Núria Gavaldà

640 total citations
14 papers, 551 citations indexed

About

Núria Gavaldà is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Developmental Neuroscience. According to data from OpenAlex, Núria Gavaldà has authored 14 papers receiving a total of 551 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Cellular and Molecular Neuroscience, 6 papers in Molecular Biology and 6 papers in Developmental Neuroscience. Recurrent topics in Núria Gavaldà's work include Nerve injury and regeneration (7 papers), Neurogenesis and neuroplasticity mechanisms (6 papers) and Immune Response and Inflammation (4 papers). Núria Gavaldà is often cited by papers focused on Nerve injury and regeneration (7 papers), Neurogenesis and neuroplasticity mechanisms (6 papers) and Immune Response and Inflammation (4 papers). Núria Gavaldà collaborates with scholars based in Spain, United Kingdom and Canada. Núria Gavaldà's co-authors include Jordi Alberch, Alun M. Davies, Humberto Gutiérrez, Esther Pérez‐Navarro, E. Gratacós, Gerard W. O’Keeffe, Josep M. Canals, Denis Gallagher, Sònia Marco and José J. Lucas and has published in prestigious journals such as Journal of Neuroscience, Development and Neuroscience.

In The Last Decade

Núria Gavaldà

14 papers receiving 540 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Núria Gavaldà Spain 13 334 254 108 78 70 14 551
Krithi Irmady United States 8 257 0.8× 198 0.8× 121 1.1× 42 0.5× 44 0.6× 10 465
G. Mercier France 10 189 0.6× 237 0.9× 88 0.8× 32 0.4× 75 1.1× 15 570
Adam Cornwell United States 7 152 0.5× 227 0.9× 118 1.1× 35 0.4× 46 0.7× 15 456
Yu‐Hui Wong Taiwan 11 206 0.6× 283 1.1× 101 0.9× 26 0.3× 52 0.7× 16 529
Sarah‐Ann Aelvoet Belgium 7 163 0.5× 180 0.7× 163 1.5× 105 1.3× 30 0.4× 7 442
Melanie Willingham Australia 6 201 0.6× 308 1.2× 303 2.8× 37 0.5× 91 1.3× 6 644
Rhalena A. Thomas Canada 11 212 0.6× 295 1.2× 69 0.6× 111 1.4× 28 0.4× 20 592
Fabienne Alfonsi France 9 199 0.6× 194 0.8× 124 1.1× 19 0.2× 31 0.4× 10 429
Emanuela Zuccaro Italy 9 203 0.6× 219 0.9× 92 0.9× 59 0.8× 21 0.3× 13 410
Faten El-Sabeawy United States 10 210 0.6× 182 0.7× 75 0.7× 21 0.3× 58 0.8× 11 582

Countries citing papers authored by Núria Gavaldà

Since Specialization
Citations

This map shows the geographic impact of Núria Gavaldà's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Núria Gavaldà with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Núria Gavaldà more than expected).

Fields of papers citing papers by Núria Gavaldà

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Núria Gavaldà. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Núria Gavaldà. The network helps show where Núria Gavaldà may publish in the future.

