Pidder Jansen‐Dürr

15.2k total citations
176 papers, 9.4k citations indexed

About

Pidder Jansen‐Dürr is a scholar working on Molecular Biology, Physiology and Oncology. According to data from OpenAlex, Pidder Jansen‐Dürr has authored 176 papers receiving a total of 9.4k indexed citations (citations by other indexed papers that have themselves been cited), including 94 papers in Molecular Biology, 56 papers in Physiology and 42 papers in Oncology. Recurrent topics in Pidder Jansen‐Dürr's work include Telomeres, Telomerase, and Senescence (41 papers), Cancer-related Molecular Pathways (37 papers) and Virus-based gene therapy research (28 papers). Pidder Jansen‐Dürr is often cited by papers focused on Telomeres, Telomerase, and Senescence (41 papers), Cancer-related Molecular Pathways (37 papers) and Virus-based gene therapy research (28 papers). Pidder Jansen‐Dürr collaborates with scholars based in Austria, Germany and France. Pidder Jansen‐Dürr's co-authors include Werner Zwerschke, Maria Cavinato, Dimitry Spitkovsky, Eveline Hütter, E. Eigenbrodt, Sybille Mazurek, Almut Schulze, Giulio Draetta, Michele Pagano and Berthold Henglein and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Pidder Jansen‐Dürr

173 papers receiving 9.2k citations

Peers

Pidder Jansen‐Dürr
Eiji Hara Japan
Glynis Scott United States
Goberdhan P. Dimri United States
Naoko Ohtani Japan
Valery Krizhanovsky Israel
Olivia M. Pereira‐Smith United States
Gerardo Ferbeyre Canada
Derek A. Mann United Kingdom
Marco Demaria Netherlands
Alejo Efeyan Spain
Eiji Hara Japan
Pidder Jansen‐Dürr
Citations per year, relative to Pidder Jansen‐Dürr Pidder Jansen‐Dürr (= 1×) peers Eiji Hara

Countries citing papers authored by Pidder Jansen‐Dürr

Since Specialization
Citations

This map shows the geographic impact of Pidder Jansen‐Dürr's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pidder Jansen‐Dürr with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pidder Jansen‐Dürr more than expected).

Fields of papers citing papers by Pidder Jansen‐Dürr

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pidder Jansen‐Dürr. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pidder Jansen‐Dürr. The network helps show where Pidder Jansen‐Dürr may publish in the future.

