Koichi Hirose

5.4k total citations
89 papers, 4.4k citations indexed

About

Koichi Hirose is a scholar working on Immunology, Physiology and Oncology. According to data from OpenAlex, Koichi Hirose has authored 89 papers receiving a total of 4.4k indexed citations (citations by other indexed papers that have themselves been cited), including 53 papers in Immunology, 30 papers in Physiology and 14 papers in Oncology. Recurrent topics in Koichi Hirose's work include Asthma and respiratory diseases (23 papers), Immune Cell Function and Interaction (19 papers) and IL-33, ST2, and ILC Pathways (18 papers). Koichi Hirose is often cited by papers focused on Asthma and respiratory diseases (23 papers), Immune Cell Function and Interaction (19 papers) and IL-33, ST2, and ILC Pathways (18 papers). Koichi Hirose collaborates with scholars based in Japan, United States and Belgium. Koichi Hirose's co-authors include Hiroshi Nakajima, Itsuo Iwamoto, Akira Suto, Shin‐ichiro Kagami, Yasushi Saito, Norihiko Watanabe, Kotaro Suzuki, Kei Ikeda, Tomohiro Tamachi and Toshio Kitamura and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Experimental Medicine and SHILAP Revista de lepidopterología.

In The Last Decade

Koichi Hirose

87 papers receiving 4.3k citations

Peers

Koichi Hirose

IMMUN

PHYSI

ONCOL

RHEUM

Tadashi Terui Japan

Leonie S. Taams United Kingdom

Francesca Barone United Kingdom

R. Gillitzer Germany

L M Wahl United States

Jean‐David Bouaziz France

L T May United States

Ioannis Mitroulis Greece

Carlo Chizzolini Switzerland

Lena Håkansson Sweden

Tadashi Terui Japan

Koichi Hirose

2.0k ×0.8

IMMUN

596 ×0.6

PHYSI

906 ×1.1

275 ×0.5

ONCOL

772 ×1.8

RHEUM

Citations per year, relative to Koichi Hirose Koichi Hirose (= 1×) peers Tadashi Terui

Countries citing papers authored by Koichi Hirose

Since Specialization

Citations

This map shows the geographic impact of Koichi Hirose's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Koichi Hirose with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Koichi Hirose more than expected).

Fields of papers citing papers by Koichi Hirose

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Koichi Hirose. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Koichi Hirose. The network helps show where Koichi Hirose may publish in the future.

Co-authorship network of co-authors of Koichi Hirose

This figure shows the co-authorship network connecting the top 25 collaborators of Koichi Hirose. A scholar is included among the top collaborators of Koichi Hirose based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Koichi Hirose. Koichi Hirose is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Furuta, Shunsuke, Michio Fujiwara, Masaki Hiraguri, et al.. (2024). Short-term and long-term outcomes of patients with anti-melanoma differentiation-associated gene 5 antibody-positive dermatomyositis. Lara D. Veeken. 64(2). 756–762. 3 indexed citations

Suto, Akira, Shigeru Tanaka, Yutaka Sugawara, et al.. (2023). TAp63, a methotrexate target in CD4+ T cells, suppresses Foxp3 expression and exacerbates autoimmune arthritis. JCI Insight. 8(10). 7 indexed citations

Yokoyama, Yusuke, Tomohiro Tamachi, Arifumi Iwata, et al.. (2022). A20 (Tnfaip3) expressed in CD4+ T cells suppresses Th2 cell-mediated allergic airway inflammation in mice. Biochemical and Biophysical Research Communications. 629. 47–53. 3 indexed citations

Takatori, Hiroaki, Ayako Matsuki, Hirotoshi Kawashima, et al.. (2020). T-bet and STAT6 Coordinately Suppress the Development of IL-9–Mediated Atopic Dermatitis–Like Skin Inflammation in Mice. Journal of Investigative Dermatology. 141(5). 1274–1285.e5. 7 indexed citations

Suto, Akira, Arifumi Iwata, Taro Iwamoto, et al.. (2020). Sox12 enhances Fbw7-mediated ubiquitination and degradation of GATA3 in Th2 cells. Cellular and Molecular Immunology. 18(7). 1729–1738. 23 indexed citations

