Tal Keren‐Kaplan

1.3k total citations · 1 hit paper
16 papers, 884 citations indexed

About

Tal Keren‐Kaplan is a scholar working on Molecular Biology, Cell Biology and Physiology. According to data from OpenAlex, Tal Keren‐Kaplan has authored 16 papers receiving a total of 884 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 10 papers in Cell Biology and 6 papers in Physiology. Recurrent topics in Tal Keren‐Kaplan's work include Cellular transport and secretion (8 papers), Ubiquitin and proteasome pathways (7 papers) and Calcium signaling and nucleotide metabolism (6 papers). Tal Keren‐Kaplan is often cited by papers focused on Cellular transport and secretion (8 papers), Ubiquitin and proteasome pathways (7 papers) and Calcium signaling and nucleotide metabolism (6 papers). Tal Keren‐Kaplan collaborates with scholars based in United States, Israel and Australia. Tal Keren‐Kaplan's co-authors include Juan S. Bonifacino, Jing Pu, Carlos M. Guardia, Gali Prag, Ilan Attali, Shay Artzi, Rui Jia, Saikat Ghosh, Yan Li and Khatereh Motamedchaboki and has published in prestigious journals such as Nature Communications, The Journal of Cell Biology and The EMBO Journal.

In The Last Decade

Tal Keren‐Kaplan

15 papers receiving 881 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mechanisms and functions of lysosome positioning

2016 396 citations Jing Pu, Carlos M. Guardia et al. Journal of Cell Science profile →

Peers

Countries citing papers authored by Tal Keren‐Kaplan

Since Specialization

Citations

This map shows the geographic impact of Tal Keren‐Kaplan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tal Keren‐Kaplan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tal Keren‐Kaplan more than expected).

Fields of papers citing papers by Tal Keren‐Kaplan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tal Keren‐Kaplan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tal Keren‐Kaplan. The network helps show where Tal Keren‐Kaplan may publish in the future.

Co-authorship network of co-authors of Tal Keren‐Kaplan

This figure shows the co-authorship network connecting the top 25 collaborators of Tal Keren‐Kaplan. A scholar is included among the top collaborators of Tal Keren‐Kaplan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tal Keren‐Kaplan. Tal Keren‐Kaplan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

