Attila Mócsai

12.5k total citations · 5 hit papers
113 papers, 9.7k citations indexed

About

Attila Mócsai is a scholar working on Immunology, Immunology and Allergy and Molecular Biology. According to data from OpenAlex, Attila Mócsai has authored 113 papers receiving a total of 9.7k indexed citations (citations by other indexed papers that have themselves been cited), including 61 papers in Immunology, 40 papers in Immunology and Allergy and 37 papers in Molecular Biology. Recurrent topics in Attila Mócsai's work include Cell Adhesion Molecules Research (39 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (33 papers) and Immune Response and Inflammation (25 papers). Attila Mócsai is often cited by papers focused on Cell Adhesion Molecules Research (39 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (33 papers) and Immune Response and Inflammation (25 papers). Attila Mócsai collaborates with scholars based in Hungary, Germany and United States. Attila Mócsai's co-authors include Clifford A. Lowell, Victor L. J. Tybulewicz, Jürgen Ruland, Tamás Németh, Krisztina Futosi, Szabina Fodor, Zoltán Jakus, Markus Sperandio, Giorgio Berton and Erzsébet Ligeti and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Attila Mócsai

111 papers receiving 9.6k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The SYK tyrosine kinase: a crucial player in diverse biological functions

2010 1.0k citations Attila Mócsai, Jürgen Ruland et al. profile →
Syk kinase signalling couples to the Nlrp3 inflammasome for anti-fungal host defence

2009 742 citations Olaf Groß, Victor L. J. Tybulewicz et al. profile →
Diverse novel functions of neutrophils in immunity, inflammation, and beyond

2013 522 citations Attila Mócsai The Journal of Experimental Medicine profile →
Neutrophil cell surface receptors and their intracellular signal transduction pathways

2013 497 citations Krisztina Futosi, Szabina Fodor et al. profile →
Neutrophils as emerging therapeutic targets

2020 492 citations Tamás Németh, Markus Sperandio et al. Nature Reviews Drug Discovery profile →

Peers

Countries citing papers authored by Attila Mócsai

Since Specialization

Citations

This map shows the geographic impact of Attila Mócsai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Attila Mócsai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Attila Mócsai more than expected).

Fields of papers citing papers by Attila Mócsai

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Attila Mócsai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Attila Mócsai. The network helps show where Attila Mócsai may publish in the future.

