Joan X. Comella

7.2k total citations · 1 hit paper
112 papers, 5.7k citations indexed

About

Joan X. Comella is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Joan X. Comella has authored 112 papers receiving a total of 5.7k indexed citations (citations by other indexed papers that have themselves been cited), including 90 papers in Molecular Biology, 47 papers in Cellular and Molecular Neuroscience and 15 papers in Cell Biology. Recurrent topics in Joan X. Comella's work include Cell death mechanisms and regulation (40 papers), Nerve injury and regeneration (25 papers) and Neuroscience and Neuropharmacology Research (17 papers). Joan X. Comella is often cited by papers focused on Cell death mechanisms and regulation (40 papers), Nerve injury and regeneration (25 papers) and Neuroscience and Neuropharmacology Research (17 papers). Joan X. Comella collaborates with scholars based in Spain, France and United States. Joan X. Comella's co-authors include Mario Encinas, Montse Iglesias, Joaquim Egea, Rosa M. Soler, Núria Llecha, Vı́ctor J. Yuste, Valentı́n Ceña, Xavier Dolcet, Carme Gallego and Marta Llovera and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and SHILAP Revista de lepidopterología.

In The Last Decade

Joan X. Comella

111 papers receiving 5.6k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Sequential Treatment of SH‐SY5Y Cells with Retinoic Acid and Brain‐Derived Neurotrophic Factor Gives Rise to Fully Differentiated, Neurotrophic Factor‐Dependent, Human Neuron‐Like Cells

2000 647 citations Mario Encinas, Joan X. Comella et al. Journal of Neurochemistry profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Joan X. Comella Spain	47	3.3k	1.7k	678	673	656	112	5.7k
Laura Conforti United States	40	2.7k 0.8×	1.6k 0.9×	489 0.7×	396 0.6×	513 0.8×	119	5.3k
Michal Hetman United States	39	2.8k 0.8×	1.7k 1.0×	620 0.9×	596 0.9×	288 0.4×	82	4.9k
Rebecca M. Pruss United States	37	3.4k 1.0×	2.1k 1.2×	908 1.3×	754 1.1×	621 0.9×	78	6.0k
Jacques Baudier France	44	4.2k 1.3×	1.1k 0.6×	801 1.2×	452 0.7×	395 0.6×	87	5.6k
Yuji Owada Japan	40	2.8k 0.8×	1.0k 0.6×	425 0.6×	316 0.5×	538 0.8×	178	5.1k
Susan L. Ackerman United States	43	5.0k 1.5×	2.2k 1.3×	1.4k 2.0×	939 1.4×	308 0.5×	79	7.2k
Ana Martín-Villalba Germany	37	3.2k 1.0×	1.3k 0.7×	403 0.6×	890 1.3×	449 0.7×	69	5.7k
Mohanish Deshmukh United States	40	4.2k 1.3×	978 0.6×	489 0.7×	377 0.6×	332 0.5×	72	5.6k
Takayuki Harada Japan	42	3.5k 1.1×	1.4k 0.8×	493 0.7×	351 0.5×	412 0.6×	197	5.9k
Aviva M. Tolkovsky United Kingdom	43	4.3k 1.3×	1.7k 1.0×	1.2k 1.8×	397 0.6×	609 0.9×	89	7.5k

Countries citing papers authored by Joan X. Comella

Since Specialization

Citations

This map shows the geographic impact of Joan X. Comella's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joan X. Comella with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joan X. Comella more than expected).

Fields of papers citing papers by Joan X. Comella

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joan X. Comella. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joan X. Comella. The network helps show where Joan X. Comella may publish in the future.

Co-authorship network of co-authors of Joan X. Comella

This figure shows the co-authorship network connecting the top 25 collaborators of Joan X. Comella. A scholar is included among the top collaborators of Joan X. Comella based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joan X. Comella. Joan X. Comella is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Comella, Joan X., et al.. (2024). Synaptic and extrasynaptic distribution of NMDA receptors in the cortex of Alzheimer's disease patients. Alzheimer s & Dementia. 20(12). 8231–8245. 13 indexed citations

Little, Karis, Aditi Singh, Àngel del Marco, et al.. (2023). Disruption of cortical cell type composition and function underlies diabetes-associated cognitive decline. Diabetologia. 66(8). 1557–1575. 11 indexed citations

Sáez, María Eugenia, Antonio González‐Pérez, Sonia Moreno–Grau, et al.. (2019). Genome Wide Meta-Analysis identifies common genetic signatures shared by heart function and Alzheimer’s disease. Scientific Reports. 9(1). 16665–16665. 2 indexed citations

Gómez-Arboledas, Ángela, José Carlos Dávila, Elisabeth Sánchez‐Mejías, et al.. (2017). Phagocytic clearance of presynaptic dystrophies by reactive astrocytes in Alzheimer's disease. Glia. 66(3). 637–653. 151 indexed citations

