Scarlett J. Barker
- Physiology top 5%
- Molecular Biology
- Cellular and Molecular Neuroscience top 5%
- Neurology top 2%
- Cognitive Neuroscience top 5%
- Co-authors
- Sarah K. KaufmanAimin LiLea T. GrinbergJulian R. ThorpeLouise C. SerpellDavid W. SandersSarah L. DeVosApurwa M Sharma
- Topics
- Neurogenesis and neuroplasticity mechanisms (3 papers)Alzheimer's disease research and treatments (2 papers)Spinal Cord Injury Research (1 paper)
- Journals
- CellNeuronJournal of Neuroscience
- Partner nations
- United StatesUnited Kingdom
In The Last Decade
Scarlett J. Barker
5 papers receiving 1.4k citations
Hit Papers
Peers
Comparison fields: 5 of 88
- Physiology 705
- Molecular Biology 542
- Cellular and Molecular Neuroscience 444
- Neurology 400
- Cognitive Neuroscience 262
Countries citing papers authored by Scarlett J. Barker
This map shows the geographic impact of Scarlett J. Barker's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Scarlett J. Barker with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Scarlett J. Barker more than expected).
Fields of papers citing papers by Scarlett J. Barker
This network shows the impact of papers produced by Scarlett J. Barker. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Scarlett J. Barker. The network helps show where Scarlett J. Barker may publish in the future.
Co-authorship network of co-authors of Scarlett J. Barker
This figure shows the co-authorship network connecting the top 25 collaborators of Scarlett J. Barker. A scholar is included among the top collaborators of Scarlett J. Barker based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Scarlett J. Barker. Scarlett J. Barker is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 58 | |
| 2 | Multi-sensory Gamma Stimulation Ameliorates Alzheimer’s-Associated Pathology and Improves Cognitionbreakdown → | 473 |
| 3 | 120 | |
| 4 | 5 | |
| 5 | Distinct Tau Prion Strains Propagate in Cells and Mice and Define Different Tauopathiesbreakdown → | 720 |
About Scarlett J. Barker
Scarlett J. Barker is a scholar working on Developmental Neuroscience, Neurology and Cellular and Molecular Neuroscience, having authored 5 papers that have together received 1.4k indexed citations. Recurring topics across this work include Neurogenesis and neuroplasticity mechanisms (3 papers), Alzheimer's disease research and treatments (2 papers) and Spinal Cord Injury Research (1 paper). The work is most often cited by research in Neurology (400 citations), Physiology (705 citations) and Cellular and Molecular Neuroscience (444 citations). Scarlett J. Barker has collaborated with scholars based in United States and United Kingdom. Frequent co-authors include Sarah K. Kaufman, Aimin Li, Lea T. Grinberg, Julian R. Thorpe, Louise C. Serpell, David W. Sanders, Sarah L. DeVos, Apurwa M Sharma, Hilda Mirbaha and Timothy M. Miller. Their work appears in journals such as Cell, Neuron and Journal of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.