Manuel Beltrán

2.2k total citations · 1 hit paper
24 papers, 1.6k citations indexed

About

Manuel Beltrán is a scholar working on Molecular Biology, Cancer Research and Surgery. According to data from OpenAlex, Manuel Beltrán has authored 24 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 11 papers in Cancer Research and 2 papers in Surgery. Recurrent topics in Manuel Beltrán's work include RNA Research and Splicing (10 papers), RNA modifications and cancer (9 papers) and Cancer-related molecular mechanisms research (8 papers). Manuel Beltrán is often cited by papers focused on RNA Research and Splicing (10 papers), RNA modifications and cancer (9 papers) and Cancer-related molecular mechanisms research (8 papers). Manuel Beltrán collaborates with scholars based in Spain, United Kingdom and Italy. Manuel Beltrán's co-authors include Antonio Garcı́a de Herreros, Félix Bonilla, Cristina Peña, Isabel Puig, Ana Belén Cid Álvarez, Raúl Peña, Richard G. Jenner, Sandra Peiró, Alex Frías and Natàlia Dave and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Manuel Beltrán

23 papers receiving 1.6k citations

Hit Papers

A natural antisense transcript regulates Zeb2/Sip1 gene e... 2008 2026 2014 2020 2008 100 200 300 400 500

Peers

Manuel Beltrán
Lovorka Stojic United Kingdom
Peng Yao United States
Fabio Carrozzino Switzerland
Gregg J. Johannes United States
Jian Chai China
Yadi Wu China
SiDe Li United States
Arthur K. Ching Hong Kong
Lovorka Stojic United Kingdom
Manuel Beltrán
Citations per year, relative to Manuel Beltrán Manuel Beltrán (= 1×) peers Lovorka Stojic

Countries citing papers authored by Manuel Beltrán

Since Specialization
Citations

This map shows the geographic impact of Manuel Beltrán's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Manuel Beltrán with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Manuel Beltrán more than expected).

Fields of papers citing papers by Manuel Beltrán

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Manuel Beltrán. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Manuel Beltrán. The network helps show where Manuel Beltrán may publish in the future.

Co-authorship network of co-authors of Manuel Beltrán

This figure shows the co-authorship network connecting the top 25 collaborators of Manuel Beltrán. A scholar is included among the top collaborators of Manuel Beltrán based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Manuel Beltrán. Manuel Beltrán is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Beltrán, Manuel, et al.. (2025). Direct circRNA-mRNA Binding Controls mRNA Fate: A New Mechanism for circRNAs. Non-Coding RNA. 11(4). 53–53.
2.
Rossi, Francesca, et al.. (2024). BRCA1 levels and DNA-damage response are controlled by the competitive binding of circHIPK3 or FMRP to the BRCA1 mRNA. Molecular Cell. 84(21). 4079–4094.e10. 9 indexed citations
3.
Rossi, Francesca, Dario Dattilo, Andrea Giuliani, et al.. (2023). CircAFF1 Is a Circular RNA with a Role in Alveolar Rhabdomyosarcoma Cell Migration. Biomedicines. 11(7). 1893–1893. 1 indexed citations
4.
Dattilo, Dario, Gaia Di Timoteo, Andrea Giuliani, et al.. (2023). The m6A reader YTHDC1 and the RNA helicase DDX5 control the production of rhabdomyosarcoma-enriched circRNAs. Nature Communications. 14(1). 1898–1898. 33 indexed citations
5.
Khandelwal, Garima, Joanne Muter, Keijo Viiri, et al.. (2022). JAZF1-SUZ12 dysregulates PRC2 function and gene expression during cell differentiation. Cell Reports. 39(9). 110889–110889. 11 indexed citations
6.
Skalska, Lenka, Manuel Beltrán, Saulius Lukauskas, et al.. (2021). Nascent RNA antagonizes the interaction of a set of regulatory proteins with chromatin. Molecular Cell. 81(14). 2944–2959.e10. 26 indexed citations
7.
Rossi, Francesca, Manuel Beltrán, Alessio Colantoni, et al.. (2021). Circular RNA ZNF609/CKAP5 mRNA interaction regulates microtubule dynamics and tumorigenicity. Molecular Cell. 82(1). 75–89.e9. 71 indexed citations
8.
Beltrán, Manuel, N. Justin, Garima Khandelwal, et al.. (2019). G-tract RNA removes Polycomb repressive complex 2 from genes. Nature Structural & Molecular Biology. 26(10). 899–909. 81 indexed citations
9.
Skalska, Lenka, Manuel Beltrán, Jernej Ule, & Richard G. Jenner. (2017). Regulatory feedback from nascent RNA to chromatin and transcription. Nature Reviews Molecular Cell Biology. 18(5). 331–337. 54 indexed citations
10.
Beltrán, Manuel, Christopher M. Yates, Lenka Skalska, et al.. (2016). The interaction of PRC2 with RNA or chromatin is mutually antagonistic. Genome Research. 26(7). 896–907. 170 indexed citations
11.
Beltrán, Manuel & Antonio Garcı́a de Herreros. (2016). Antisense non-coding RNAs and regulation of gene transcription. Transcription. 7(2). 39–43. 11 indexed citations
12.
Ibáñez‐Costa, Alejandro, Manuel D. Gahete, Esther Rivero‐Cortés, et al.. (2015). In1-ghrelin splicing variant is overexpressed in pituitary adenomas and increases their aggressive features. Scientific Reports. 5(1). 8714–8714. 46 indexed citations
13.
Beltrán, Manuel, et al.. (2015). Splicing of a non-coding antisense transcript controls LEF1 gene expression. Nucleic Acids Research. 43(12). 5785–5797. 8 indexed citations
14.
García-Baquero, Rodrigo, et al.. (2014). Methylation of tumor suppressor genes in a novel panel predicts clinical outcome in paraffin-embedded bladder tumors. Tumor Biology. 35(6). 5777–5786. 21 indexed citations
15.
Dave, Natàlia, Sandra Guaita‐Esteruelas, Susana Gutarra, et al.. (2011). Functional Cooperation between Snail1 and Twist in the Regulation of ZEB1 Expression during Epithelial to Mesenchymal Transition. Journal of Biological Chemistry. 286(14). 12024–12032. 238 indexed citations
16.
Viñas-Castells, Rosa, Manuel Beltrán, Irene Gómez, et al.. (2009). The Hypoxia-controlled FBXL14 Ubiquitin Ligase Targets SNAIL1 for Proteasome Degradation. Journal of Biological Chemistry. 285(6). 3794–3805. 147 indexed citations
17.
Beltrán, Manuel, Isabel Puig, Cristina Peña, et al.. (2008). A natural antisense transcript regulates Zeb2/Sip1 gene expression during Snail1-induced epithelial–mesenchymal transition. Genes & Development. 22(6). 756–769. 511 indexed citations breakdown →
18.
Soriguer, Federico, Isabel Esteva, Gemma Rojo‐Martínez, et al.. (2004). Oleic acid from cooking oils is associated with lower insulin resistance in the general population (Pizarra study). European Journal of Endocrinology. 150(1). 33–39. 54 indexed citations
19.
Galán‐Sánchez, Fátima, et al.. (1997). Onychoprotothecosis due to Prototheca wickerhamii. Mycopathologia. 137(2). 75–77. 14 indexed citations
20.
Santos, Miguel M., et al.. (1984). Effect of smoking on plasma and platelet fatty acid composition in middle-aged men. Atherosclerosis. 50(1). 53–62. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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