Co-authorship network of co-authors of Attila Mócsai

This figure shows the co-authorship network connecting the top 25 collaborators of Attila Mócsai. A scholar is included among the top collaborators of Attila Mócsai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Attila Mócsai. Attila Mócsai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Time-restricted feeding alleviates arthritis symptoms augmented by high-fat diet 2025 ·Frontiers in Immunology ·(unknown), (unknown), (unknown), (unknown), (unknown), Attila Mócsai, Krisztina Káldi, Krisztina Ella	1
2	Are Artificial Intelligence-Assisted Three-Dimensional Histological Reconstructions Reliable for the Assessment of Trabecular Microarchitecture? 2024 ·Journal of Clinical Medicine ·(unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Miklós Szócska, (unknown), Csaba Dobó‐Nagy, (unknown), Attila Mócsai, Orsolya Németh, Péter Pollner, Eitan Mijiritsky, Márton Kivovics	2
3	Neutrophils and NADPH Oxidases Are Major Contributors to Mild but Not Severe Ischemic Acute Kidney Injury in Mice 2024 ·International Journal of Molecular Sciences ·Csaba Révész, Tamás Kaucsár, Mária Godó, (unknown), Tibor Krenács, Attila Mócsai, Gábor Szénási, Péter Hamar	3
4	The Syk Inhibitor Entospletinib Abolishes Dermal–Epidermal Separation in a Fully Human Ex Vivo Model of Bullous Pemphigoid 2024 ·Journal of Investigative Dermatology ·(unknown), (unknown), (unknown), Miklós Sárdy, Attila Mócsai	1
5	The selective inhibition of the Syk tyrosine kinase ameliorates experimental autoimmune arthritis 2023 ·Frontiers in Immunology ·(unknown), (unknown), Attila Mócsai, (unknown), Zoltán Jakus, Tamás Németh	3
6	Investigation of the Role of the TRPA1 Ion Channel in Conveying the Effect of Dimethyl Trisulfide on Vascular and Histological Changes in Serum-Transfer Arthritis 2022 ·Pharmaceuticals ·(unknown), (unknown), Éva Borbély, (unknown), Ferenc Papp, Zoltán Varga, Attila Mócsai, Zsuzsanna Helyes, Erika Pintér, Gábor Pozsgai	3
7	In Vivo Functions of Mouse Neutrophils Derived from HoxB8-Transduced Conditionally Immortalized Myeloid Progenitors 2020 ·The Journal of Immunology ·(unknown), Barbara Walzog, Attila Mócsai	10
8	Neutrophils as emerging therapeutic targets breakdown → 2020 ·Nature Reviews Drug Discovery ·Tamás Németh, Markus Sperandio, Attila Mócsai	492
9	Importance of Fc Receptor γ-Chain ITAM Tyrosines in Neutrophil Activation and in vivo Autoimmune Arthritis 2019 ·Frontiers in Immunology ·Tamás Németh, Krisztina Futosi, (unknown), (unknown), Takashi Saito, Attila Mócsai, Zoltán Jakus	12
10	Myeloid-Specific Deletion of Mcl-1 Yields Severely Neutropenic Mice That Survive and Breed in Homozygous Form 2018 ·The Journal of Immunology ·Janka Zsófia Csepregi, (unknown), (unknown), (unknown), Tamás Németh, Edina Simon, Szabina Fodor, Katalin Csonka, (unknown), Dorottya Kövesdi, You‐Wen He, Attila Gácser, Attila Mócsai	33
11	Oral administration of the selective GPR120/FFA4 agonist compound A is not effective in alleviating tissue inflammation in mouse models of prototypical autoimmune diseases 2018 ·Pharmacology Research & Perspectives ·(unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Michaela Seeling, Falk Nimmerjahn, Attila Mócsai, Detlef Zillikens, Tanya Sezin, Christian D. Sadik	16
12	The Syk Tyrosine Kinase Is Required for Skin Inflammation in an In Vivo Mouse Model of Epidermolysis Bullosa Acquisita 2017 ·Journal of Investigative Dermatology ·Tamás Németh, (unknown), Cassian Sitaru, Attila Mócsai	32
13	Repercussion of Megakaryocyte-Specific Gata1 Loss on Megakaryopoiesis and the Hematopoietic Precursor Compartment 2016 ·PLoS ONE ·Marjolein Meinders, Mark Hoogenboezem, Maaike R. Scheenstra, Iris M. De Cuyper, Petros Papadopoulos, Tamás Németh, Attila Mócsai, Timo K. van den Berg, Taco W. Kuijpers, Laura Gutiérrez	13
14	The role of neutrophils in autoimmune diseases 2012 ·Immunology Letters ·Tamás Németh, Attila Mócsai	154
15	Phospholipase Cγ2 is required for basal but not oestrogen deficiency–induced bone resorption 2011 ·European Journal of Clinical Investigation ·(unknown), Dávid Győri, (unknown), Tünde Fekete, Katalin Kis‐Tóth, Zoltán Jakus, Georg Schett, Éva Rajnavölgyi, Csaba Dobó‐Nagy, Attila Mócsai	29
16	Adjuvanticity of a synthetic cord factor analogue for subunit Mycobacterium tuberculosis vaccination requires FcRγ–Syk–Card9–dependent innate immune activation 2009 ·The Journal of Experimental Medicine ·(unknown), (unknown), Olaf Groß, Christoph Hölscher, (unknown), Else Marie Agger, Jörg Mages, Attila Mócsai, Hanne Schoenen, Katrin Finger, Falk Nimmerjahn, Gordon D. Brown, Carsten J. Kirschning, Antje Heit, Peter Andersen, Hermann Wagner, Jürgen Ruland, Roland Lang	258
17	Responses of Neutrophils to Anti-Integrin Antibodies Depends on Costimulation through Low Affinity FcγRs: Full Activation Requires Both Integrin and Nonintegrin Signals 2004 ·The Journal of Immunology ·Zoltán Jakus, Giorgio Berton, Erzsébet Ligeti, Clifford A. Lowell, Attila Mócsai	35
18	Regulation of Blood and Lymphatic Vascular Separation by Signaling Proteins SLP-76 and Syk 2003 ·Science ·Farhad Abtahian, Anastasia Guerriero, Eric Sebzda, Minmin Lu, Rong Zhou, Attila Mócsai, (unknown), Bin Huang, David G. Jackson, Victor A. Ferrari, Victor L. J. Tybulewicz, Clifford A. Lowell, John J. Lepore, Gary A. Koretzky, Mark L. Kahn	330
19	Separation of blood and lymphatic vascular networks is regulated by the hematopoietic signaling proteins SLP-76 and Syk. 2002 ·Blood ·Farhad Abtahian, Anastasia Guerriero, (unknown), (unknown), Attila Mócsai, (unknown), Bin Huang, Darryl Jackson, Victor A. Ferrari, Victor L. J. Tybulewicz, Clifford A. Lowell, John J. Lepore, Gary A. Koretzky, Mark L. Kahn	1
20	Resting Murine Neutrophils Express Functional α4 Integrins that Signal Through Src Family Kinases 2001 ·The Journal of Immunology ·Shalini Pereira, Meijuan Zhou, Attila Mócsai, Clifford A. Lowell	49

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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