Ralph P. Schneider

654 citations
9 papers · 471 indexed · h-index 7
Topics
Pluripotent Stem Cells Research (3 papers)Telomeres, Telomerase, and Senescence (3 papers)Mesenchymal stem cell research (2 papers)

In The Last Decade

Ralph P. Schneider

9 papers receiving 468 citations

Peers

Ralph P. Schneider
Comparison fields: 5 of 66
  • Molecular Biology 305
  • Physiology 157
  • Endocrinology, Diabetes and Metabolism 96
  • Oncology 71
  • Genetics 63
Replace Francesco Antonica with:
Francesco Antonica Italy
Jengmin Kang South Korea
Scott Haston United Kingdom
Siham Yennek Denmark
Omar El-Mounayri Canada
Leonidas Panousopoulos United Kingdom
Nathan C. Jones United States
Tina Beckmann Germany
Darwin S. Dichmann United States
Xike Zhu China
Ralph P. Schneider relative to Francesco Antonica Italy Francesco Antonica's profile →
Citations per field
00.5×4.8×
Francesco Antonica · 1×
Citations per year

Countries citing papers authored by Ralph P. Schneider

Since Specialization
Citations

This map shows the geographic impact of Ralph P. Schneider's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ralph P. Schneider with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ralph P. Schneider more than expected).

Fields of papers citing papers by Ralph P. Schneider

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ralph P. Schneider. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ralph P. Schneider. The network helps show where Ralph P. Schneider may publish in the future.

Co-authorship network of co-authors of Ralph P. Schneider

This figure shows the co-authorship network connecting the top 25 collaborators of Ralph P. Schneider. A scholar is included among the top collaborators of Ralph P. Schneider based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ralph P. Schneider. Ralph P. Schneider is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
#WorkIndexed citations
1 3
2 1
3 37
4 19
5 45
6 70
7 91
8 131
9 74

About Ralph P. Schneider

Ralph P. Schneider is a scholar working on Aging, Genetics and Physiology, having authored 9 papers that have together received 471 indexed citations. Recurring topics across this work include Pluripotent Stem Cells Research (3 papers), Telomeres, Telomerase, and Senescence (3 papers) and Mesenchymal stem cell research (2 papers). The work is most often cited by research in Aging (55 citations), Physiology (157 citations) and Genetics (63 citations). Ralph P. Schneider has collaborated with scholars based in Spain, United States and United Kingdom. Frequent co-authors include Marı́a A. Blasco, Sagrario Ortega, Elisa Varela, Gonzalo Goméz-López, David G. Pisano, Miguel Foronda, Cynthia L. Andoniadou, Carles Gaston‐Massuet, Rukmini Reddy and Juan Pedro Martı́nez-Barberá. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nature Communications and The EMBO Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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