Richard L. Maas
- Molecular Biology top 0.2%
- Genetics top 0.2%
- Oral Surgery top 0.2%
- Immunology top 2%
- Cell Biology top 0.5%
- Co-authors
- Ichiro SatokataMarianna BeiGail V. BensonJonathan A. EpsteinLisa I. JepealPin‐Xian XuDavid S. WaltonAlan Brash
- Topics
- Developmental Biology and Gene Regulation (29 papers)dental development and anomalies (27 papers)Connexins and lens biology (19 papers)
- Partner nations
- United StatesUnited KingdomJapan
In The Last Decade
Richard L. Maas
136 papers receiving 16.1k citations
Hit Papers
Peers
Comparison fields: 5 of 167
- Molecular Biology 11.8k
- Genetics 4.2k
- Oral Surgery 1.4k
- Immunology 1.3k
- Cell Biology 1.3k
Countries citing papers authored by Richard L. Maas
This map shows the geographic impact of Richard L. Maas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard L. Maas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard L. Maas more than expected).
Fields of papers citing papers by Richard L. Maas
This network shows the impact of papers produced by Richard L. Maas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard L. Maas. The network helps show where Richard L. Maas may publish in the future.
Co-authorship network of co-authors of Richard L. Maas
This figure shows the co-authorship network connecting the top 25 collaborators of Richard L. Maas. A scholar is included among the top collaborators of Richard L. Maas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Richard L. Maas. Richard L. Maas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 19 | |
| 2 | 6 | |
| 3 | 24 | |
| 4 | 41 | |
| 5 | 84 | |
| 6 | 119 | |
| 7 | 92 | |
| 8 | 15 | |
| 9 | 42 | |
| 10 | 105 | |
| 11 | 146 | |
| 12 | 57 | |
| 13 | 50 | |
| 14 | 52 | |
| 15 | Eya protein phosphatase activity regulates Six1–Dach–Eya transcriptional effects in mammalian organogenesisbreakdown → | 506 |
| 16 | 273 | |
| 17 | Mutations of the homeobox gene MSX2 cause symmetric parietal foramina: contrasting effects of loss and gain of function mutations for skull development. | 1 |
| 18 | 220 | |
| 19 | 240 | |
| 20 | 173 |
About Richard L. Maas
Richard L. Maas is a scholar working on Genetics, Molecular Biology and Oral Surgery, having authored 138 papers that have together received 16.5k indexed citations. Recurring topics across this work include Developmental Biology and Gene Regulation (29 papers), dental development and anomalies (27 papers) and Connexins and lens biology (19 papers). The work is most often cited by research in Oral Surgery (1.4k citations), Molecular Biology (11.8k citations) and Genetics (4.2k citations). Richard L. Maas has collaborated with scholars based in United States, United Kingdom and Japan. Frequent co-authors include Ichiro Satokata, Marianna Bei, Gail V. Benson, Jonathan A. Epstein, Lisa I. Jepeal, Pin‐Xian Xu, David S. Walton, Alan Brash, Hyunjung Jade Lim and John A. Oates. Their work appears in journals such as Nature, Science and New England Journal of Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.