Solly Weiler

8.8k citations

37 papers · 6.9k indexed · 5 hit papers · h-index 18

Co-authors: Stanley J. Korsmeyer Michael C. Wei Tullia Lindsten Mona Ashiya Kyoung Joon Oh Michael C. Bassik Loren D. Walensky Anthony Letai
Topics: Lysosomal Storage Disorders Research (8 papers)Glycosylation and Glycoproteins Research (6 papers)Cell death mechanisms and regulation (6 papers)
Cited by: Molecular Biology Cell Biology Immunology
Journals: Science Proceedings of the National Academy of Sciences Journal of Biological Chemistry
Partner nations: United States Canada Italy

In The Last Decade

Solly Weiler

34 papers receiving 6.8k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

BCL-2, BCL-XL Sequester BH3 Domain-Only Molecules Preventing BAX- and BAK-Mediated Mitochondrial Apoptosis

2001 1.4k citations Emily H. Cheng, Michael C. Wei et al. Molecular Cell profile →
Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics

2002 1.3k citations Anthony Letai, Michael C. Bassik et al. Cancer Cell profile →
tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c

2000 1.0k citations Michael C. Wei, Tullia Lindsten et al. profile →
A Distinct Pathway Remodels Mitochondrial Cristae and Mobilizes Cytochrome c during Apoptosis

2002 890 citations Luca Scorrano, Mona Ashiya et al. Developmental Cell profile →
Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c

2000 827 citations S J Korsmeyer, Michael C. Wei et al. Cell Death and Differentiation profile →

Peers

Countries citing papers authored by Solly Weiler

Since Specialization

Citations

This map shows the geographic impact of Solly Weiler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Solly Weiler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Solly Weiler more than expected).

Fields of papers citing papers by Solly Weiler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Solly Weiler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Solly Weiler. The network helps show where Solly Weiler may publish in the future.

