Nathan C. Jones

669 total citations
9 papers, 549 citations indexed

About

Nathan C. Jones is a scholar working on Molecular Biology, Cell Biology and Aging. According to data from OpenAlex, Nathan C. Jones has authored 9 papers receiving a total of 549 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Cell Biology and 3 papers in Aging. Recurrent topics in Nathan C. Jones's work include Muscle Physiology and Disorders (6 papers), Genetics, Aging, and Longevity in Model Organisms (3 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). Nathan C. Jones is often cited by papers focused on Muscle Physiology and Disorders (6 papers), Genetics, Aging, and Longevity in Model Organisms (3 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). Nathan C. Jones collaborates with scholars based in United States. Nathan C. Jones's co-authors include Bradley B. Olwin, Yu. V. Fedorov, R. Scott Rosenthal, DDW Cornelison, Arthur J. Kudla, Kari Clase, Michael K. Matheny, Elisabeth R. Barton, Hanqin Lei and Michael E. Matheny and has published in prestigious journals such as The Journal of Cell Biology, Molecular and Cellular Biology and The FASEB Journal.

In The Last Decade

Nathan C. Jones

8 papers receiving 538 citations

Peers

Nathan C. Jones

MB

PHYSI

SURGE

CB

GENET

Namrata Mastey United States

Aurore L’honoré France

Virginie Jacquemin France

Léo Machado France

Adam B. Cadwallader United States

Elija Schirwis France

Sharon Soueid‐Baumgarten Israel

Brendan Evano France

Annarita Scaramozza Italy

Audrey Der Vartanian France

Namrata Mastey United States

Nathan C. Jones

427 ×0.9

MB

127 ×1.0

PHYSI

78 ×0.7

SURGE

81 ×1.0

CB

69 ×1.0

GENET

Citations per year, relative to Nathan C. Jones Nathan C. Jones (= 1×) peers Namrata Mastey

Countries citing papers authored by Nathan C. Jones

Since Specialization

Citations

This map shows the geographic impact of Nathan C. Jones's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan C. Jones with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan C. Jones more than expected).

Fields of papers citing papers by Nathan C. Jones

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan C. Jones. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan C. Jones. The network helps show where Nathan C. Jones may publish in the future.

Co-authorship network of co-authors of Nathan C. Jones

This figure shows the co-authorship network connecting the top 25 collaborators of Nathan C. Jones. A scholar is included among the top collaborators of Nathan C. Jones based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nathan C. Jones. Nathan C. Jones is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Matheny, Michael K., et al.. (2021). Deletion of muscle Igf1 exacerbates disuse atrophy weakness in mice. Journal of Applied Physiology. 131(3). 881–894. 4 indexed citations

2.

Matheny, Michael E., et al.. (2019). Loss of Muscle IGF‐I Production Delays Functional Recovery of Skeletal Muscle Following Disuse. The FASEB Journal. 33(S1). 1 indexed citations

3.

Jones, Nathan C.. (2014). Charting a New Course. Collections A Journal for Museum and Archives Professionals. 10(2). 218–229.

4.

Jones, Nathan C., et al.. (2005). The p38α/β MAPK functions as a molecular switch to activate the quiescent satellite cell. The Journal of Cell Biology. 169(1). 105–116. 206 indexed citations

5.

Fedorov, Yu. V., Nathan C. Jones, & Bradley B. Olwin. (2002). Atypical Protein Kinase Cs Are the Ras Effectors That Mediate Repression of Myogenic Satellite Cell Differentiation. Molecular and Cellular Biology. 22(4). 1140–1149. 12 indexed citations

6.

Jones, Nathan C., Yu. V. Fedorov, R. Scott Rosenthal, & Bradley B. Olwin. (2001). ERK1/2 is required for myoblast proliferation but is dispensable for muscle gene expression and cell fusion. Journal of Cellular Physiology. 186(1). 104–115. 7 indexed citations

7.

Jones, Nathan C., Yu. V. Fedorov, R. Scott Rosenthal, & Bradley B. Olwin. (2000). ERK1/2 is required for myoblast proliferation but is dispensable for muscle gene expression and cell fusion. Journal of Cellular Physiology. 186(1). 104–115. 226 indexed citations

8.

Kudla, Arthur J., et al.. (1998). The FGF Receptor–1 Tyrosine Kinase Domain Regulates Myogenesis but Is Not Sufficient to Stimulate Proliferation. The Journal of Cell Biology. 142(1). 241–250. 33 indexed citations

9.

Fedorov, Yu. V., Nathan C. Jones, & Bradley B. Olwin. (1998). Regulation of Myogenesis by Fibroblast Growth Factors Requires Beta-Gamma Subunits of Pertussis Toxin-Sensitive G Proteins. Molecular and Cellular Biology. 18(10). 5780–5787. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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