Naoko Ohtani

11.9k total citations · 4 hit papers
76 papers, 8.3k citations indexed

About

Naoko Ohtani is a scholar working on Molecular Biology, Physiology and Oncology. According to data from OpenAlex, Naoko Ohtani has authored 76 papers receiving a total of 8.3k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 31 papers in Physiology and 23 papers in Oncology. Recurrent topics in Naoko Ohtani's work include Telomeres, Telomerase, and Senescence (23 papers), Cancer-related Molecular Pathways (16 papers) and Epigenetics and DNA Methylation (14 papers). Naoko Ohtani is often cited by papers focused on Telomeres, Telomerase, and Senescence (23 papers), Cancer-related Molecular Pathways (16 papers) and Epigenetics and DNA Methylation (14 papers). Naoko Ohtani collaborates with scholars based in Japan, United Kingdom and United States. Naoko Ohtani's co-authors include Eiji Hara, Akiko Takahashi, Shin Yoshimoto, Tze Mun Loo, Kimi Yamakoshi, Seidai Sato, Yuichi Ishikawa, Hiroaki Kanda, Masahira Hattori and Kenya Honda and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Naoko Ohtani

73 papers receiving 8.2k citations

Hit Papers

4 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Obesity-induced gut microbial metabolite promotes liver cancer through senescence secretome

2013 1.7k citations Shin Yoshimoto, Hiroaki Kanda et al. profile →
An Essential Role for Senescent Cells in Optimal Wound Healing through Secretion of PDGF-AA

2014 1.4k citations Marco Demaria, Naoko Ohtani et al. Developmental Cell profile →
Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence

2001 531 citations Naoko Ohtani, Eiji Hara et al. profile →
The roles and mechanisms of senescence-associated secretory phenotype (SASP): can it be controlled by senolysis?

2022 164 citations Naoko Ohtani Inflammation and Regeneration profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Naoko Ohtani Japan	40	4.6k	3.0k	1.6k	1.5k	1.4k	76	8.3k
Jan van Deursen United States	40	4.4k 1.0×	1.8k 0.6×	1.3k 0.8×	1.2k 0.8×	1.4k 1.0×	71	8.3k
Eiji Hara Japan	50	7.1k 1.5×	3.8k 1.3×	2.7k 1.6×	1.3k 0.9×	1.8k 1.3×	108	11.3k
Gretchen J. Darlington United States	41	5.4k 1.2×	1.8k 0.6×	1.1k 0.6×	1.3k 0.9×	1.2k 0.8×	91	8.8k
Satoru Kyo Japan	57	4.7k 1.0×	2.8k 0.9×	1.9k 1.2×	811 0.5×	1.6k 1.1×	325	10.3k
Pidder Jansen‐Dürr Austria	55	5.2k 1.1×	1.9k 0.6×	2.6k 1.6×	1.5k 1.0×	1.3k 0.9×	176	9.4k
Lixin Wei China	55	4.1k 0.9×	852 0.3×	2.4k 1.4×	2.1k 1.4×	1.5k 1.0×	244	9.1k
Jelena Mann United Kingdom	36	2.0k 0.4×	1.1k 0.4×	468 0.3×	1.5k 1.0×	913 0.6×	65	5.0k
Lawrence Chan United States	52	4.0k 0.9×	1.9k 0.6×	776 0.5×	2.1k 1.4×	1.5k 1.0×	125	9.5k
Shuichi Kaneko Japan	42	3.2k 0.7×	1.0k 0.3×	885 0.5×	2.3k 1.6×	760 0.5×	191	7.1k
Derek A. Mann United Kingdom	64	4.4k 0.9×	1.3k 0.4×	1.3k 0.8×	4.0k 2.7×	1.7k 1.2×	198	12.1k

Countries citing papers authored by Naoko Ohtani

Since Specialization

Citations

This map shows the geographic impact of Naoko Ohtani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Naoko Ohtani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Naoko Ohtani more than expected).

Fields of papers citing papers by Naoko Ohtani

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Naoko Ohtani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Naoko Ohtani. The network helps show where Naoko Ohtani may publish in the future.

Co-authorship network of co-authors of Naoko Ohtani

This figure shows the co-authorship network connecting the top 25 collaborators of Naoko Ohtani. A scholar is included among the top collaborators of Naoko Ohtani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Naoko Ohtani. Naoko Ohtani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ohtani, Naoko, et al.. (2024). The connection between aging, cellular senescence and gut microbiome alterations: A comprehensive review. Aging Cell. 23(10). e14315–e14315. 22 indexed citations

Echizen, Kanae, Ryota Yamagishi, Miho Kumagai, et al.. (2024). Regular exercise suppresses steatosis‐associated liver cancer development by degrading E2F1 and c‐Myc via circadian gene upregulation. Genes to Cells. 29(11). 1012–1025.

