Simon Topp

9.2k total citations
29 papers, 861 citations indexed

About

Simon Topp is a scholar working on Neurology, Molecular Biology and Genetics. According to data from OpenAlex, Simon Topp has authored 29 papers receiving a total of 861 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Neurology, 11 papers in Molecular Biology and 10 papers in Genetics. Recurrent topics in Simon Topp's work include Amyotrophic Lateral Sclerosis Research (15 papers), Neurogenetic and Muscular Disorders Research (10 papers) and Parkinson's Disease Mechanisms and Treatments (6 papers). Simon Topp is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (15 papers), Neurogenetic and Muscular Disorders Research (10 papers) and Parkinson's Disease Mechanisms and Treatments (6 papers). Simon Topp collaborates with scholars based in United Kingdom, Denmark and United States. Simon Topp's co-authors include Craig H. Meyer, Albert Macovski, Elfar Adalsteinsson, Daniel M. Spielman, Pablo Irarrázabal, Christopher E. Shaw, Bradley Smith, Sverre Rosenbaum, Claire Troakes and Lene Werdelin and has published in prestigious journals such as Brain, Neurology and Brain Research.

In The Last Decade

Simon Topp

29 papers receiving 850 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Simon Topp United Kingdom 15 360 297 242 184 134 29 861
Caroline Guglielmetti United States 18 112 0.3× 248 0.8× 329 1.4× 41 0.2× 153 1.1× 28 1.2k
Jean-Guy Villemure Canada 9 57 0.2× 329 1.1× 368 1.5× 192 1.0× 180 1.3× 10 937
Vladimir V. Bamm Canada 23 156 0.4× 720 2.4× 56 0.2× 42 0.2× 150 1.1× 43 1.2k
Frank Riemer United Kingdom 17 76 0.2× 173 0.6× 440 1.8× 79 0.4× 52 0.4× 42 987
Detlef Stiller Germany 20 160 0.4× 203 0.7× 376 1.6× 24 0.1× 108 0.8× 49 1.2k
Pierre-Gilles Henry United States 14 192 0.5× 364 1.2× 358 1.5× 12 0.1× 422 3.1× 21 941
André Sauter Switzerland 20 110 0.3× 317 1.1× 305 1.3× 19 0.1× 322 2.4× 41 1.3k
G. O. Sperber Sweden 17 108 0.3× 373 1.3× 483 2.0× 17 0.1× 109 0.8× 40 1.2k
M. Lowry United Kingdom 17 139 0.4× 186 0.6× 473 2.0× 20 0.1× 85 0.6× 29 886
F. Soussaline France 15 182 0.5× 311 1.0× 593 2.5× 82 0.4× 191 1.4× 42 1.3k

Countries citing papers authored by Simon Topp

Since Specialization
Citations

This map shows the geographic impact of Simon Topp's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Simon Topp with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Simon Topp more than expected).

Fields of papers citing papers by Simon Topp

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Simon Topp. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Simon Topp. The network helps show where Simon Topp may publish in the future.