Hirose, Koichi, Tetsufumi Ito, Arifumi Iwata, et al.. (2019). Fucosyltransferase 2 induces lung epithelial fucosylation and exacerbates house dust mite–induced airway inflammation. Journal of Allergy and Clinical Immunology. 144(3). 698–709.e9. 35 indexed citations

Muraosa, Yasunori, et al.. (2019). Schizophyllum commune induces IL-17-mediated neutrophilic airway inflammation in OVA-induced asthma model mice. Scientific Reports. 9(1). 19321–19321. 12 indexed citations

Tanaka, Shigeru, Akira Suto, Taro Iwamoto, et al.. (2018). Sox12 promotes T reg differentiation in the periphery during colitis. The Journal of Experimental Medicine. 215(10). 2509–2519. 11 indexed citations

Kageyama, Takahiro, Akira Suto, Taro Iwamoto, et al.. (2017). IL-21 Exacerbates Autoimmune Myositis by Enhancing the Accumulation of GM-CSF–Producing γδ T Cells in the Muscle. ImmunoHorizons. 1(8). 176–187. 5 indexed citations

10.

Matsuki, Ayako, Hiroaki Takatori, Masaya Yokota, et al.. (2016). T-bet inhibits innate lymphoid cell–mediated eosinophilic airway inflammation by suppressing IL-9 production. Journal of Allergy and Clinical Immunology. 139(4). 1355–1367.e6. 22 indexed citations

11.

Hirose, Koichi, Arifumi Iwata, Tomohiro Tamachi, et al.. (2014). Dectin-2 Promotes House Dust Mite–Induced T Helper Type 2 and Type 17 Cell Differentiation and Allergic Airway Inflammation in Mice. American Journal of Respiratory Cell and Molecular Biology. 51(2). 201–209. 65 indexed citations

12.

Hirose, Koichi, Kentaro Takahashi, & Hiroshi Nakajima. (2013). Roles of IL-22 in Allergic Airway Inflammation. PubMed. 2013. 1–5. 16 indexed citations

13.

Takahashi, Kentaro, Koichi Hirose, Yusuke Niwa, et al.. (2012). IL-22 attenuates IL-25 production by lung epithelial cells and inhibits antigen-induced eosinophilic airway inflammation (59.8). The Journal of Immunology. 188(1_Supplement). 59.8–59.8. 3 indexed citations

14.

Kagami, Shin‐ichiro, Takayoshi Owada, Yukari Saito, et al.. (2009). Protein geranylgeranylation regulates the balance between Th17 cells and Foxp3+ regulatory T cells. International Immunology. 21(6). 679–689. 83 indexed citations

15.

Suto, Akira, Daisuke Kashiwakuma, Shin‐ichiro Kagami, et al.. (2008). Development and characterization of IL-21–producing CD4+ T cells. The Journal of Experimental Medicine. 205(6). 1369–1379. 205 indexed citations

16.

Furuta, Shunsuke, Shin‐ichiro Kagami, Tomohiro Tamachi, et al.. (2008). Overlapping and Distinct Roles of STAT4 and T-bet in the Regulation of T Cell Differentiation and Allergic Airway Inflammation. The Journal of Immunology. 180(10). 6656–6662. 20 indexed citations

17.

Wakashin, Hidefumi, Koichi Hirose, Yoshiro Maezawa, et al.. (2008). IL-23 and Th17 Cells Enhance Th2-Cell–mediated Eosinophilic Airway Inflammation in Mice. American Journal of Respiratory and Critical Care Medicine. 178(10). 1023–1032. 349 indexed citations

18.

Takatori, Hiroaki, Hiroshi Nakajima, Koichi Hirose, et al.. (2005). Indispensable Role of Stat5a in Stat6-Independent Th2 Cell Differentiation and Allergic Airway Inflammation. The Journal of Immunology. 174(6). 3734–3740. 37 indexed citations

19.

Takatori, Hiroaki, Hiroshi Nakajima, Koichi Hirose, et al.. (2005). Stat5a Inhibits IL-12-Induced Th1 Cell Differentiation through the Induction of Suppressor of Cytokine Signaling 3 Expression. The Journal of Immunology. 174(7). 4105–4112. 47 indexed citations

20.

Isogai, Eriko, et al.. (2003). Sensitivity of genera Porphyromonas and Prevotella to the bactericidal action of C‐terminal domain of human CAP18 and its analogues. Oral Microbiology and Immunology. 18(5). 329–332. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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