Bosch, Carles, Núria Masachs, David Exposito-Alonso, et al.. (2016). Reelin Regulates the Maturation of Dendritic Spines, Synaptogenesis and Glial Ensheathment of Newborn Granule Cells. Cerebral Cortex. 26(11). 4282–4298. 48 indexed citations

Gozzelino, Raffaella, Koen M.O. Galenkamp, Stéphanie Reix, et al.. (2013). TNFα induces survival through the FLIP-L-dependent activation of the MAPK/ERK pathway. Cell Death and Disease. 4(2). e493–e493. 61 indexed citations

Ye, Junmei, et al.. (2012). Translation of Myocyte Enhancer Factor-2 is induced by hypertrophic stimuli in cardiomyocytes through a Calcineurin-dependent pathway. Journal of Molecular and Cellular Cardiology. 53(4). 578–587. 15 indexed citations

Badiola, Nahuai, Clara Penas, Alfredo J. Miñano‐Molina, et al.. (2011). Induction of ER stress in response to oxygen-glucose deprivation of cortical cultures involves the activation of the PERK and IRE-1 pathways and of caspase-12. Cell Death and Disease. 2(4). e149–e149. 135 indexed citations

Moubarak, Rana S., Carme Solé, Marta Pascual, et al.. (2010). The Death Receptor Antagonist FLIP-L Interacts with Trk and Is Necessary for Neurite Outgrowth Induced by Neurotrophins. Journal of Neuroscience. 30(17). 6094–6105. 13 indexed citations

10.

Garcerá, Ana, et al.. (2009). Specific vulnerability of mouse spinal cord motoneurons to membrane depolarization. Journal of Neurochemistry. 110(6). 1842–1854. 24 indexed citations

11.

Gómez-Lázaro, María, Marı́a F. Galindo, Caoimhín G. Concannon, et al.. (2007). 6‐Hydroxydopamine activates the mitochondrial apoptosis pathway through p38 MAPK‐mediated, p53‐independent activation of Bax and PUMA. Journal of Neurochemistry. 104(6). 1599–1612. 116 indexed citations

12.

Benito‐Gutiérrez, Èlia, Christian Näke, Marta Llovera, Joan X. Comella, & Jordi García‐Fernàndez. (2005). The single AmphiTrk receptor highlights increased complexity of neurotrophin signalling in vertebrates and suggests an early role in developing sensory neuroepidermal cells. Development. 132(9). 2191–2202. 58 indexed citations

13.

Benito‐Gutiérrez, Èlia, et al.. (2005). Outlining the nascent nervous system of Branchiostoma floridae (amphioxus) by the pan-neural marker AmphiElav. Brain Research Bulletin. 66(4-6). 518–521. 20 indexed citations

14.

Yuste, Vı́ctor J., et al.. (2001). The Absence of Oligonucleosomal DNA Fragmentation during Apoptosis of IMR-5 Neuroblastoma Cells. Journal of Biological Chemistry. 276(25). 22323–22331. 61 indexed citations

15.

Egea, Joaquim, Carme Espinet, Rosa M. Soler, et al.. (2001). Neuronal survival induced by neurotrophins requires calmodulin. The Journal of Cell Biology. 154(3). 585–598. 50 indexed citations

16.

Egea, Joaquim, Carme Espinet, Rosa M. Soler, et al.. (2000). Nerve Growth Factor Activation of the Extracellular Signal-Regulated Kinase Pathway Is Modulated by Ca ²⁺ and Calmodulin. Molecular and Cellular Biology. 20(6). 1931–1946. 47 indexed citations

17.

Soler, Rosa M., Xavier Dolcet, Mario Encinas, et al.. (1999). Receptors of the Glial Cell Line-Derived Neurotrophic Factor Family of Neurotrophic Factors Signal Cell Survival through the Phosphatidylinositol 3-Kinase Pathway in Spinal Cord Motoneurons. Journal of Neuroscience. 19(21). 9160–9169. 148 indexed citations

18.

Egea, Joaquim, Carme Espinet, & Joan X. Comella. (1998). Calmodulin Modulates Mitogen‐Activated Protein Kinase Activation in Response to Membrane Depolarization in PC12 Cells. Journal of Neurochemistry. 70(6). 2554–2564. 30 indexed citations

19.

Becker, Elena, Rosa M. Soler, Vı́ctor J. Yuste, et al.. (1998). Development of Survival Responsiveness to Brain-Derived Neurotrophic Factor, Neurotrophin 3 and Neurotrophin 4/5, But Not to Nerve Growth Factor, in Cultured Motoneurons from Chick Embryo Spinal Cord. Journal of Neuroscience. 18(19). 7903–7911. 55 indexed citations

20.

Esquerda, Josep E., et al.. (1989). Absence of histochemical immunoreactivity to calcitonin gene-related peptide (CGRP) in spinal cord motoneurons from (+)-tubocurarine-treated chick embryos. Neuroscience Letters. 105(1-2). 1–6. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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