Rafał Pawliński

8.5k total citations · 1 hit paper
103 papers, 6.7k citations indexed

About

Rafał Pawliński is a scholar working on Hematology, Genetics and Molecular Biology. According to data from OpenAlex, Rafał Pawliński has authored 103 papers receiving a total of 6.7k indexed citations (citations by other indexed papers that have themselves been cited), including 72 papers in Hematology, 41 papers in Genetics and 20 papers in Molecular Biology. Recurrent topics in Rafał Pawliński's work include Blood Coagulation and Thrombosis Mechanisms (55 papers), Hemoglobinopathies and Related Disorders (23 papers) and Hemophilia Treatment and Research (16 papers). Rafał Pawliński is often cited by papers focused on Blood Coagulation and Thrombosis Mechanisms (55 papers), Hemoglobinopathies and Related Disorders (23 papers) and Hemophilia Treatment and Research (16 papers). Rafał Pawliński collaborates with scholars based in United States, Germany and Poland. Rafał Pawliński's co-authors include Nigel Mackman, Erica Sparkenbaugh, Michael Tencati, Brian Pedersen, Nigel S. Key, Yuji Yamanishi, Gary S. Firestein, Victor Nizet, Björn E. Clausen and Napoleone Ferrara and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Rafał Pawliński

100 papers receiving 6.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

HIF-1α Is Essential for Myeloid Cell-Mediated Inflammation

2003 1.6k citations Thorsten Cramer, Yuji Yamanishi et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Rafał Pawliński United States	40	2.2k	2.1k	1.8k	1.2k	965	103	6.7k
Henri H. Versteeg Netherlands	37	1.7k 0.8×	2.4k 1.1×	1.1k 0.6×	706 0.6×	827 0.9×	129	7.1k
L. Vijaya Mohan Rao United States	45	1.7k 0.8×	4.2k 2.0×	1.0k 0.6×	689 0.6×	1.4k 1.4×	181	7.5k
C. Arnold Spek Netherlands	41	1.7k 0.8×	1.5k 0.7×	946 0.5×	485 0.4×	547 0.6×	165	5.1k
Éric Boilard Canada	45	3.5k 1.6×	1.6k 0.8×	1.7k 0.9×	1.1k 1.0×	307 0.3×	125	7.1k
Bernd Engelmann Germany	32	1.6k 0.8×	1.7k 0.8×	1.6k 0.9×	338 0.3×	528 0.5×	69	5.7k
José A. Fernández United States	42	937 0.4×	3.1k 1.5×	835 0.5×	529 0.5×	821 0.9×	151	5.8k
Wolfgang Bergmeier United States	47	1.5k 0.7×	3.4k 1.6×	1.5k 0.8×	329 0.3×	478 0.5×	139	6.8k
Peter J. Lenting France	57	1.8k 0.9×	5.9k 2.8×	2.2k 1.2×	334 0.3×	1.5k 1.6×	208	10.0k
Philipp von Hundelshausen Germany	35	1.4k 0.6×	1.4k 0.7×	2.6k 1.4×	407 0.4×	264 0.3×	61	5.7k
Benjamin T. Kile Australia	50	4.6k 2.1×	1.9k 0.9×	2.8k 1.5×	671 0.6×	615 0.6×	137	8.3k

Countries citing papers authored by Rafał Pawliński

Since Specialization

Citations

This map shows the geographic impact of Rafał Pawliński's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rafał Pawliński with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rafał Pawliński more than expected).

Fields of papers citing papers by Rafał Pawliński

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rafał Pawliński. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rafał Pawliński. The network helps show where Rafał Pawliński may publish in the future.

Co-authorship network of co-authors of Rafał Pawliński

This figure shows the co-authorship network connecting the top 25 collaborators of Rafał Pawliński. A scholar is included among the top collaborators of Rafał Pawliński based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rafał Pawliński. Rafał Pawliński is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Grover, Steven P., Chatphatai Moonla, Michael W. Henderson, et al.. (2025). Addressing the pathophysiology of venous thrombosis and chronic kidney disease in sickle cell trait using a mouse model. Blood Advances. 9(11). 2709–2721. 1 indexed citations

Lee, Robert H., Paul Y. Kim, Rafał Pawliński, et al.. (2024). 4D intravital imaging studies identify platelets as the predominant cellular procoagulant surface in a mouse hemostasis model. Blood. 144(10). 1116–1126. 7 indexed citations

Wan, Jun, Anton Ilich, Prakash Saha, et al.. (2024). Plasma kallikrein supports FXII-independent thrombin generation in mouse whole blood. Blood Advances. 8(12). 3045–3057. 1 indexed citations