Takasugi, Masaki, Yuya Yoshida, Frank Stein, et al.. (2024). An atlas of the aging mouse proteome reveals the features of age-related post-transcriptional dysregulation. Nature Communications. 15(1). 8520–8520. 11 indexed citations

Thủy, Lê Thị Thanh, Hoang Hai, Misako Sato, et al.. (2024). Poorly Differentiated Hepatocellular Carcinoma Cells Avoid Apoptosis by Interacting with T Cells via CD40–CD40 Ligand Linkage. American Journal Of Pathology. 194(7). 1230–1247. 1 indexed citations

Nakayama, Yoichi, Mirei Shirakashi, Ran Nakashima, et al.. (2024). Association of gut commensal translocation with autoantibody production in systemic lupus erythematosus. Lara D. Veeken. 64(5). 3104–3115.

Takasugi, Masaki, et al.. (2023). Gene expressions associated with longer lifespan and aging exhibit similarity in mammals. Nucleic Acids Research. 51(14). 7205–7219. 5 indexed citations

Kawamoto, Shimpei, Lena Takayasu, Yusuke Konishi, et al.. (2023). Bacterial induction of B cell senescence promotes age-related changes in the gut microbiota. Nature Cell Biology. 25(6). 865–876. 40 indexed citations

Ohtani, Naoko. (2022). The roles and mechanisms of senescence-associated secretory phenotype (SASP): can it be controlled by senolysis?. Inflammation and Regeneration. 42(1). 11–11. 164 indexed citations breakdown →

Sato, Misako, Yasutoshi Kido, Hayato Urushima, et al.. (2022). Nitric Oxide Derived from Cytoglobin-Deficient Hepatic Stellate Cells Causes Suppression of Cytochrome c Oxidase Activity in Hepatocytes. Antioxidants and Redox Signaling. 38(7-9). 463–479. 9 indexed citations

10.

Thủy, Lê Thị Thanh, Hoang Hai, Kenjiro Kimura, et al.. (2022). Cytoglobin attenuates pancreatic cancer growth via scavenging reactive oxygen species. Oncogenesis. 11(1). 23–23. 9 indexed citations

11.

Ohtani, Naoko & Eiji Hara. (2021). Gut‐liver axis‐mediated mechanism of liver cancer: A special focus on the role of gut microbiota. Cancer Science. 112(11). 4433–4443. 84 indexed citations

12.

Iwamoto, Masashi, Dawei Cai, Masaya Sugiyama, et al.. (2017). Functional association of cellular microtubules with viral capsid assembly supports efficient hepatitis B virus replication. Scientific Reports. 7(1). 10620–10620. 41 indexed citations

13.

Sato, Seidai, Akiko Takahashi, Yoshinori Imai, et al.. (2015). Ablation of the p16INK4a tumour suppressor reverses ageing phenotypes of klotho mice. Nature Communications. 6(1). 7035–7035. 60 indexed citations

14.

Johmura, Yoshikazu, Midori Shimada, Aya Naiki‐Ito, et al.. (2014). Necessary and Sufficient Role for a Mitosis Skip in Senescence Induction. Molecular Cell. 55(1). 73–84. 143 indexed citations

15.

Demaria, Marco, Naoko Ohtani, Sameh A. Youssef, et al.. (2014). An Essential Role for Senescent Cells in Optimal Wound Healing through Secretion of PDGF-AA. Developmental Cell. 31(6). 722–733. 1413 indexed citations breakdown →

16.

Saito, Ken, Nagio Takigawa, Naoko Ohtani, et al.. (2013). Antitumor Impact of p14ARF on Gefitinib-Resistant Non–Small Cell Lung Cancers. Molecular Cancer Therapeutics. 12(8). 1616–1628. 12 indexed citations

17.

Takahashi, Akiko, Noriko Motoi, Shin Yoshimoto, et al.. (2010). Intrinsic Cooperation between p16INK4a and p21Waf1/Cip1 in the Onset of Cellular Senescence and Tumor Suppression In vivo. Cancer Research. 70(22). 9381–9390. 94 indexed citations

18.

Ohtani, Naoko, et al.. (2009). The Properties and Application of a Novel Amphiphilic Polymer as an Active Interfacial Modifier. Journal of Society of Cosmetic Chemists of Japan. 43(4). 247–253. 1 indexed citations

19.

Murao, Kazutoshi, Yoshiaki Kubo, Naoko Ohtani, Eiji Hara, & S. Arase. (2006). Epigenetic abnormalities in cutaneous squamous cell carcinomas: frequent inactivation of the RB1/p16 and p53 pathways. British Journal of Dermatology. 155(5). 999–1005. 74 indexed citations

20.

Takahashi, Akiko, Naoko Ohtani, Kimi Yamakoshi, et al.. (2006). Mitogenic signalling and the p16INK4a–Rb pathway cooperate to enforce irreversible cellular senescence. Nature Cell Biology. 8(11). 1291–1297. 409 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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