#	Work	Indexed citations
1	Structure of ClfA002 in Complex With Neutralizing Antibody AZD7745 Provides Insight into Its Broad Neutralization Mechanism in Staphylococcus aureus Infection 2025 ·The Journal of Infectious Diseases ·Christine Tkaczyk, Tal Keren‐Kaplan, (unknown), Paul Warrener, Е. А. Семенова, (unknown), Arnita Barnes, (unknown), Bret R. Sellman, Ondřej Podlaha, Vaheh Oganesyan	0
2	ARMH3 is an ARL5 effector that promotes PI4KB-catalyzed PI4P synthesis at the trans-Golgi network 2024 ·Nature Communications ·Morié Ishida, Adriana E. Golding, Tal Keren‐Kaplan, Yan Li, Tamás Balla, Juan S. Bonifacino	4
3	Consecutive functions of small GTPases guide HOPS-mediated tethering of late endosomes and lysosomes 2023 ·Cell Reports ·(unknown), (unknown), Tal Keren‐Kaplan, Jing Pu, Paul Säftig, Juan S. Bonifacino, Albert Haas, (unknown)	29
4	PI4P and BLOC-1 remodel endosomal membranes into tubules 2022 ·The Journal of Cell Biology ·(unknown), Aurélie Di Cicco, Tal Keren‐Kaplan, Sílvia Vale-Costa, (unknown), Ilse Hurbain, Feng‐Ching Tsai, (unknown), Anne‐Sophie Macé, Yueyao Zhu, Maria João Amorim, Patricia Bassereau, Juan S. Bonifacino, Agathe Subtil, Michael S. Marks, Daniel Lévy, Graça Raposo, Cédric Delevoye	16
5	RUFY3 and RUFY4 are ARL8 effectors that promote coupling of endolysosomes to dynein-dynactin 2022 ·Nature Communications ·Tal Keren‐Kaplan, Amra Sarić, Saikat Ghosh, Chad D. Williamson, Rui Jia, Yan Li, Juan S. Bonifacino	40
6	ARL8 Relieves SKIP Autoinhibition to Enable Coupling of Lysosomes to Kinesin-1 2020 ·Current Biology ·Tal Keren‐Kaplan, Juan S. Bonifacino	42
7	Phagolysosome resolution requires contacts with the endoplasmic reticulum and phosphatidylinositol-4-phosphate signalling 2019 ·Nature Cell Biology ·(unknown), (unknown), Tal Keren‐Kaplan, Li Ren, Braeden K. Ego, Sivakami Mylvaganam, (unknown), William S. Trimble, Michael C. Bassik, Juan S. Bonifacino, Gregory D. Fairn, Sergio Grinstein	69
8	E. coli-Based Selection and Expression Systems for Discovery, Characterization, and Purification of Ubiquitylated Proteins 2018 ·Methods in molecular biology ·(unknown), Tal Keren‐Kaplan, Ilan Attali, (unknown), (unknown), (unknown), (unknown), Avinash K. Persaud, (unknown), (unknown), Gali Prag	1
9	Ubiquitylation‐dependent oligomerization regulates activity of Nedd4 ligases 2017 ·The EMBO Journal ·Ilan Attali, William S. Tobelaim, Avinash K. Persaud, Khatereh Motamedchaboki, Kobi J. Simpson-Lavy, (unknown), (unknown), Tal Keren‐Kaplan, (unknown), Martin Kupiec, Reuven Wiener, Dieter A Wolf, Daniela Rotin, Gali Prag	40
10	A Ragulator–BORC interaction controls lysosome positioning in response to amino acid availability 2017 ·The Journal of Cell Biology ·Jing Pu, Tal Keren‐Kaplan, Juan S. Bonifacino	91
11	A bacterial genetic selection system for ubiquitylation cascade discovery 2016 ·Nature Methods ·(unknown), (unknown), (unknown), Ilan Attali, Tal Keren‐Kaplan, (unknown), Shay Artzi, Alexander Varvak, (unknown), Xiaojing Shi, (unknown), (unknown), (unknown), (unknown), Shay Ben‐Aroya, Gali Prag	19
12	Structure of ubiquitylated-Rpn10 provides insight into its autoregulation mechanism 2016 ·Nature Communications ·Tal Keren‐Kaplan, (unknown), (unknown), Ilan Attali, Oded Kleifeld, (unknown), Shay Artzi, (unknown), (unknown), Noa Reis, Michael H. Glickman, Shay Ben‐Aroya, Gali Prag	28
13	Mechanisms and functions of lysosome positioning breakdown → 2016 ·Journal of Cell Science ·Jing Pu, Carlos M. Guardia, Tal Keren‐Kaplan, Juan S. Bonifacino	396
14	Structure‐based in silico identification of ubiquitin‐binding domains provides insights into the ALIX‐V:ubiquitin complex and retrovirus budding 2013 ·The EMBO Journal ·Tal Keren‐Kaplan, Ilan Attali, (unknown), Lillian S. Kuo, (unknown), Moran Jerabek‐Willemsen, (unknown), Shay Artzi, (unknown), Eric O. Freed, Haim J. Wolfson, Gali Prag	54
15	Purification and crystallization of mono-ubiquitylated ubiquitin receptor Rpn10 2012 ·Acta Crystallographica Section F Structural Biology and Crystallization Communications ·Tal Keren‐Kaplan, Gali Prag	8
16	Synthetic biology approach to reconstituting the ubiquitylation cascade in bacteria 2011 ·The EMBO Journal ·Tal Keren‐Kaplan, Ilan Attali, Khatereh Motamedchaboki, Brian A. Davis, (unknown), (unknown), (unknown), Mikhail Kolot, (unknown), Oded Kleifeld, Michael H. Glickman, Bruce Horazdovsky, Dieter A Wolf, Gali Prag	47

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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