciences. 70(6). 1697–1700. 17 indexed citations
19.
20.
Shih, Thomas Y. & Robert S. Lake. (1972). Studies on the structure of metaphase and interphase chromatin of Chinese hamster cells by circular dichroism and thermal denaturation. Biochemistry. 11(25). 4811–4817. 45 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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