Christopher E. Shaw

57.2k citations

242 papers · 19.0k indexed · 7 hit papers · h-index 69

Neurology top 0.01%
- Amyotrophic Lateral Sclerosis Research 171
- Parkinson's Disease Mechanisms and Treatments 51
- Neurological diseases and metabolism 33
Genetics top 0.02%
- Neurogenetic and Muscular Disorders Research 117
Neurology top 0.1%
- Amyotrophic Lateral Sclerosis Research 171
- Parkinson's Disease Mechanisms and Treatments 51
- Neurological diseases and metabolism 33
Cellular and Molecular Neuroscience top 0.5%
- Genetic Neurodegenerative Diseases 15
Molecular Biology top 0.5%
- Prion Diseases and Protein Misfolding 24
- RNA Research and Splicing 14
Physiology
- Alzheimer's disease research and treatments 24

Co-authors: Ammar Al‐Chalabi Christopher C.J. Miller P. Nigel Leigh Boris Rogelj Caroline Vance Steven Ackerley Agnes L. Nishimura Tibor Hortobágyi
Cited by: Neurology Genetics
Journals: Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration (15 papers)Journal of Neurology Neurosurgery & Psychiatry (12 papers)Neurobiology of Aging (12 papers)
Partner nations: United Kingdom United States Slovenia

In The Last Decade

Christopher E. Shaw

241 papers receiving 18.8k citations

Hit Papers

7 papers align trajectories log scale

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

2014 Neuron
2014 Nature Chemical Biology
2014 Nature Communications
2011 Nature Neuroscience
2011 Human Molecular Genetics
2008 Science
2004 Neurobiology of Disease

Peers

Countries citing papers authored by Christopher E. Shaw

Since Specialization

Citations

This map shows the geographic impact of Christopher E. Shaw's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher E. Shaw with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher E. Shaw more than expected).

Fields of papers citing papers by Christopher E. Shaw

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher E. Shaw. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher E. Shaw. The network helps show where Christopher E. Shaw may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Christopher E. Shaw, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Christopher E. Shaw Line = papers co-authored together Christopher E. Shaw links everyone, so they are left out of the graph.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

