Bennett G. Childs
- Aging top 0.1%
- Genetics, Aging, and Longevity in Model Organisms 2
- Physiology top 0.2%
- Telomeres, Telomerase, and Senescence 9
- Immunology top 1%
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 4
- Atherosclerosis and Cardiovascular Diseases 2
- Geriatrics and Gerontology top 1%
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- Skin Protection and Aging 2
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- Cell death mechanisms and regulation 1
- Single-cell and spatial transcriptomics 1
- DNA Repair Mechanisms 1
- Co-authors
- Jan M. van DeursenDarren J. BakerMatej DurikTobias WijshakeJames L. KirklandTamar TchkoniaBart van de SluisNathan K. LeBrasseur
- Cited by
- AgingPhysiologyImmunology
- Partner nations
- United StatesNetherlandsSouth Korea
In The Last Decade
Bennett G. Childs
12 papers receiving 9.0k citations
Hit Papers
Peers
Comparison fields: 5 of 139
- Aging 1.3k
- Physiology 4.9k
- Immunology 2.1k
- Geriatrics and Gerontology 350
- Endocrine and Autonomic Systems 449
Countries citing papers authored by Bennett G. Childs
This map shows the geographic impact of Bennett G. Childs's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bennett G. Childs with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bennett G. Childs more than expected).
Fields of papers citing papers by Bennett G. Childs
This network shows the impact of papers produced by Bennett G. Childs. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bennett G. Childs. The network helps show where Bennett G. Childs may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Bennett G. Childs, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 52 | |
| 2 | 2021 | 79 | |
| 3 | 2018 | 21 | |
| 4 | 2018 | 135 | |
| 5 | Senescent cells: an emerging target for diseases of ageingbreakdown → | 2017 | 888 |
| 6 | Senescent intimal foam cells are deleterious at all stages of atherosclerosisbreakdown → | 2016 | 857 |
| 7 | Naturally occurring p16Ink4a-positive cells shorten healthy lifespanbreakdown → | 2016 | 1984 |
| 8 | Cellular senescence in aging and age-related disease: from mechanisms to therapybreakdown → | 2015 | 1670 |
| 9 | 2015 | 9 | |
| 10 | 2014 | 88 | |
| 11 | Senescence and apoptosis: dueling or complementary cell fates?breakdown → | 2014 | 650 |
| 12 | Clearance of p16Ink4a-positive senescent cells delays ageing-associated disordersbreakdown → | 2011 | 2693 |
About Bennett G. Childs
Bennett G. Childs is a scholar working on Aging, Physiology and Dermatology, having authored 12 papers that have together received 9.1k indexed citations. Recurring topics across this work include Telomeres, Telomerase, and Senescence (9 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (4 papers), Skin Protection and Aging (2 papers), Genetics, Aging, and Longevity in Model Organisms (2 papers), Atherosclerosis and Cardiovascular Diseases (2 papers), Cell death mechanisms and regulation (1 paper), Single-cell and spatial transcriptomics (1 paper) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Aging (1.3k citations), Physiology (4.9k citations) and Immunology (2.1k citations). Bennett G. Childs has collaborated with scholars based in United States, Netherlands and South Korea. Frequent co-authors include Jan M. van Deursen, Darren J. Baker, Matej Durik, Tobias Wijshake, James L. Kirkland, Tamar Tchkonia, Bart van de Sluis, Nathan K. LeBrasseur, Judith Campisi and Karthik B. Jeganathan. Their work appears in journals such as Nature, Science and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.