László Siklós

3.8k total citations · 1 hit paper
48 papers, 3.1k citations indexed

About

László Siklós is a scholar working on Neurology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, László Siklós has authored 48 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Neurology, 16 papers in Cellular and Molecular Neuroscience and 15 papers in Molecular Biology. Recurrent topics in László Siklós's work include Amyotrophic Lateral Sclerosis Research (25 papers), Parkinson's Disease Mechanisms and Treatments (13 papers) and Neuroinflammation and Neurodegeneration Mechanisms (9 papers). László Siklós is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (25 papers), Parkinson's Disease Mechanisms and Treatments (13 papers) and Neuroinflammation and Neurodegeneration Mechanisms (9 papers). László Siklós collaborates with scholars based in Hungary, United States and Germany. László Siklós's co-authors include Stanley H. Appel, David R. Beers, Jenny S. Henkel, József I. Engelhardt, J. Engelhardt, Scott R. McKercher, Weihua Zhao, Qin Xiao, Jinghong Wang and Albert Yen and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Neurology.

In The Last Decade

László Siklós

47 papers receiving 3.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Wild-type microglia extend survival in PU.1 knockout mice with familial amyotrophic lateral sclerosis

2006 582 citations David R. Beers, Jenny S. Henkel et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
László Siklós Hungary	27	1.7k	1.0k	905	759	603	48	3.1k
Makiko Nagai Japan	27	2.1k 1.2×	788 0.8×	1.2k 1.3×	1.0k 1.3×	803 1.3×	61	3.4k
Patricia Cassina Uruguay	28	1.3k 0.8×	711 0.7×	1.1k 1.3×	624 0.8×	641 1.1×	45	2.9k
Thomas Philips United States	11	1.6k 0.9×	693 0.7×	870 1.0×	858 1.1×	465 0.8×	15	2.5k
Carlos J. Miranda United States	19	1.2k 0.7×	675 0.7×	1.4k 1.5×	693 0.9×	754 1.3×	35	2.8k
Christelle Guégan France	21	1.3k 0.8×	612 0.6×	1.3k 1.4×	457 0.6×	813 1.3×	25	2.8k
Cédric Raoul France	28	1.2k 0.7×	359 0.4×	1.3k 1.4×	664 0.9×	688 1.1×	70	2.7k
Hidefumi Ito Japan	38	1.7k 1.0×	1.0k 1.0×	1.5k 1.6×	564 0.7×	838 1.4×	145	3.9k
Nadia D’Ambrosi Italy	32	757 0.4×	940 0.9×	1.0k 1.1×	280 0.4×	595 1.0×	56	3.2k
Smita Saxena Switzerland	22	1.4k 0.8×	366 0.4×	1.5k 1.7×	774 1.0×	1.1k 1.8×	40	3.6k
Genevíève Gowing United States	27	1.1k 0.6×	1.9k 1.9×	1.3k 1.4×	748 1.0×	881 1.5×	37	4.1k

Countries citing papers authored by László Siklós

Since Specialization

Citations

This map shows the geographic impact of László Siklós's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by László Siklós with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites László Siklós more than expected).

Fields of papers citing papers by László Siklós

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by László Siklós. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by László Siklós. The network helps show where László Siklós may publish in the future.

Co-authorship network of co-authors of László Siklós

This figure shows the co-authorship network connecting the top 25 collaborators of László Siklós. A scholar is included among the top collaborators of László Siklós based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with László Siklós. László Siklós is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Polgár, Tamás F., et al.. (2025). Photobleaching alters the morphometric analysis of fluorescently labeled neurons and microglial cells. Pathology & Oncology Research. 31. 1612087–1612087.

Mészáros, Mária, Lóránd Kiss, Zoltán Kóta, et al.. (2018). Niosomes decorated with dual ligands targeting brain endothelial transporters increase cargo penetration across the blood-brain barrier. European Journal of Pharmaceutical Sciences. 123. 228–240. 44 indexed citations

Patai, Roland, Massimo Tortarolo, Caterina Bendotti, et al.. (2017). Presymptomatically applied AMPA receptor antagonist prevents calcium increase in vulnerable type of motor axon terminals of mice modeling amyotrophic lateral sclerosis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1863(7). 1739–1748. 6 indexed citations

Patai, Roland, et al.. (2016). Calcium in the pathomechanism of amyotrophic lateral sclerosis – Taking center stage?. Biochemical and Biophysical Research Communications. 483(4). 1031–1039. 17 indexed citations

Baka, Judith, Nikoletta Dobos, László Siklós, et al.. (2016). Stress induces equivalent remodeling of hippocampal spine synapses in a simulated postpartum environment and in a female rat model of major depression. Neuroscience. 343. 384–397. 22 indexed citations

Hoyk, Zsófia, et al.. (2014). Aromatase and estrogen receptor beta expression in the rat olfactory bulb: Neuroestrogen action in the first relay station of the olfactory pathway?. Acta Neurobiologiae Experimentalis. 74(1). 1–14. 15 indexed citations

Tortarolo, Massimo, et al.. (2011). Talampanel reduces the level of motoneuronal calcium in transgenic mutant SOD1 mice only if applied presymptomatically. Amyotrophic Lateral Sclerosis. 12(5). 340–344. 29 indexed citations

Engelhardt, József I., et al.. (2009). Hypoglossal motor neurons display a reduced calcium increase after axotomy in mice with upregulated parvalbumin. The Journal of Comparative Neurology. 518(11). 1946–1961. 8 indexed citations

Hamar, J., et al.. (2006). Capsaicin delays regeneration of the neuromuscular junction of rat extensor digitorum longus muscle after ischemia. Muscle & Nerve. 33(4). 556–567. 1 indexed citations

10.

Smith, R G, et al.. (2005). Sublethal dose of 4-hydroxynonenal reduces intracellular calcium in surviving motor neurons in vivo. Acta Neuropathologica. 109(6). 567–575. 12 indexed citations

11.

Kim, Seung Hyun, Jenny S. Henkel, David R. Beers, et al.. (2003). PARP Expression Is Increased in Astrocytes but Decreased in Motor Neurons in the Spinal Cord of Sporadic ALS Patients. Journal of Neuropathology & Experimental Neurology. 62(1). 88–103. 48 indexed citations

12.

Henkel, Jenny S., J. Engelhardt, László Siklós, et al.. (2003). Presence of dendritic cells, MCP‐1, and activated microglia/macrophages in amyotrophic lateral sclerosis spinal cord tissue. Annals of Neurology. 55(2). 221–235. 439 indexed citations

13.

Adalbert, Róbert, József I. Engelhardt, & László Siklós. (2002). DL -Homocysteic acid application disrupts calcium homeostasis and induces degeneration of spinal motor neurons in vivo. Acta Neuropathologica. 103(5). 428–436. 30 indexed citations

14.

Mosier, Dennis R., László Siklós, Maria E. Alexianu, et al.. (2002). Immunoglobulin Fcγ receptor promotes immunoglobulin uptake, immunoglobulin‐mediated calcium increase, and neurotransmitter release in motor neurons. Journal of Neuroscience Research. 69(1). 110–116. 65 indexed citations

15.

Beers, David R., László Siklós, Maria E. Alexianu, et al.. (2001). Parvalbumin overexpression alters immune‐mediated increases in intracellular calcium, and delays disease onset in a transgenic model of familial amyotrophic lateral sclerosis. Journal of Neurochemistry. 79(3). 499–509. 134 indexed citations

16.

Siklós, László, József I. Engelhardt, Róbert Adalbert, & Stanley H. Appel. (1999). Calcium-containing endosomes at oculomotor terminals in animal models of ALS. Neuroreport. 10(12). 2539–2545. 9 indexed citations

17.

Engelhardt, József I., László Siklós, & Stanley H. Appel. (1997). Altered Calcium Homeostasis and Ultrastructure in Motoneurons of Mice Caused by Passively Transferred Anti-motoneuronal IgG. Journal of Neuropathology & Experimental Neurology. 56(1). 21–39. 32 indexed citations

18.

Siklós, László, József I. Engelhardt, Yadollah Harati, et al.. (1996). Ultrastructural evidence for altered calcium in motor nerve terminals in amyotrophc lateral sclerosis. Annals of Neurology. 39(2). 203–216. 246 indexed citations

19.

Engelhardt, József I., et al.. (1995). Antibodies to calcium channels from ALS patients passively transferred to mice selectively increase intracellular calcium and induce ultrastructural changes in motoneurons. Synapse. 20(3). 185–199. 75 indexed citations

20.

Wolff, Joachim, József Toldi, László Siklós, O. Fehér, & Ferenc Joó. (1992). Neonatal enucleation induces correlated modification in sensory responsive areas and pial angioarchitecture of the parietal and occipital cortex of albino rats. The Journal of Comparative Neurology. 317(2). 187–194. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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