Co-authorship network of co-authors of Simon Topp

This figure shows the co-authorship network connecting the top 25 collaborators of Simon Topp. A scholar is included among the top collaborators of Simon Topp based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Simon Topp. Simon Topp is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Spargo, Thomas P, Sarah Opie-Martin, Ahmad Al Khleifat, et al.. (2023). SOD1-ALS-Browser: a web-utility for investigating the clinical phenotype in SOD1 amyotrophic lateral sclerosis. Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration. 24(7-8). 736–745. 3 indexed citations
2.
Gotkine, Marc, Martina de Majo, Chun Hao Wong, et al.. (2021). A recessive S174X mutation in Optineurin causes amyotrophic lateral sclerosis through a loss of function via allele-specific nonsense-mediated decay. Neurobiology of Aging. 106. 1–6. 5 indexed citations
3.
4.
Gkazi, Soragia Athina, Claire Troakes, Simon Topp, et al.. (2018). Striking phenotypic variation in a family with the P506S UBQLN2 mutation including amyotrophic lateral sclerosis, spastic paraplegia, and frontotemporal dementia. Neurobiology of Aging. 73. 229.e5–229.e9. 15 indexed citations
5.
Mehta, Puja R., Ashley Jones, Sarah Opie-Martin, et al.. (2018). Younger age of onset in familial amyotrophic lateral sclerosis is a result of pathogenic gene variants, rather than ascertainment bias. Journal of Neurology Neurosurgery & Psychiatry. 90(3). 268–271. 29 indexed citations
6.
Niblock, Michael, Bradley Smith, Youn‐Bok Lee, et al.. (2016). Retention of hexanucleotide repeat-containing intron in C9orf72 mRNA: implications for the pathogenesis of ALS/FTD. Acta Neuropathologica Communications. 4(1). 18–18. 45 indexed citations
7.
Gotkine, Marc, Martina de Majo, Simon Topp, et al.. (2016). A novel optineurin truncation mutation identified in a consanguineous Palestinian family with Amyotrophic Lateral Sclerosis confirms loss of function as a disease mechanism. Faculty of 1000 Research Ltd. 5. 3 indexed citations
8.
Wong, Chun Hao, Simon Topp, Soragia Athina Gkazi, et al.. (2015). The CHCHD10 P34S variant is not associated with ALS in a UK cohort of familial and sporadic patients. Neurobiology of Aging. 36(10). 2908.e17–2908.e18. 14 indexed citations
9.
Smith, Bradley, Caroline Vance, Emma L. Scotter, et al.. (2014). Novel mutations support a role for Profilin 1 in the pathogenesis of ALS. Neurobiology of Aging. 36(3). 1602.e17–1602.e27. 81 indexed citations
10.
Smith, Bradley, Simon Topp, Caroline Vance, et al.. (2014). Autosomal dominant inheritance of rapidly progressive amyotrophic lateral sclerosis due to a truncation mutation in the fused in sarcoma (FUS) gene. Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration. 15(7-8). 557–562. 13 indexed citations
11.
Miller, Jack W., Bradley Smith, Simon Topp, et al.. (2012). Mutation analysis of VCP in British familial and sporadic amyotrophic lateral sclerosis patients. Neurobiology of Aging. 33(11). 2721.e1–2721.e2. 16 indexed citations
12.
Johnson, Lauren, Jack W. Miller, Soragia Athina Gkazi, et al.. (2012). Screening for OPTN mutations in a cohort of British amyotrophic lateral sclerosis patients. Neurobiology of Aging. 33(12). 2948.e15–2948.e17. 16 indexed citations
13.
Rubio, Justin P., Simon Topp, Liling Warren, et al.. (2012). Deep sequencing of theLRRK2gene in 14,002 individuals reveals evidence of purifying selection and independent origin of the p.Arg1628Pro mutation in Europe. Human Mutation. 33(7). 1087–1098. 24 indexed citations
14.
Topp, Simon, Raymond T. Doty, Bhavesh Borate, et al.. (2010). Feline leukemia virus integrase and capsid packaging functions do not change the insertion profile of standard Moloney retroviral vectors. Gene Therapy. 17(6). 799–804. 4 indexed citations
15.
Foord, Steven M., et al.. (2005). New Methods for Researching Accessory Proteins. Journal of Molecular Neuroscience. 26(2-3). 265–276. 10 indexed citations
16.
17.
Topp, Simon, et al.. (1999). Localizedin vivo1H NMR spectroscopy of murine tumours: effect of blood flow reduction. NMR in Biomedicine. 12(4). 175–183. 4 indexed citations
18.
Adalsteinsson, Elfar, Pablo Irarrázabal, Simon Topp, et al.. (1998). Volumetric spectroscopic imaging with spiral‐based k‐space trajectories. Magnetic Resonance in Medicine. 39(6). 889–898. 165 indexed citations
19.
20.
Olsen, Dan, et al.. (1996). Quantitative 1D saturation profiles on chalk by NMR. Magnetic Resonance Imaging. 14(7-8). 847–851. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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