Tetsuya Nosaka

9.1k total citations · 4 hit papers
120 papers, 7.7k citations indexed

About

Tetsuya Nosaka is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Tetsuya Nosaka has authored 120 papers receiving a total of 7.7k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Immunology, 39 papers in Molecular Biology and 30 papers in Oncology. Recurrent topics in Tetsuya Nosaka's work include T-cell and Retrovirus Studies (22 papers), Cytokine Signaling Pathways and Interactions (21 papers) and Acute Myeloid Leukemia Research (19 papers). Tetsuya Nosaka is often cited by papers focused on T-cell and Retrovirus Studies (22 papers), Cytokine Signaling Pathways and Interactions (21 papers) and Acute Myeloid Leukemia Research (19 papers). Tetsuya Nosaka collaborates with scholars based in Japan, United States and Norway. Tetsuya Nosaka's co-authors include Toshio Kitamura, Hideaki Nakajima, Hidetoshi Kumagai, James N. Ihle, Fumi Shibata, Toshihiko Oki, Peter C. Doherty, Ralph A. Tripp, Gerard C. Grosveld and Frederick W. Quelle and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Tetsuya Nosaka

116 papers receiving 7.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Lack of IL-4-induced Th2 response and IgE class switching in mice with disrupted State6 gene

1996 1.1k citations Kazuya Shimoda, Frederick W. Quelle et al. profile →
Retrovirus-mediated gene transfer and expression cloning: powerful tools in functional genomics

2003 591 citations Toshio Kitamura, Toshihiko Oki et al. profile →
Retrovirus-mediated gene transfer and expression cloning: powerful tools in functional genomics

2003 560 citations Toshio Kitamura, Toshihiko Oki et al. profile →
Defective Lymphoid Development in Mice Lacking Jak3

1995 544 citations Tetsuya Nosaka, William E. Thierfelder et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Tetsuya Nosaka Japan	37	3.3k	3.2k	2.4k	987	757	120	7.7k
Judith C. Gasson United States	42	3.4k 1.0×	2.6k 0.8×	1.2k 0.5×	1.3k 1.3×	653 0.9×	78	7.4k
James C. Mulloy United States	41	2.0k 0.6×	4.0k 1.2×	1.3k 0.5×	2.5k 2.6×	253 0.3×	114	6.7k
Mitsuo Maruyama Japan	25	2.4k 0.7×	2.1k 0.7×	1.3k 0.5×	611 0.6×	168 0.2×	61	4.7k
David C. Scherer United States	19	3.4k 1.0×	3.4k 1.1×	1.3k 0.5×	889 0.9×	346 0.5×	25	7.1k
Shoichiro Miyatake Japan	29	2.7k 0.8×	1.8k 0.6×	776 0.3×	165 0.2×	629 0.8×	65	5.0k
Fumihiko Ishikawa Japan	33	2.3k 0.7×	2.0k 0.6×	1.6k 0.7×	1.5k 1.5×	154 0.2×	90	6.2k
Kenneth B. Marcu United States	49	2.6k 0.8×	5.1k 1.6×	1.8k 0.8×	449 0.5×	301 0.4×	122	8.6k
Tatsuo Kinashi Japan	45	4.0k 1.2×	2.3k 0.7×	935 0.4×	742 0.8×	1.2k 1.6×	96	7.0k
Jun–ichi Fujisawa Japan	40	3.0k 0.9×	3.0k 0.9×	1.1k 0.4×	130 0.1×	367 0.5×	101	6.5k
Kiyoshi Ohtani Japan	33	2.1k 0.6×	2.6k 0.8×	1.9k 0.8×	88 0.1×	416 0.5×	78	5.0k

Countries citing papers authored by Tetsuya Nosaka

Since Specialization

Citations

This map shows the geographic impact of Tetsuya Nosaka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tetsuya Nosaka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tetsuya Nosaka more than expected).

Fields of papers citing papers by Tetsuya Nosaka

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tetsuya Nosaka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tetsuya Nosaka. The network helps show where Tetsuya Nosaka may publish in the future.

Co-authorship network of co-authors of Tetsuya Nosaka

This figure shows the co-authorship network connecting the top 25 collaborators of Tetsuya Nosaka. A scholar is included among the top collaborators of Tetsuya Nosaka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tetsuya Nosaka. Tetsuya Nosaka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ohtsuka, Junpei, Masaki Imai, Masayuki Fukumura, et al.. (2021). Non-propagative human parainfluenza virus type 2 nasal vaccine robustly protects the upper and lower airways against SARS-CoV-2. iScience. 24(12). 103379–103379. 13 indexed citations

Kawashima, Toshiyuki, Ying Bao, Yukinori Minoshima, et al.. (2009). A Rac GTPase-Activating Protein, MgcRacGAP, Is a Nuclear Localizing Signal-Containing Nuclear Chaperone in the Activation of STAT Transcription Factors. Molecular and Cellular Biology. 29(7). 1796–1813. 68 indexed citations

