Satomi Maekawa

1.4k citations

13 papers · 1.1k indexed · h-index 11

Neurology top 1%
- Amyotrophic Lateral Sclerosis Research 11
- Parkinson's Disease Mechanisms and Treatments 8
- Neurological diseases and metabolism 5
Genetics top 2%
- Neurogenetic and Muscular Disorders Research 3
Neurology top 5%
- Amyotrophic Lateral Sclerosis Research 11
- Parkinson's Disease Mechanisms and Treatments 8
- Neurological diseases and metabolism 5
Physiology top 10%
- Alzheimer's disease research and treatments 4
Cellular and Molecular Neuroscience top 10%
Molecular Biology
- Prion Diseases and Protein Misfolding 2
- S100 Proteins and Annexins 1
Pharmacology
- Cholinesterase and Neurodegenerative Diseases 1

Co-authors: Safa Al‐Sarraj Andrew King Claire Troakes István Bódi Christopher E. Shaw Bradley Smith Tibor Hortobágyi Ammar Al‐Chalabi
Cited by: Neurology Genetics Physiology
Journals: Neuropathology (5 papers)Acta Neuropathologica (3 papers)Neuropathology and Applied Neurobiology (2 papers)
Partner nations: United Kingdom United States Italy

In The Last Decade

Satomi Maekawa

13 papers receiving 1.1k citations

Peers

Countries citing papers authored by Satomi Maekawa

Since Specialization

Citations

This map shows the geographic impact of Satomi Maekawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Satomi Maekawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Satomi Maekawa more than expected).

Fields of papers citing papers by Satomi Maekawa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Satomi Maekawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Satomi Maekawa. The network helps show where Satomi Maekawa may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Satomi Maekawa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Satomi Maekawa Line = papers co-authored together Satomi Maekawa links everyone, so they are left out of the graph.

