Sílvia Ginés

4.3k total citations
60 papers, 3.1k citations indexed

About

Sílvia Ginés is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Sílvia Ginés has authored 60 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 47 papers in Cellular and Molecular Neuroscience, 40 papers in Molecular Biology and 15 papers in Neurology. Recurrent topics in Sílvia Ginés's work include Genetic Neurodegenerative Diseases (35 papers), Mitochondrial Function and Pathology (21 papers) and Neuroscience and Neuropharmacology Research (14 papers). Sílvia Ginés is often cited by papers focused on Genetic Neurodegenerative Diseases (35 papers), Mitochondrial Function and Pathology (21 papers) and Neuroscience and Neuropharmacology Research (14 papers). Sílvia Ginés collaborates with scholars based in Spain, United States and France. Sílvia Ginés's co-authors include Jordi Alberch, Albert Giralt, Verónica Brito, Marta Cherubini, Laura López-Molina, James F. Gusella, Marcy E. MacDonald, Mar Puigdellívol, Josep M. Canals and Paola Paoletti and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Sílvia Ginés

59 papers receiving 3.1k citations

Peers

Sílvia Ginés
Andrew D. Medhurst United Kingdom
Emanuele Sher United Kingdom
Byung Kwan Jin South Korea
Sílvia Ginés
Citations per year, relative to Sílvia Ginés Sílvia Ginés (= 1×) peers Akiko Furuta

Countries citing papers authored by Sílvia Ginés

Since Specialization
Citations

This map shows the geographic impact of Sílvia Ginés's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sílvia Ginés with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sílvia Ginés more than expected).

Fields of papers citing papers by Sílvia Ginés

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sílvia Ginés. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sílvia Ginés. The network helps show where Sílvia Ginés may publish in the future.

Co-authorship network of co-authors of Sílvia Ginés

This figure shows the co-authorship network connecting the top 25 collaborators of Sílvia Ginés. A scholar is included among the top collaborators of Sílvia Ginés based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sílvia Ginés. Sílvia Ginés is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
López-Molina, Laura, et al.. (2025). Mitochondria from huntington´s disease striatal astrocytes are hypermetabolic and compromise neuronal branching. Cell Communication and Signaling. 23(1). 341–341.
2.
Castañé, Anna, Albert Adell, Leticia Campa, et al.. (2024). A combination of Δ9-tetrahydrocannabinol and cannabidiol modulates glutamate dynamics in the hippocampus of an animal model of Alzheimer's disease. Neurotherapeutics. 21(5). e00439–e00439. 2 indexed citations
3.
Sathasivam, Kirupa, Ankita Singh, Daniel del Toro, et al.. (2024). m6A modification of mutant huntingtin RNA promotes the biogenesis of pathogenic huntingtin transcripts. EMBO Reports. 25(11). 5026–5052. 3 indexed citations
4.
Martín‐Flores, Núria, Jordi Creus‐Muncunill, Mercè Masana, et al.. (2020). Synaptic RTP801 contributes to motor-learning dysfunction in Huntington’s disease. Cell Death and Disease. 11(7). 569–569. 12 indexed citations
6.
Puigdellívol, Mar, Verónica Brito, Florian Plattner, et al.. (2017). Cdk5 Contributes to Huntington’s Disease Learning and Memory Deficits via Modulation of Brain Region-Specific Substrates. Molecular Neurobiology. 55(8). 6250–6268. 20 indexed citations
7.
Illa, M., Verónica Brito, E. Eixarch, et al.. (2017). Early Environmental Enrichment Enhances Abnormal Brain Connectivity in a Rabbit Model of Intrauterine Growth Restriction. Fetal Diagnosis and Therapy. 44(3). 184–193. 14 indexed citations
8.
Cherubini, Marta, Mar Puigdellívol, Jordi Alberch, & Sílvia Ginés. (2015). Cdk5-mediated mitochondrial fission: A key player in dopaminergic toxicity in Huntington's disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1852(10). 2145–2160. 31 indexed citations
9.
Fernández‐Nogales, Marta, Félix Hernández, Andrés Miguez, et al.. (2015). Decreased glycogen synthase kinase-3 levels and activity contribute to Huntington's disease. Human Molecular Genetics. 24(17). 5040–5052. 33 indexed citations
10.
Puigdellívol, Mar, Marta Cherubini, Verónica Brito, et al.. (2015). A role for Kalirin-7 in corticostriatal synaptic dysfunction in Huntington's disease. Human Molecular Genetics. 24(25). 7265–7285. 41 indexed citations
11.
Miguez, Andrés, Gerardo Garcia-Díaz Barriga, Verónica Brito, et al.. (2015). Fingolimod (FTY720) enhances hippocampal synaptic plasticity and memory in Huntington's disease by preventing p75NTRup-regulation and astrocyte-mediated inflammation. Human Molecular Genetics. 24(17). 4958–4970. 108 indexed citations
12.
Brito, Verónica, Albert Giralt, Lilian Enríquez-Barreto, et al.. (2014). Neurotrophin receptor p75NTR mediates Huntington’s disease–associated synaptic and memory dysfunction. Journal of Clinical Investigation. 124(10). 4411–4428. 87 indexed citations
13.
Giralt, Albert, Mar Puigdellívol, Paola Paoletti, et al.. (2011). Long-term memory deficits in Huntington's disease are associated with reduced CBP histone acetylase activity. Human Molecular Genetics. 21(6). 1203–1216. 119 indexed citations
14.
Almajano, María Pilar, et al.. (2011). Neuroprotective Effects of White Tea Against Oxidative Stress-Induced Toxicity in Striatal Cells. Neurotoxicity Research. 20(4). 372–378. 45 indexed citations
15.
Ginés, Sílvia, Paola Paoletti, & Jordi Alberch. (2010). Impaired TrkB-mediated ERK1/2 Activation in Huntington Disease Knock-in Striatal Cells Involves Reduced p52/p46 Shc Expression. Journal of Biological Chemistry. 285(28). 21537–21548. 55 indexed citations
16.
Chen‐Plotkin, Alice, Ghazaleh Sadri‐Vakili, George J. Yohrling, et al.. (2006). Decreased association of the transcription factor Sp1 with genes downregulated in Huntington's disease. Neurobiology of Disease. 22(2). 233–241. 90 indexed citations
17.
Wang, Jin, Sílvia Ginés, Marcy E. MacDonald, & James F. Gusella. (2005). Reversal of a full-length mutant huntingtin neuronal cell phenotype by chemical inhibitors of polyglutamine-mediated aggregation. BMC Neuroscience. 6(1). 1–1. 152 indexed citations
18.
MacDonald, Marcy E., Sílvia Ginés, James F. Gusella, & Vanessa C. Wheeler. (2003). Huntington's Disease. NeuroMolecular Medicine. 4(1-2). 7–20. 75 indexed citations
19.
Ginés, Sílvia, et al.. (2003). Enhanced Akt Signaling Is an Early Pro-survival Response That Reflects N-Methyl-D-aspartate Receptor Activation in Huntington's Disease Knock-in Striatal Cells. Journal of Biological Chemistry. 278(50). 50514–50522. 120 indexed citations
20.
Torvinen, Maria, Sílvia Ginés, Jöelle Hillion, et al.. (2002). Interactions among adenosine deaminase, adenosine A1 receptors and dopamine D1 receptors in stably cotransfected fibroblast cells and neurons. Neuroscience. 113(3). 709–719. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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