Co-authorship network of co-authors of Solly Weiler

This figure shows the co-authorship network connecting the top 25 collaborators of Solly Weiler. A scholar is included among the top collaborators of Solly Weiler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Solly Weiler. Solly Weiler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Abstract 3589: Preclinical evaluation of mesothelin-specific T cell receptor (TCR) fusion constructs (TRuC™s) utilizing the signaling power of the complete TCR complex: A new opportunity for solid tumor therapy 2018 ·Cancer Research ·Jian Ding,Ekta Patel,(unknown),(unknown),Rashmi Choudhary,Bonnie Le,(unknown),Solly Weiler,Daniel R. Getts,Patrick A. Baeuerle,Robert Hofmeister	2
2	23814, an Inhibitory Antibody of Ligand-Mediated Notch1 Activation, Modulates Angiogenesis and Inhibits Tumor Growth without Gastrointestinal Toxicity 2015 ·Molecular Cancer Therapeutics ·Theresa A. Proia,Feng Jiang,(unknown),Richard Nicoletti,Lingxin Kong,(unknown),Laura L. Poling,William M. Winston,(unknown),Solly Weiler,(unknown),Rónán C. O’Hagan,Jie Lin,Jenő Gyuris,Heidi Okamura	8
3	Neuregulin 1 Expression Is a Predictive Biomarker for Response to AV-203, an ERBB3 Inhibitory Antibody, in Human Tumor Models 2014 ·Clinical Cancer Research ·Kristan Meetze,Sylvie Vincent,(unknown),(unknown),(unknown),Steve Bottega,Donna McIntosh,Richard Nicoletti,William M. Winston,Solly Weiler,Bin Feng,Jenő Gyuris,Zhigang Weng	41
4	GP369, an FGFR2-IIIb–Specific Antibody, Exhibits Potent Antitumor Activity against Human Cancers Driven by Activated FGFR2 Signaling 2010 ·Cancer Research ·Ailin Bai,Kristan Meetze,(unknown),(unknown),(unknown),William M. Winston,Solly Weiler,Laura L. Poling,Ting Chen,Nesreen S. Ismail,Jinwei Jiang,Lorena Lerner,Jenő Gyuris,Zhigang Weng	123
5	Prodrug thiamine analogs as inhibitors of the enzyme transketolase 2007 ·Bioorganic & Medicinal Chemistry Letters ·Yvan Le Huérou,Indrani W. Gunawardana,Allen A. Thomas,Steven A. Boyd,(unknown),Walter E. DeWolf,(unknown),May Han,(unknown),(unknown),Christine Lemieux,Patrice Lee,(unknown),G Pöch,Todd T. Romoff,Francis Sullivan,Solly Weiler,S. Kirk Wright,Jie Lin	20
6	Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeuticsbreakdown → 2002 ·Cancer Cell ·Anthony Letai,Michael C. Bassik,Loren D. Walensky,(unknown),Solly Weiler,Stanley J. Korsmeyer	1298
7	A Distinct Pathway Remodels Mitochondrial Cristae and Mobilizes Cytochrome c during Apoptosisbreakdown → 2002 ·Developmental Cell ·Luca Scorrano,Mona Ashiya,Karolyn Buttle,Solly Weiler,Scott A. Oakes,Carmen A. Mannella,Stanley J. Korsmeyer	890
8	BCL-2, BCL-XL Sequester BH3 Domain-Only Molecules Preventing BAX- and BAK-Mediated Mitochondrial Apoptosisbreakdown → 2001 ·Molecular Cell ·Emily H. Cheng,Michael C. Wei,Solly Weiler,Richard A. Flavell,Tak W. Mak,Tullia Lindsten,Stanley J. Korsmeyer	1386
9	Backbone Dynamics for the Wild Type and a Double H52R/T56W Mutant of the vnd/NK-2 Homeodomain from Drosophila melanogaster 2001 ·Biochemistry ·(unknown),Solly Weiler,Carla Cuniberti,(unknown),Kyoung Tai No,James M. Gruschus,Angelo Perico,Marshall W. Nirenberg,James A. Ferretti	7
10	Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome cbreakdown → 2000 ·Cell Death and Differentiation ·S J Korsmeyer,Michael C. Wei,Mitsuyoshi Saito,Solly Weiler,Kyoung Joon Oh,Paul H. Schlesinger	827
11	Site-directed Mutations in the vnd/NK-2 Homeodomain 1998 ·Journal of Biological Chemistry ·Solly Weiler,James M. Gruschus,Désirée H.H. Tsao,(unknown),(unknown),Marshall W. Nirenberg,James A. Ferretti	38
12	Structural basis of an embryonically lethal single Ala → Thr mutation in the vnd/NK-2 homeodomain 1998 ·Proceedings of the National Academy of Sciences ·Bosong Xiang,Solly Weiler,Marshall W. Nirenberg,James A. Ferretti	12
13	Identification of the binding and activating sites of the sphingolipid activator protein, saposin C, with glucocerebrosidase 1995 ·Protein Science ·Solly Weiler,John M. Tomich,(unknown),(unknown),John A. Barranger	26
14	Molecular biology of glucocerebrosidase and the treatment of Gaucher disease. 1995 ·PubMed ·J. A. Barranger,John M. Tomich,Solly Weiler,(unknown),(unknown),Theodore E. Mifflin,(unknown),(unknown),Jianshe Wei,(unknown)	1
15	Position of the sulfhydryl group and the disulfide bonds of human glucocerebrosidase 1995 ·Journal of Protein Chemistry ·(unknown),Haruyuki Kinoshita,Gary A. Radke,Solly Weiler,(unknown),John M. Tomich	2
16	Assignment of a Human Melanoma Associated Gene MG50 (D2S448) to Chromosome 2p25.3 by Fluorescence in Situ Hybridization 1994 ·Genomics ·Solly Weiler,(unknown),R. Deans,June Kan‐Mitchell,M S Mitchell,Jeffrey M. Trent	11
17	Synthesis and characterization of a bioactive 82-residue sphingolipid activator protein, saposin C 1993 ·Journal of Molecular Neuroscience ·Solly Weiler,(unknown),(unknown),David B. Teplow,Yasuo Kishimoto,John S. O’Brien,John A. Barranger,John M. Tomich	9
18	Sequence of Two Alleles Responsible for Gaucher Disease 1990 ·DNA and Cell Biology ·(unknown),Toya Ohashi,Xinhua Yu,Solly Weiler,J. A. Barranger	74
19	Saposins (Sphingolipid Activator Proteins) in the Twitcher Mutant Mouse 1990 ·Journal of Neurochemistry ·(unknown),Satoshi Morimoto,Yasuo Kishimoto,Solly Weiler,John M. Tomich,John A. Barranger,Mitsuko Shinohara,Andrew M. Yeager,John S. O’Brien	1
20	The preparation of poly (dT)-5'-transferrin conjugates and hybridisation studies with poly (dA)-tailed linearised pBR322 plasmid DNA 1988 ·Biochemical Pharmacology ·Solly Weiler,Mario Ariatti,(unknown)	1

About Solly Weiler

Solly Weiler is a scholar working on Molecular Biology, Biological Psychiatry and Cell Biology, having authored 37 papers that have together received 6.9k indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (8 papers), Glycosylation and Glycoproteins Research (6 papers) and Cell death mechanisms and regulation (6 papers). The work is most often cited by research in Molecular Biology (5.6k citations), Cell Biology (747 citations) and Immunology (941 citations). Solly Weiler has collaborated with scholars based in United States, Canada and Italy. Frequent co-authors include Stanley J. Korsmeyer, Michael C. Wei, Tullia Lindsten, Mona Ashiya, Kyoung Joon Oh, Michael C. Bassik, Loren D. Walensky, Anthony Letai, Emily H. Cheng and Richard A. Flavell. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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