L. Ashley Watson

1.2k total citations
13 papers, 810 citations indexed

About

L. Ashley Watson is a scholar working on Molecular Biology, Physiology and Genetics. According to data from OpenAlex, L. Ashley Watson has authored 13 papers receiving a total of 810 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 6 papers in Physiology and 4 papers in Genetics. Recurrent topics in L. Ashley Watson's work include Genetics and Neurodevelopmental Disorders (4 papers), Epigenetics and DNA Methylation (4 papers) and DNA Repair Mechanisms (3 papers). L. Ashley Watson is often cited by papers focused on Genetics and Neurodevelopmental Disorders (4 papers), Epigenetics and DNA Methylation (4 papers) and DNA Repair Mechanisms (3 papers). L. Ashley Watson collaborates with scholars based in United States, Canada and United Kingdom. L. Ashley Watson's co-authors include Li‐Huei Tsai, Nathalie G. Bérubé, Alexi Nott, Fan Gao, Yea Jin Kaeser‐Woo, Carol Liu, Anthony J. Martorell, Omer Durak, Richard Rueda and Jay Penney and has published in prestigious journals such as Journal of Clinical Investigation, Nature Communications and Journal of Neuroscience.

In The Last Decade

L. Ashley Watson

13 papers receiving 809 citations

Peers

L. Ashley Watson
Mark J. Zylka United States
Laura A. Jansen United States
John J. McMahon United States
Marjelo A. Mines United States
Sungbo Shim South Korea
L. Ashley Watson
Citations per year, relative to L. Ashley Watson L. Ashley Watson (= 1×) peers Aleksandra Janusz

Countries citing papers authored by L. Ashley Watson

Since Specialization
Citations

This map shows the geographic impact of L. Ashley Watson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by L. Ashley Watson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites L. Ashley Watson more than expected).

Fields of papers citing papers by L. Ashley Watson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by L. Ashley Watson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by L. Ashley Watson. The network helps show where L. Ashley Watson may publish in the future.

Co-authorship network of co-authors of L. Ashley Watson

This figure shows the co-authorship network connecting the top 25 collaborators of L. Ashley Watson. A scholar is included among the top collaborators of L. Ashley Watson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with L. Ashley Watson. L. Ashley Watson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Watson, L. Ashley & Hiruy S. Meharena. (2023). From neurodevelopment to neurodegeneration: utilizing human stem cell models to gain insight into Down syndrome. Frontiers in Genetics. 14. 1198129–1198129. 7 indexed citations
2.
Meharena, Hiruy S., Asaf Marco, Vishnu Dileep, et al.. (2022). Down-syndrome-induced senescence disrupts the nuclear architecture of neural progenitors. Cell stem cell. 29(1). 116–130.e7. 56 indexed citations
3.
Pao, Ping‐Chieh, Debasis Patnaik, L. Ashley Watson, et al.. (2020). HDAC1 modulates OGG1-initiated oxidative DNA damage repair in the aging brain and Alzheimer’s disease. Nature Communications. 11(1). 2484–2484. 157 indexed citations
4.
Ralvenius, William T., Alexi Nott, L. Ashley Watson, et al.. (2020). Phosphoproteomics identifies microglial Siglec‐F inflammatory response during neurodegeneration. Molecular Systems Biology. 16(12). e9819–e9819. 24 indexed citations
5.
Seo, Jinsoo, Oleg Kritskiy, L. Ashley Watson, et al.. (2017). Inhibition of p25/Cdk5 Attenuates Tauopathy in Mouse and iPSC Models of Frontotemporal Dementia. Journal of Neuroscience. 37(41). 9917–9924. 120 indexed citations
6.
Watson, L. Ashley & Li‐Huei Tsai. (2016). In the loop: how chromatin topology links genome structure to function in mechanisms underlying learning and memory. Current Opinion in Neurobiology. 43. 48–55. 33 indexed citations
7.
Durak, Omer, Fan Gao, Yea Jin Kaeser‐Woo, et al.. (2016). Chd8 mediates cortical neurogenesis via transcriptional regulation of cell cycle and Wnt signaling. Nature Neuroscience. 19(11). 1477–1488. 160 indexed citations
8.
Ingle, Gordon, Valeria Iodice, Ekawat Vichayanrat, et al.. (2015). Autonomic involvement in Holmes-Adie-Ross spectrum. Autonomic Neuroscience. 192. 132–132. 1 indexed citations
9.
Watson, L. Ashley, et al.. (2015). Emerging Roles of ATRX in Cancer. Epigenomics. 7(8). 1365–1378. 49 indexed citations
10.
Watson, L. Ashley, et al.. (2014). Dual Effect of CTCF Loss on Neuroprogenitor Differentiation and Survival. Journal of Neuroscience. 34(8). 2860–2870. 64 indexed citations
11.
Watson, L. Ashley, et al.. (2014). ATRX is required for maintenance of the neuroprogenitor cell pool in the embryonic mouse brain. Biology Open. 3(12). 1158–1163. 27 indexed citations
12.
Watson, L. Ashley, Lauren A. Solomon, Yan Jiang, et al.. (2013). Atrx deficiency induces telomere dysfunction, endocrine defects, and reduced life span. Journal of Clinical Investigation. 123(5). 2049–2063. 94 indexed citations
13.
Solomon, Lauren A., et al.. (2013). Targeted loss of the ATR-X syndrome protein in the limb mesenchyme of mice causes brachydactyly. Human Molecular Genetics. 22(24). 5015–5025. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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