Nicole Hannesschläger
- Immunology top 2%
- Immune Response and Inflammation 3
- Immune Cell Function and Interaction 2
- interferon and immune responses 1
- Infectious Diseases top 5%
- Viral Infections and Vectors 1
- Antifungal resistance and susceptibility 1
- Epidemiology top 5%
- Fungal Infections and Studies 1
- Molecular Biology top 10%
- Inflammasome and immune disorders 3
- Nephrology top 10%
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- Ion Channels and Receptors 1
- Co-authors
- Jürgen RulandOlaf GroßStefan EndresHendrik PoeckMichael BscheiderGunther HartmannCatherine DostertEdina Schweighoffer
- Partner nations
- GermanyUnited StatesSwitzerland
In The Last Decade
Nicole Hannesschläger
5 papers receiving 2.0k citations
Hit Papers
Peers
Comparison fields: 5 of 83
- Immunology 1.2k
- Infectious Diseases 548
- Epidemiology 533
- Molecular Biology 919
- Nephrology 82
Countries citing papers authored by Nicole Hannesschläger
This map shows the geographic impact of Nicole Hannesschläger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicole Hannesschläger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicole Hannesschläger more than expected).
Fields of papers citing papers by Nicole Hannesschläger
This network shows the impact of papers produced by Nicole Hannesschläger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicole Hannesschläger. The network helps show where Nicole Hannesschläger may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Nicole Hannesschläger, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2014 | 141 | |
| 2 | Syk kinase signalling couples to the Nlrp3 inflammasome for anti-fungal host defencebreakdown → | 2009 | 742 |
| 3 | 2009 | 422 | |
| 4 | A Homozygous CARD9 Mutation in a Family with Susceptibility to Fungal Infectionsbreakdown → | 2009 | 574 |
| 5 | 2008 | 91 |
About Nicole Hannesschläger
Nicole Hannesschläger is a scholar working on Immunology, Sensory Systems, Infectious Diseases, Cancer Research and Molecular Biology, having authored 5 papers that have together received 2.0k indexed citations. Recurring topics across this work include Immune Response and Inflammation (3 papers), Inflammasome and immune disorders (3 papers), Immune Cell Function and Interaction (2 papers), interferon and immune responses (1 paper), Fungal Infections and Studies (1 paper), Viral Infections and Vectors (1 paper), Ion Channels and Receptors (1 paper) and Antifungal resistance and susceptibility (1 paper). The work is most often cited by research in Immunology (1.2k citations), Infectious Diseases (548 citations), Epidemiology (533 citations), Molecular Biology (919 citations) and Nephrology (82 citations). Nicole Hannesschläger has collaborated with scholars based in Germany, United States and Switzerland. Frequent co-authors include Jürgen Ruland, Olaf Groß, Stefan Endres, Hendrik Poeck, Michael Bscheider, Gunther Hartmann, Catherine Dostert, Edina Schweighoffer, Victor L. J. Tybulewicz and Jürg Tschopp. Their work appears in journals such as Nature Immunology, Nature, Blood, Immunity and New England Journal of Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.