Richard C. Mulligan
- Genetics top 0.02%
- Virus-based gene therapy research 71
- Immunology top 0.1%
- Immunotherapy and Immune Responses 32
- Immune Cell Function and Interaction 15
- T-cell and B-cell Immunology 15
- Genetics top 0.05%
- Virus-based gene therapy research 71
- Hematology top 0.1%
- Molecular Biology top 0.05%
- RNA Interference and Gene Delivery 34
- CRISPR and Genetic Engineering 26
- Viral Infectious Diseases and Gene Expression in Insects 13
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- CAR-T cell therapy research 25
- Co-authors
- Daniel OryKatja BroseMargaret A. GoodellDavid BaltimoreG ParadisRichard S. MannDidier TronoPhilippe Gallay
- Cited by
- GeneticsImmunology
- Partner nations
- United StatesFranceBelgium
In The Last Decade
Richard C. Mulligan
200 papers receiving 39.8k citations
Hit Papers
Peers
Comparison fields: 5 of 182
- Genetics 11.9k
- Immunology 8.6k
- Genetics 4.0k
- Hematology 3.9k
- Molecular Biology 23.1k
Countries citing papers authored by Richard C. Mulligan
This map shows the geographic impact of Richard C. Mulligan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard C. Mulligan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard C. Mulligan more than expected).
Fields of papers citing papers by Richard C. Mulligan
This network shows the impact of papers produced by Richard C. Mulligan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard C. Mulligan. The network helps show where Richard C. Mulligan may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Richard C. Mulligan, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 10 | |
| 2 | 2012 | 14 | |
| 3 | 2008 | 95 | |
| 4 | 2007 | 53 | |
| 5 | 2006 | 129 | |
| 6 | 2006 | 46 | |
| 7 | 2004 | 210 | |
| 8 | 2004 | 224 | |
| 9 | 2003 | 23 | |
| 10 | 2001 | 21 | |
| 11 | 2000 | 51 | |
| 12 | 2000 | 62 | |
| 13 | 1996 | 207 | |
| 14 | 1995 | 110 | |
| 15 | 1995 | 34 | |
| 16 | 1994 | 7 | |
| 17 | 1994 | 54 | |
| 18 | 1993 | 292 | |
| 19 | 1991 | 208 | |
| 20 | 1987 | 120 |
About Richard C. Mulligan
Richard C. Mulligan is a scholar working on Genetics, Immunology and Hematology, having authored 202 papers that have together received 41.2k indexed citations. Recurring topics across this work include Virus-based gene therapy research (71 papers), RNA Interference and Gene Delivery (34 papers), Immunotherapy and Immune Responses (32 papers), CRISPR and Genetic Engineering (26 papers), CAR-T cell therapy research (25 papers), Immune Cell Function and Interaction (15 papers), T-cell and B-cell Immunology (15 papers) and Viral Infectious Diseases and Gene Expression in Insects (13 papers). The work is most often cited by research in Genetics (11.9k citations), Immunology (8.6k citations) and Genetics (4.0k citations). Richard C. Mulligan has collaborated with scholars based in United States, France and Belgium. Frequent co-authors include Daniel Ory, Katja Brose, Margaret A. Goodell, David Baltimore, G Paradis, Richard S. Mann, Didier Trono, Philippe Gallay, Inder M. Verma and Luigi Naldini. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.