Co-authorship network of co-authors of Pidder Jansen‐Dürr

This figure shows the co-authorship network connecting the top 25 collaborators of Pidder Jansen‐Dürr. A scholar is included among the top collaborators of Pidder Jansen‐Dürr based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pidder Jansen‐Dürr. Pidder Jansen‐Dürr is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Araújo, Mariana E. G. de, Jlenia Monfregola, Lukas A. Huber, et al.. (2025). TFEB Orchestrates Stress Recovery and Paves the Way for Senescence Induction in Human Dermal Fibroblasts. Aging Cell. 24(7). e70083–e70083.
2.
Cavinato, Maria, Sophia Wedel, Rafał Kozieł, et al.. (2024). Elimination of damaged mitochondria during UVB‐induced senescence is orchestrated by NIX‐dependent mitophagy. Aging Cell. 23(8). e14186–e14186. 12 indexed citations
3.
Ploner, Christian, et al.. (2024). CLCA2: A Potential Guardian against Premature Senescence and Skin Aging. Biomedicines. 12(3). 592–592.
4.
Leman, Géraldine, Petra Pavel, Martin Hermann, et al.. (2022). Mitochondrial Activity Is Upregulated in Nonlesional Atopic Dermatitis and Amenable to Therapeutic Intervention. Journal of Investigative Dermatology. 142(10). 2623–2634.e12. 25 indexed citations
5.
6.
Jansen‐Dürr, Pidder, et al.. (2022). Effects of Air Pollution on Cellular Senescence and Skin Aging. Cells. 11(14). 2220–2220. 53 indexed citations
7.
Salti, Ahmad, et al.. (2021). High Glycolytic Activity Enhances Stem Cell Reprogramming of Fahd1-KO Mouse Embryonic Fibroblasts. Cells. 10(8). 2040–2040. 3 indexed citations
8.
Wedel, Sophia, Imen Kallel, Maria Cavinato, et al.. (2021). Extraction yield optimization of Oleaster (Olea europaea var. sylvestris) fruits using response surface methodology, LC/MS profiling and evaluation of its effects on antioxidant activity and autophagy in HFF cells. Journal of Food Measurement & Characterization. 15(6). 4946–4959. 6 indexed citations
9.
Chatzistamatiou, Κimon, Theodoros Moysiadis, Εkaterini Chatzaki, et al.. (2018). Cigarette Smoking Promotes Infection of Cervical Cells by High-Risk Human Papillomaviruses, but not Subsequent E7 Oncoprotein Expression. International Journal of Molecular Sciences. 19(2). 422–422. 11 indexed citations
10.
Waltenberger, Birgit, Maria Halabalaki, Stefan Schwaiger, et al.. (2018). Novel Natural Products for Healthy Ageing from the Mediterranean Diet and Food Plants of Other Global Sources—The MediHealth Project. Molecules. 23(5). 1097–1097. 14 indexed citations
11.
Cavinato, Maria, et al.. (2017). Plant extracts and natural compounds used against UVB-induced photoaging. Biogerontology. 18(4). 499–516. 171 indexed citations
12.
Khodo, Stellor Nlandu, Udo Hasler, Frank‐Mattias Schäfer, et al.. (2016). NADPH oxidase 4 deficiency increases tubular cell death during acute ischemic reperfusion injury. Scientific Reports. 6(1). 38598–38598. 38 indexed citations
13.
Kozieł, Rafał, Georg Schäfer, Haymo Pircher, et al.. (2015). ROS signaling by NADPH oxidase 5 modulates the proliferation and survival of prostate carcinoma cells. Molecular Carcinogenesis. 55(1). 27–39. 69 indexed citations
14.
Hofer, Edith, Gerhard Laschober, Matthias Hackl, et al.. (2011). GiSAO.db: a database for ageing research. BMC Genomics. 12(1). 262–262. 2 indexed citations
15.
Heeren, Gino, Mark Rinnerthaler, H. Klinger, et al.. (2009). The mitochondrial ribosomal of the large subunit, afo1p, determines cellular longevity through mitochondrial back-signaling via TOR1. ISBN. 622–636. 1 indexed citations
16.
Lepperdinger, Günter, Peter Berger, Michael Breitenbach, et al.. (2008). The use of genetically engineered model systems for research on human aging. ISBN. 1 indexed citations
17.
Fiedler, Marc, Elisabeth Müller‐Holzner, Andreas Widschwendter, et al.. (2004). High level HPV‐16 E7 oncoprotein expression correlates with reduced pRb‐levels in cervical biopsies. The FASEB Journal. 18(10). 1120–1122. 54 indexed citations
18.
Brehm, Alexander, et al.. (1999). Synergism with Germ Line Transcription Factor Oct-4: Viral Oncoproteins Share the Ability To Mimic a Stem Cell-Specific Activity. Molecular and Cellular Biology. 19(4). 2635–2643. 43 indexed citations
19.
Zwerschke, Werner, et al.. (1996). Identification of domains required for transcriptional activation and protein dimerization in the human papillomavirus type-16 E7 protein.. PubMed. 12(1). 213–20. 28 indexed citations
20.
Spitkovsky, Dimitry, Hajo Delius, Beate Vogt, et al.. (1996). Cell Cycle Regulation of the Murine Cyclin E Gene Depends on an E2F Binding Site in the Promoter. Molecular and Cellular Biology. 16(7). 3401–3409. 218 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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