Tamar Tchkonia

50.3k citations
193 papers · 28.3k · 25 hit papers · h-index 79

Impact in

  • Aging top 0.02%
    • Genetics, Aging, and Longevity in Model Organisms
  • Physiology top 0.01%
    • Telomeres, Telomerase, and Senescence
    • Adipose Tissue and Metabolism

Papers in

Tamar Tchkonia

185 papers receiving 27.9k citations

Tamar Tchkonia's Hit Papers

A pilot study of senolytics to improve cognition and mobility in older adults at risk for Alzheimer’s disease 2025 · 27 citations
270+3+6Years since publication250500750

Peers

Tamar Tchkonia
Comparison fields: 5 of 162
  • Aging 3.4k
  • Physiology 15.0k
  • Geriatrics and Gerontology 1.1k
  • Endocrine and Autonomic Systems 1.6k
  • Immunology 4.5k
Replace James L. Kirkland with:
James L. Kirkland United States
Darren J. Baker United States
Nathan K. LeBrasseur United States
Kenneth Walsh United States
Benoı̂t Viollet France
Massimiliano Bonafè Italy
João F. Passos United Kingdom
Peter S. Rabinovitch United States
Issei Komuro Japan
Fabiola Olivieri Italy
Tamar Tchkonia relative to James L. Kirkland United States James L. Kirkland's profile →
Citations per field
00.5×1.5×
James L. Kirkland · 1×
Citations per year

Countries citing papers authored by Tamar Tchkonia

Since Specialization
Citations

This map shows the geographic impact of Tamar Tchkonia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tamar Tchkonia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tamar Tchkonia more than expected).

Fields of papers citing papers by Tamar Tchkonia

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tamar Tchkonia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tamar Tchkonia. The network helps show where Tamar Tchkonia may publish in the future.

Co-authors

The 25 scholars most cited alongside Tamar Tchkonia, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tamar Tchkonia Line = papers co-authored together Tamar Tchkonia links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 193 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders
Hit paper breakdown →
20112848
2
Cellular senescence and the senescent secretory phenotype: therapeutic opportunities
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20131379
3
Senolytics in idiopathic pulmonary fibrosis: Results from a first-in-human, open-label, pilot study
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2019909
4
Targeting cellular senescence prevents age-related bone loss in mice
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2017881
5
Identification of a novel senolytic agent, navitoclax, targeting the Bcl‐2 family of anti‐apoptotic factors
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2015862
6
Fat tissue, aging, and cellular senescence
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2010854
7
Senolytic drugs: from discovery to translation
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2020790
8
Cellular senescence drives age-dependent hepatic steatosis
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2017776
9
Cellular Senescence: A Translational Perspective
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2017771
10
Cellular senescence and senolytics: the path to the clinic
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2022703
11
JAK inhibition alleviates the cellular senescence-associated secretory phenotype and frailty in old age
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2015674
12
Chronic senolytic treatment alleviates established vasomotor dysfunction in aged or atherosclerotic mice
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2016605
13
New agents that target senescent cells: the flavone, fisetin, and the BCL-XL inhibitors, A1331852 and A1155463
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2017577
14
Identification of HSP90 inhibitors as a novel class of senolytics
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2017545
15
A new gene set identifies senescent cells and predicts senescence-associated pathways across tissues
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2022523
16
Mechanisms and Metabolic Implications of Regional Differences among Fat Depots
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2013521
17
Targeting senescent cells enhances adipogenesis and metabolic function in old age
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2015480
18
Identification of Senescent Cells in the Bone Microenvironment
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2016435
19
The Clinical Potential of Senolytic Drugs
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2017414
20 2010401

About Tamar Tchkonia

Tamar Tchkonia is a scholar working on Physiology, Molecular Biology, Epidemiology, Immunology and Aging, having authored 193 papers that have together received 28.3k indexed citations. Recurring topics across this work include Telomeres, Telomerase, and Senescence (91 papers), Adipokines, Inflammation, and Metabolic Diseases (41 papers), Adipose Tissue and Metabolism (30 papers), Genetics, Aging, and Longevity in Model Organisms (27 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (22 papers), Circadian rhythm and melatonin (13 papers), MicroRNA in disease regulation (10 papers) and Mesenchymal stem cell research (9 papers). The work is most often cited by research in Aging (3.4k citations), Physiology (15.0k citations), Geriatrics and Gerontology (1.1k citations), Endocrine and Autonomic Systems (1.6k citations) and Immunology (4.5k citations). Tamar Tchkonia has collaborated with scholars based in United States, United Kingdom and China. Frequent co-authors include James L. Kirkland, Nathan K. LeBrasseur, Yi Zhu, Tamar Pirtskhalava, Michael D. Jensen, Nino Giorgadze, Sundeep Khosla, Jan van Deursen, Darren J. Baker and Jan M. van Deursen. Their work appears in journals such as Aging Cell, Aging, Obesity, American Journal of Physiology-Endocrinology and Metabolism and GeroScience.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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