#	Work
1	Mutations in FUS lead to synaptic dysregulation in ALS-iPSC derived neurons Stem Cell Reports ·Carole Shum,T.G. Veldman,Nguyen Ba Liem,Youn‐Bok Lee,Natalia Arias,Graham Cocks,Siddharthan Chandran,Marc‐David Ruepp,Christopher E. Shaw,Agnes L. Nishimura	2024	4
2	SOD1-ALS-Browser: a web-utility for investigating the clinical phenotype in SOD1 amyotrophic lateral sclerosis Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration ·Thomas P Spargo,Sarah Opie-Martin,Ryan Alexander,Xiaoqin Lu,Ahmad Al Khleifat,Simon Topp,Christopher E. Shaw,Ammar Al‐Chalabi,Alfredo Iacoangeli,(unknown)	2023	3
3	Generation of an Open-Access Patient-Derived iPSC Biobank for Amyotrophic Lateral Sclerosis Disease Modelling Genes ·T.G. Veldman,Graham Cocks,Christopher E. Shaw,Agnes L. Nishimura	2023	4
4	A recessive S174X mutation in Optineurin causes amyotrophic lateral sclerosis through a loss of function via allele-specific nonsense-mediated decay Neurobiology of Aging ·Marc Gotkine,Martina de Majo,Chun Hao Wong,Simon Topp,Rachel Michaelson‐Cohen,Silvina Epsztejn‐Litman,Rachel Eiges,Juan D. Jiménez,Moien Kanaan,Febi Iswandi,英仁遠藤,Caroline Vance,Stephen Newhouse,Gerome Breen,Agnes L. Nishimura,Christopher E. Shaw,Bradley Smith	2021	5
5	The use of biotelemetry to explore disease progression markers in amyotrophic lateral sclerosis Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration ·Yang G,Arseniy Lavrov,Luis Garcia‐Gancedo,Jim Parr,Robert G. Hart,Joris Boulo,Christopher E. Shaw,Ammar Al‐Chalabi,Rachael Marsden,Martin R. Turner,Kevin Talbot	2020	17
6	Cross-reactive probes on Illumina DNA methylation arrays: a large study on ALS shows that a cautionary approach is warranted in interpreting epigenome-wide association studies NAR Genomics and Bioinformatics ·Paul J. Hop,Ramona A. J. Zwamborn,Eilís Hannon,Annelot M. Dekker,Kristel R. van Eijk,Emma Walker,Alfredo Iacoangeli,Ashley Jones,Aleksey Shatunov,Ahmad Al Khleifat,Sarah Opie-Martin,Christopher E. Shaw,Karen Morrison,Pamela J. Shaw,Russell L. McLaughlin,Orla Hardiman,Ammar Al‐Chalabi,Leonard H. van den Berg,Jonathan Mill,Jan H. Veldink	2020	15
7	Objectively Monitoring Amyotrophic Lateral Sclerosis Patient Symptoms During Clinical Trials With Sensors: Observational Study JMIR mhealth and uhealth ·Luis Garcia‐Gancedo,Yang G,Arseniy Lavrov,Jim Parr,R G Hart,Rachael Marsden,Martin R. Turner,Kevin Talbot,Joris Boulo,Christopher E. Shaw,Ammar Al‐Chalabi	2019	41
8	Striking phenotypic variation in a family with the P506S UBQLN2 mutation including amyotrophic lateral sclerosis, spastic paraplegia, and frontotemporal dementia Neurobiology of Aging ·Soragia Athina Gkazi,Claire Troakes,Simon Topp,Jack W. Miller,Caroline Vance,Jemeen Sreedharan,Ammar Al‐Chalabi,Janine Kirby,Pamela J. Shaw,Safa Al‐Sarraj,Andrew King,Bradley Smith,Christopher E. Shaw	2018	15
9	Younger age of onset in familial amyotrophic lateral sclerosis is a result of pathogenic gene variants, rather than ascertainment bias Journal of Neurology Neurosurgery & Psychiatry ·Puja R. Mehta,Ashley Jones,Sarah Opie-Martin,Aleksey Shatunov,Alfredo Iacoangeli,Ahmad Al Khleifat,Bradley Smith,Simon Topp,Karen Morrison,Pamela J. Shaw,Christopher E. Shaw,Sarah Morgan,Alan Pittman,Ammar Al‐Chalabi	2018	29
10	Psychological as well as illness factors influence acceptance of non-invasive ventilation (NIV) and gastrostomy in amyotrophic lateral sclerosis (ALS): A prospective population study Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration ·Naomi Martin,Sabine Landau,Anna Janssen,Rebecca Lyall,Irene J Higginson,Rachel Burman,Paul McCrone,方刚,C. M. Ellis,Christopher E. Shaw,Ammar Al‐Chalabi,P. Nigel Leigh,Laura H. Goldstein	2014	44
11	Novel mutations support a role for Profilin 1 in the pathogenesis of ALS Neurobiology of Aging ·Bradley Smith,Caroline Vance,Emma L. Scotter,Claire Troakes,Chun Hao Wong,Simon Topp,Satomi Maekawa,Andrew King,Jacqueline C. Mitchell,张翠玉,Ammar Al‐Chalabi,Nicola Ticozzi,Vincenzo Silani,Peter C. Sapp,Robert H. Brown,John E. Landers,Safa Al‐Sarraj,Christopher E. Shaw	2014	81
12	Mutant induced pluripotent stem cell lines recapitulate aspects of TDP-43 proteinopathies and reveal cell-specific vulnerability Proceedings of the National Academy of Sciences ·Bilada Bilican,Andrea Serio,Sami J. Barmada,Agnes L. Nishimura,Gareth J. Sullivan,Mónica A. Carrasco,Hemali Phatnani,Clare A. Puddifoot,David Story,Judy Fletcher,In-Hyun Park,Brad A. Friedman,George Q. Daley,David J. A. Wyllie,Giles E. Hardingham,Ian Wilmut,Steven Finkbeiner,Tom Maniatis,Christopher E. Shaw,Siddharthan Chandran	2012	257
13	VAPB interacts with the mitochondrial protein PTPIP51 to regulate calcium homeostasisbreakdown → Human Molecular Genetics ·Kurt J. De Vos,Gábor M. Mórotz,Radu Stoica,Elizabeth L. Tudor,Kwok‐Fai Lau,Steven Ackerley,Alice Warley,Christopher E. Shaw,Christopher C.J. Miller	2011	454
14	Initial Effects of Prescribed Burning and Understory Fertilization on Browse Production in Closed-Canopy Hardwood Stands Journal of Fish and Wildlife Management ·Christopher E. Shaw,Craig A. Harper,Michael W. Black,Allan E. Houston	2010	16
15	TDP-43 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosisbreakdown → Science ·Jemeen Sreedharan,Ian P. Blair,Yu-bao CHEN,Xun Hu,Caroline Vance,Boris Rogelj,Steven Ackerley,Jennifer C. Durnall,Kelly L. Williams,Emanuele Buratti,Francisco E. Baralle,Jacqueline de Belleroche,John D. Mitchell,P. Nigel Leigh,Ammar Al‐Chalabi,Christopher C.J. Miller,Garth A. Nicholson,Christopher E. Shaw	2008	2037
16	Progressive loss of PAX6, TBR2, NEUROD and TBR1 mRNA gradients correlates with translocation of EMX2 to the cortical plate during human cortical development European Journal of Neuroscience ·Nadhim Bayatti,Darren Chaney,Christopher E. Shaw,J A Eyre,Demetrius A. Vouyiouklis,Susan Lindsay,Gavin J. Clowry	2008	59
17	Building user participation to reshape services for people affected by motor neurone disease. PubMed ·S. Field,Weichu Fu,Rhidian Hughes,Irene J Higginson,Nigel Leigh,Christopher E. Shaw	2002	1
18	Intron 7 retention and exon 9 skipping EAAT2 mRNA variants are not associated with amyotrophic lateral sclerosis Annals of Neurology ·Audra M. Akins,John Powell,P. Nigel Leigh,Peter M. Andersen,Christopher E. Shaw	2001	54
19	Using GFP-tagged neurofilament middle chain to investigate slow transport in cultured cortical neurons European Journal of Neuroscience ·Steven Ackerley,Andrew J. Grierson,Janet Brownlees,P. Nigel Leigh,Christopher E. Shaw,Christopher C.J. Miller	2000	1
20	Regulatory Peptides in Helminth Parasites Advances in Parasitology ·D.W. Halton,Christopher E. Shaw,Aaron G. Maule,David Smart	1994	64

