Richard V. Parry

5.9k citations
32 papers · 4.6k indexed · 3 hit papers · h-index 23
Topics
T-cell and B-cell Immunology (12 papers)Immune Cell Function and Interaction (11 papers)PI3K/AKT/mTOR signaling in cancer (9 papers)

In The Last Decade

Richard V. Parry

31 papers receiving 4.6k citations

Hit Papers

CTLA-4 and PD-1 Receptors Inhibit T-Cell Activation by Di...200220262010201820052002200450010001.5k

Peers

Richard V. Parry
Comparison fields: 5 of 108
  • Immunology 3.0k
  • Oncology 2.2k
  • Molecular Biology 1.3k
  • Cancer Research 440
  • Epidemiology 290
Replace Masahisa Jinushi with:
Masahisa Jinushi Japan
T. Kevin Howcroft United States
Matteo Bellone Italy
Andrea Schietinger United States
Andrew M. Intlekofer United States
Tatiana Akimova United States
Filippo Veglia United States
Jürgen Müllberg Germany
Paolo Serafini United States
Jeong Kim United States
Richard V. Parry relative to Masahisa Jinushi Japan Masahisa Jinushi's profile →
Citations per field
00.5×1.5×
Masahisa Jinushi · 1×
Citations per year

Countries citing papers authored by Richard V. Parry

Since Specialization
Citations

This map shows the geographic impact of Richard V. Parry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard V. Parry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard V. Parry more than expected).

Fields of papers citing papers by Richard V. Parry

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Richard V. Parry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard V. Parry. The network helps show where Richard V. Parry may publish in the future.

Co-authorship network of co-authors of Richard V. Parry

This figure shows the co-authorship network connecting the top 25 collaborators of Richard V. Parry. A scholar is included among the top collaborators of Richard V. Parry based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Richard V. Parry. Richard V. Parry is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 26
3 32
4 58
5 51
6 54
7 26
8 20
9 38
10
CTLA-4 and PD-1 Receptors Inhibit T-Cell Activation by Distinct Mechanismsbreakdown →
1541
11 29
12
SHP-1 and SHP-2 Associate with Immunoreceptor Tyrosine-Based Switch Motif of Programmed Death 1 upon Primary Human T Cell Stimulation, but Only Receptor Ligation Prevents T Cell Activationbreakdown →
927
13 131
14
The CD28 Signaling Pathway Regulates Glucose Metabolismbreakdown →
1134
15 30
16 105
17 10
18 23
19 24
20 29

About Richard V. Parry

Richard V. Parry is a scholar working on Immunology, Genetics and Toxicology, having authored 32 papers that have together received 4.6k indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (12 papers), Immune Cell Function and Interaction (11 papers) and PI3K/AKT/mTOR signaling in cancer (9 papers). The work is most often cited by research in Immunology (3.0k citations), Oncology (2.2k citations) and Cancer Research (440 citations). Richard V. Parry has collaborated with scholars based in United Kingdom, United States and France. Frequent co-authors include James L. Riley, Jens M. Chemnitz, Carl H. June, Kenneth A. Frauwirth, Craig B. Thompson, Kim E. Nichols, Sumire Kobayashi, Inbal Braunstein, Peter S. Linsley and Anthony R. Lanfranco. Their work appears in journals such as Journal of Biological Chemistry, Blood and Immunity.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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