Ikeya, Makoto, Masako Kawada, Sachiko Onishi, et al.. (2009). Cv2, functioning as a pro-BMP factor via twisted gastrulation, is required for early development of nephron precursors. Developmental Biology. 337(2). 405–414. 36 indexed citations

Watanabe‐Okochi, Naoko, Jiro Kitaura, Ryoichi Ono, et al.. (2008). AML1 mutations induced MDS and MDS/AML in a mouse BMT model. Blood. 111(8). 4297–4308. 115 indexed citations

Ikeya, Makoto, Tetsuya Nosaka, Masako Kawada, et al.. (2008). Twisted gastrulation mutation suppresses skeletal defect phenotypes in Crossveinless 2 mutant mice. Mechanisms of Development. 125(9-10). 832–842. 14 indexed citations

Matsushita, Hiromichi, Hideaki Nakajima, Yoshihiko Nakamura, et al.. (2008). C/EBPα and C/EBPɛ induce the monocytic differentiation of myelomonocytic cells with the MLL-chimeric fusion gene. Oncogene. 27(53). 6749–6760. 20 indexed citations

Yang, Lu, Jiro Kitaura, Toshihiko Oki, et al.. (2007). Identification of TSC-22 as a potential tumor suppressor that is upregulated by Flt3-D835V but not Flt3-ITD. Leukemia. 21(11). 2246–2257. 21 indexed citations

Ozaki, Katsutoshi, Keiko Hatanaka, Hideaki Nakajima, et al.. (2006). Overexpression of Interleukin 21 Induces Expansion of Hematopoietic Progenitor Cells. International Journal of Hematology. 84(3). 224–230. 19 indexed citations

Ono, Ryoichi, Hideaki Nakajima, Katsutoshi Ozaki, et al.. (2005). Dimerization of MLL fusion proteins and FLT3 activation synergize to induce multiple-lineage leukemogenesis. Journal of Clinical Investigation. 115(4). 919–929. 90 indexed citations

10.

Kawashima, Toshiyuki, Ken T. Murata, Shizuo Akira, et al.. (2001). STAT5 Induces Macrophage Differentiation of M1 Leukemia Cells Through Activation of IL-6 Production Mediated by NF-κB p65. The Journal of Immunology. 167(7). 3652–3660. 49 indexed citations

11.

Fujio, Keishi, Yoshikata Misaki, Keigo Setoguchi, et al.. (2000). Functional Reconstitution of Class II MHC-Restricted T Cell Immunity Mediated by Retroviral Transfer of the αβ TCR Complex. The Journal of Immunology. 165(1). 528–532. 72 indexed citations

12.

Onishi, Mayumi, Tetsuya Nosaka, Kazuhide Misawa, et al.. (1998). Identification and Characterization of a Constitutively Active STAT5 Mutant That Promotes Cell Proliferation. Molecular and Cellular Biology. 18(7). 3871–3879. 364 indexed citations

13.

Onishi, Mayumi, Tetsuya Nosaka, & Toshio Kitamura. (1998). Cytokine Receptors: Structures and Signal Transduction. International Reviews of Immunology. 16(5-6). 617–634. 21 indexed citations

14.

Ihle, James N., Tetsuya Nosaka, William E. Thierfelder, Frederick W. Quelle, & Kazuya Shimoda. (1997). Jaks and stats in cytokine signaling. Stem Cells. 15(S2). 105–112. 92 indexed citations

15.

Hirata, Satoshi, et al.. (1996). Clinical Analysis of N2 Patients with Skip Lymph Node Metastases in Non Small Cell Lung Cancer.. Haigan. 36(7). 893–898.

16.

Nosaka, Tetsuya, et al.. (1993). Novel internal promoter⊟;enhancer of HTLV-I for Tax expression. Nucleic Acids Research. 21(22). 5124–5129. 17 indexed citations

17.

Yamaoka, Shoji, et al.. (1992). Transforming activity and the level of tax protein: Effect of one point mutation in HTLV‐I tax gene. International Journal of Cancer. 52(2). 323–328. 8 indexed citations

18.

White, Kenneth, Tetsuya Nosaka, Hiroshi Kanamori, Masakazu Hatanaka, & Tasuku Honjo. (1991). The nucleolar localisation signal of the HTLV-I protein p27rex is important for stabilisation of IL-2 receptor α subunit mRNA by p27rex. Biochemical and Biophysical Research Communications. 175(1). 98–103. 12 indexed citations

19.

Nosaka, Tetsuya, Hitoshi Ohno, S Fukuhara, et al.. (1988). Phenotypic conversion of T lymphoblastic lymphoma to acute biphenotypic leukemia composed of lymphoblasts and myeloblasts. Molecular genetic evidence of the same clonal origin.. Journal of Clinical Investigation. 81(6). 1824–1828. 23 indexed citations

20.

Miwa, Hiroshi, Tetsuya Nosaka, Kenkichi Kita, et al.. (1988). Immunogenotypes of Lymphoid Malignancies; the Rearrangement of T Cell Receptor β Chain Gene Can Occur before the γ Chain Gene Rearrangement. Japanese Journal of Cancer Research. 79(4). 484–490. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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