All Works

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13 of 13 papers shown

#	Work
1	Clusterin expression is upregulated following acute head injury and localizes to astrocytes in old head injury Neuropathology ·Claire Troakes,HumaKousar Sangreskop,Farzana Noor,Satomi Maekawa,Richard Killick,Andrew King,Safa Al‐Sarraj	2016	23
2	Novel mutations support a role for Profilin 1 in the pathogenesis of ALS Neurobiology of Aging ·Bradley Smith,Caroline Vance,Emma L. Scotter,Claire Troakes,Chun Hao Wong,Simon Topp,Satomi Maekawa,Andrew King,Jacqueline C. Mitchell,张翠玉,Ammar Al‐Chalabi,Nicola Ticozzi,Vincenzo Silani,Peter C. Sapp,Robert H. Brown,John E. Landers,Safa Al‐Sarraj,Christopher E. Shaw	2014	81
3	Neurofibrillary tangle formation by introducing wild-type human tau into APP transgenic mice Acta Neuropathologica ·Tomohiro Umeda,Satomi Maekawa,Tetsuya Kimura,Akihiko Takashima,Takami Tomiyama,Hiroshi Mori	2014	55
4	Simulated surgical-type cerebral biopsies from post-mortem brains allows accurate neuropathological diagnoses in the majority of neurodegenerative disease groups Acta Neuropathologica Communications ·Andrew King,Satomi Maekawa,István Bódi,Claire Troakes,Olimpia E. Curran,Keyoumars Ashkan,Safa Al‐Sarraj	2013	5
5	Mixed tau, TDP-43 and p62 pathology in FTLD associated with a C9ORF72 repeat expansion and p.Ala239Thr MAPT (tau) variant Acta Neuropathologica ·Andrew King,Safa Al‐Sarraj,Claire Troakes,Bradley Smith,Satomi Maekawa,Mariangela Iovino,Maria Grazia Spillantini,Christopher E. Shaw	2012	63
6	p62 positive, TDP-43 negative, neuronal cytoplasmic and intranuclear inclusions in the cerebellum and hippocampus define the pathology of C9orf72-linked FTLD and MND/ALS Acta Neuropathologica ·Safa Al‐Sarraj,Andrew King,Claire Troakes,Bradley Smith,Satomi Maekawa,István Bódi,Boris Rogelj,Ammar Al‐Chalabi,Tibor Hortobágyi,Christopher E. Shaw	2011	369
7	An MND/ALS phenotype associated with C9orf72 repeat expansion: Abundant p62‐positive, TDP‐43‐negative inclusions in cerebral cortex, hippocampus and cerebellum but without associated cognitive decline Neuropathology ·Claire Troakes,Satomi Maekawa,Lokesh Wijesekera,Boris Rogelj,László Siklós,Christopher Bell,Bradley Smith,Stephen Newhouse,Caroline Vance,Lauren Johnson,Tibor Hortobágyi,Aleksey Shatunov,Ammar Al‐Chalabi,Nigel Leigh,Christopher E. Shaw,Andrew King,Safa Al‐Sarraj	2011	105
8	Abnormal TDP-43 expression is identified in the neocortex in cases of dementia pugilistica, but is mainly confined to the limbic system when identified in high and moderate stages of Alzheimer's disease Neuropathology ·Andrew King,С. В. Дмитриев,István Bódi,Claire Troakes,Satomi Maekawa,Safa Al‐Sarraj	2010	91
9	Ubiquitinated, p62 immunopositive cerebellar cortical neuronal inclusions are evident across the spectrum of TDP-43 proteinopathies but are only rarely additionally immunopositive for phosphorylation-dependent TDP-43 Neuropathology ·Andrew King,Satomi Maekawa,István Bódi,Claire Troakes,Safa Al‐Sarraj	2010	56
10	TDP‐43 is consistently co‐localized with ubiquitinated inclusions in sporadic and Guam amyotrophic lateral sclerosis but not in familial amyotrophic lateral sclerosis with and without SOD1 mutations Neuropathology ·Satomi Maekawa,P. Nigel Leigh,Andrew King,Grupo Español para el Desarrollo de la Farmacia Oncológica,John C. Steele,István Bódi,Christopher E. Shaw,Tibor Hortobágyi,Safa Al‐Sarraj	2009	93
11	Cortical selective vulnerability in motor neuron disease: a morphometric study Brain ·Satomi Maekawa,Safa Al‐Sarraj,Milla Kibble,Sabine Landau,John G. Parnavelas,David Cotter,Ian Everall,P. Nigel Leigh	2004	135
12	Ubiquitin‐only intraneuronal inclusion in the substantia nigra is a characteristic feature of motor neurone disease with dementia Neuropathology and Applied Neurobiology ·Safa Al‐Sarraj,Satomi Maekawa,Milla Kibble,Ian Everall,Nigel Leigh	2002	26
13	Selective loss of inhibitory neurons in the cerebral cortex in motor neuron disease Neuropathology and Applied Neurobiology ·Satomi Maekawa,David Cotter,Nigel Leigh,Safa Al‐Sarraj	2002	1

About Satomi Maekawa

Satomi Maekawa is a scholar working on Neurology, Neurology, Genetics, Physiology and Pharmacology, having authored 13 papers that have together received 1.1k indexed citations. Recurring topics across this work include Amyotrophic Lateral Sclerosis Research (11 papers), Parkinson's Disease Mechanisms and Treatments (8 papers), Neurological diseases and metabolism (5 papers), Alzheimer's disease research and treatments (4 papers), Neurogenetic and Muscular Disorders Research (3 papers), Prion Diseases and Protein Misfolding (2 papers), Cholinesterase and Neurodegenerative Diseases (1 paper) and S100 Proteins and Annexins (1 paper). The work is most often cited by research in Neurology (926 citations), Genetics (367 citations), Neurology (220 citations), Physiology (323 citations) and Cellular and Molecular Neuroscience (225 citations). Satomi Maekawa has collaborated with scholars based in United Kingdom, United States and Italy. Frequent co-authors include Safa Al‐Sarraj, Andrew King, Claire Troakes, István Bódi, Christopher E. Shaw, Bradley Smith, Tibor Hortobágyi, Ammar Al‐Chalabi, Boris Rogelj and P. Nigel Leigh. Their work appears in journals such as Neuropathology, Acta Neuropathologica, Neuropathology and Applied Neurobiology, Neurobiology of Aging and Acta Neuropathologica Communications.

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