Co-authorship network of co-authors of Núria Gavaldà

This figure shows the co-authorship network connecting the top 25 collaborators of Núria Gavaldà. A scholar is included among the top collaborators of Núria Gavaldà based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Núria Gavaldà. Núria Gavaldà is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
O’Keeffe, Gerard W., Humberto Gutiérrez, Laura Howard, et al.. (2016). Region-specific role of growth differentiation factor-5 in the establishment of sympathetic innervation. Neural Development. 11(1). 4–4. 22 indexed citations
2.
Twohig, Jason P., Núria Gavaldà, Albert Giralt, et al.. (2010). Age-Dependent Maintenance of Motor Controland Corticostriatal Innervation by Death Receptor 3. Journal of Neuroscience. 30(10). 3782–3792. 20 indexed citations
3.
Gavaldà, Núria, Humberto Gutiérrez, & Alun M. Davies. (2009). Developmental Regulation of Sensory Neurite Growth by the Tumor Necrosis Factor Superfamily Member LIGHT. Journal of Neuroscience. 29(6). 1599–1607. 33 indexed citations
4.
Gavaldà, Núria, Humberto Gutiérrez, & Alun M. Davies. (2009). Developmental switch in NF-κB signalling required for neurite growth. Development. 136(20). 3405–3412. 36 indexed citations
5.
Gutiérrez, Humberto, Gerard W. O’Keeffe, Núria Gavaldà, Denis Gallagher, & Alun M. Davies. (2008). Nuclear Factor κB Signaling Either Stimulates or Inhibits Neurite Growth Depending on the Phosphorylation Status of p65/RelA. Journal of Neuroscience. 28(33). 8246–8256. 75 indexed citations
6.
Gavaldà, Núria, Esther Pérez‐Navarro, Juan Manuel García‐Martínez, et al.. (2008). Bax deficiency promotes an up-regulation of BimEL and Bak during striatal and cortical postnatal development, and after excitotoxic injury. Molecular and Cellular Neuroscience. 37(4). 663–672. 8 indexed citations
7.
Gallagher, Denis, Humberto Gutiérrez, Núria Gavaldà, et al.. (2007). Nuclear Factor-κB Activation via Tyrosine Phosphorylation of Inhibitor κB-α Is Crucial for Ciliary Neurotrophic Factor-Promoted Neurite Growth from Developing Neurons. Journal of Neuroscience. 27(36). 9664–9669. 51 indexed citations
8.
Ginés, Sílvia, Miquel Bosch, Sònia Marco, et al.. (2006). Reduced expression of the TrkB receptor in Huntington's disease mouse models and in human brain. European Journal of Neuroscience. 23(3). 649–658. 114 indexed citations
9.
Torres-Peraza, Jesús F., Josep M. Canals, Núria Gavaldà, et al.. (2006). Mice heterozygous for neurotrophin-3 display enhanced vulnerability to excitotoxicity in the striatum through increased expression of N-methyl-d-aspartate receptors. Neuroscience. 144(2). 462–471. 16 indexed citations
10.
Pérez‐Navarro, Esther, Núria Gavaldà, E. Gratacós, & Jordi Alberch. (2005). Brain‐derived neurotrophic factor prevents changes in Bcl‐2 family members and caspase‐3 activation induced by excitotoxicity in the striatum. Journal of Neurochemistry. 92(3). 678–691. 58 indexed citations
11.
García‐Martínez, Juan Manuel, Esther Pérez‐Navarro, Núria Gavaldà, & Jordi Alberch. (2005). Glial cell line-derived neurotrophic factor promotes the arborization of cultured striatal neurons through the p42/p44 mitogen-activated protein kinase pathway. Journal of Neuroscience Research. 83(1). 68–79. 20 indexed citations
12.
Gavaldà, Núria, Esther Pérez‐Navarro, E. Gratacós, Joan X. Comella, & Jordi Alberch. (2004). Differential involvement of phosphatidylinositol 3-kinase and p42/p44 mitogen activated protein kinase pathways in brain-derived neurotrophic factor-induced trophic effects on cultured striatal neurons. Molecular and Cellular Neuroscience. 25(3). 460–468. 32 indexed citations
13.
Gratacós, E., Núria Gavaldà, & Jordi Alberch. (2002). Bone morphogenetic protein‐6 is a neurotrophic factor for calbindin‐positive striatal neurons. Journal of Neuroscience Research. 70(5). 638–644. 20 indexed citations
14.
Marco, Sònia, Anna M. Canudas, Josep M. Canals, et al.. (2002). Excitatory Amino Acids Differentially Regulate the Expression of GDNF, Neurturin, and Their Receptors in the Adult Rat Striatum. Experimental Neurology. 174(2). 243–252. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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