Ramadas, Nirupama, J. J. Dutton, Camille Faës, et al.. (2024). Biased agonism of protease-activated receptor-1 regulates thromboinflammation in murine sickle cell disease. Blood Advances. 8(12). 3272–3283. 2 indexed citations

Ellsworth, Patrick, Anton Ilich, Chatphatai Moonla, et al.. (2024). Hypertonicity and/or acidosis induce marked rheological changes under hypoxic conditions in sickle trait red blood cells. British Journal of Haematology. 205(4). 1565–1569. 3 indexed citations

Henderson, Michael W., Erica Sparkenbaugh, Shaobin Wang, et al.. (2021). Plasmin-mediated cleavage of high-molecular-weight kininogen contributes to acetaminophen-induced acute liver failure. Blood. 138(3). 259–272. 20 indexed citations

Ansari, Junaid, Elena Y. Senchenkova, Shantel Vital, et al.. (2021). Targeting the AnxA1/Fpr2/ALX pathway regulates neutrophil function, promoting thromboinflammation resolution in sickle cell disease. Blood. 137(11). 1538–1549. 46 indexed citations

Ataga, Kenneth I., Laila Elsherif, David Wichlan, et al.. (2021). A pilot study of the effect of rivaroxaban in sickle cell anemia. Transfusion. 61(6). 1694–1698. 3 indexed citations

Henderson, Michael W., Denis F. Noubouossie, Anton Ilich, et al.. (2020). Protease: Serpin complexes to assess contact system and intrinsic pathway activation. Research and Practice in Thrombosis and Haemostasis. 4(5). 789–798. 9 indexed citations

10.

Xu, Yongmei, et al.. (2020). Synthetic anticoagulant heparan sulfate attenuates liver ischemia reperfusion injury. Scientific Reports. 10(1). 17187–17187. 19 indexed citations

11.

Noubouossie, Denis F., Michael W. Henderson, Micah J. Mooberry, et al.. (2020). Red blood cell microvesicles activate the contact system, leading to factor IX activation via 2 independent pathways. Blood. 135(10). 755–765. 66 indexed citations

12.

Sparkenbaugh, Erica, Małgorzata Kasztan, Michael W. Henderson, et al.. (2020). High molecular weight kininogen contributes to early mortality and kidney dysfunction in a mouse model of sickle cell disease. Journal of Thrombosis and Haemostasis. 18(9). 2329–2340. 9 indexed citations

13.

Faës, Camille, Anton Ilich, Erica Sparkenbaugh, et al.. (2019). Red blood cells modulate structure and dynamics of venous clot formation in sickle cell disease. Blood. 133(23). 2529–2541. 52 indexed citations

14.

Antoniak, Silvio, et al.. (2018). Protease-activated receptor 1 activation enhances doxorubicin-induced cardiotoxicity. Journal of Molecular and Cellular Cardiology. 122. 80–87. 21 indexed citations

15.

Faës, Camille, Erica Sparkenbaugh, & Rafał Pawliński. (2018). Hypercoagulable state in sickle cell disease. Clinical Hemorheology and Microcirculation. 68(2-3). 301–318. 32 indexed citations

16.

Xu, Yongmei, Kasemsiri Chandarajoti, Xing Zhang, et al.. (2017). Synthetic oligosaccharides can replace animal-sourced low–molecular weight heparins. Science Translational Medicine. 9(406). 87 indexed citations

17.

Magnus, Nathalie, Delphine Garnier, Brian Meehan, et al.. (2014). Tissue factor expression provokes escape from tumor dormancy and leads to genomic alterations. Proceedings of the National Academy of Sciences. 111(9). 3544–3549. 86 indexed citations

18.

Antoniak, Silvio, A. Phillip Owens, Julie Williams, et al.. (2013). PAR-1 contributes to the innate immune response during viral infection. Journal of Clinical Investigation. 123(3). 1310–1322. 110 indexed citations

19.

Luyendyk, James P., Gernot Schabbauer, Michael Tencati, et al.. (2008). Genetic Analysis of the Role of the PI3K-Akt Pathway in Lipopolysaccharide-Induced Cytokine and Tissue Factor Gene Expression in Monocytes/Macrophages. The Journal of Immunology. 180(6). 4218–4226. 221 indexed citations

20.

Cramer, Thorsten, Yuji Yamanishi, Björn E. Clausen, et al.. (2003). HIF-1α Is Essential for Myeloid Cell-Mediated Inflammation. Cell. 112(5). 645–657. 1641 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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