About Christopher E. Shaw

Christopher E. Shaw is a scholar working on Neurology, Genetics and Neurology, having authored 242 papers that have together received 19.0k indexed citations. Recurring topics across this work include Amyotrophic Lateral Sclerosis Research (171 papers), Neurogenetic and Muscular Disorders Research (117 papers), Parkinson's Disease Mechanisms and Treatments (51 papers), Neurological diseases and metabolism (33 papers), Prion Diseases and Protein Misfolding (24 papers), Alzheimer's disease research and treatments (24 papers), Genetic Neurodegenerative Diseases (15 papers) and RNA Research and Splicing (14 papers). The work is most often cited by research in Neurology (12.3k citations), Genetics (6.9k citations) and Neurology (2.8k citations). Christopher E. Shaw has collaborated with scholars based in United Kingdom, United States and Slovenia. Frequent co-authors include Ammar Al‐Chalabi, Christopher C.J. Miller, P. Nigel Leigh, Boris Rogelj, Caroline Vance, Steven Ackerley, P. Nigel Leigh, Agnes L. Nishimura, Tibor Hortobágyi and Siddharthan Chandran. Their work appears in journals such as Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration, Journal of Neurology Neurosurgery & Psychiatry, Neurobiology of Aging, Brain and Human Molecular Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact