Sarah D. Diermeier
- Molecular Biology top 5%
- Cancer Research top 1%
- Oncology
- Immunology
- Genetics
- Co-authors
- David L. SpectorGayatri ArunRakesh N. VeeduGernot LängstHaoyu XiongEdi BrogiAdrian R. KrainerJohn E. Wilkinson
- Topics
- Cancer-related molecular mechanisms research (17 papers)RNA Research and Splicing (15 papers)RNA modifications and cancer (12 papers)
- Partner nations
- New ZealandUnited StatesGermany
In The Last Decade
Sarah D. Diermeier
31 papers receiving 1.9k citations
Hit Papers
Peers
Comparison fields: 5 of 86
- Molecular Biology 1.7k
- Cancer Research 1.2k
- Oncology 143
- Immunology 110
- Genetics 108
Countries citing papers authored by Sarah D. Diermeier
This map shows the geographic impact of Sarah D. Diermeier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah D. Diermeier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah D. Diermeier more than expected).
Fields of papers citing papers by Sarah D. Diermeier
This network shows the impact of papers produced by Sarah D. Diermeier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah D. Diermeier. The network helps show where Sarah D. Diermeier may publish in the future.
Co-authorship network of co-authors of Sarah D. Diermeier
This figure shows the co-authorship network connecting the top 25 collaborators of Sarah D. Diermeier. A scholar is included among the top collaborators of Sarah D. Diermeier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah D. Diermeier. Sarah D. Diermeier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 1 | |
| 3 | 4 | |
| 4 | 4 | |
| 5 | 1 | |
| 6 | 29 | |
| 7 | 72 | |
| 8 | Therapeutic Targeting of Long Non-Coding RNAs in Cancerbreakdown → | 454 |
| 9 | 11 | |
| 10 | 51 | |
| 11 | 48 | |
| 12 | 30 | |
| 13 | Differentiation of mammary tumors and reduction in metastasis upon Malat1 lncRNA lossbreakdown → | 468 |
| 14 | 31 | |
| 15 | 18 | |
| 16 | 1 | |
| 17 | 1 | |
| 18 | 97 | |
| 19 | 148 | |
| 20 | 83 |
About Sarah D. Diermeier
Sarah D. Diermeier is a scholar working on Cancer Research, Molecular Biology and Pulmonary and Respiratory Medicine, having authored 31 papers that have together received 1.9k indexed citations. Recurring topics across this work include Cancer-related molecular mechanisms research (17 papers), RNA Research and Splicing (15 papers) and RNA modifications and cancer (12 papers). The work is most often cited by research in Cancer Research (1.2k citations), Molecular Biology (1.7k citations) and Oncology (143 citations). Sarah D. Diermeier has collaborated with scholars based in New Zealand, United States and Germany. Frequent co-authors include David L. Spector, Gayatri Arun, Rakesh N. Veedu, Gernot Längst, Haoyu Xiong, Edi Brogi, Adrian R. Krainer, John E. Wilkinson, Mikala Egeblad and Youngsoo Kim. Their work appears in journals such as Nucleic Acids Research, Journal of Biological Chemistry and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.