Nicholas A. Willis

7.7k total citations · 5 hit papers
37 papers, 5.4k citations indexed

About

Nicholas A. Willis is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Nicholas A. Willis has authored 37 papers receiving a total of 5.4k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 12 papers in Oncology and 7 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Nicholas A. Willis's work include DNA Repair Mechanisms (23 papers), CRISPR and Genetic Engineering (16 papers) and PARP inhibition in cancer therapy (7 papers). Nicholas A. Willis is often cited by papers focused on DNA Repair Mechanisms (23 papers), CRISPR and Genetic Engineering (16 papers) and PARP inhibition in cancer therapy (7 papers). Nicholas A. Willis collaborates with scholars based in United States, United Kingdom and Czechia. Nicholas A. Willis's co-authors include Tyler Jacks, David A. Tuveson, Ralph Scully, Roderick T. Bronson, Denise Crowley, Arvind Panday, Rajula Elango, Kim L. Mercer, Erica L. Jackson and R Montoya and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Nicholas A. Willis

37 papers receiving 5.3k citations

Hit Papers

Analysis of lung tumor initiation and progression using c... 2001 2026 2009 2017 2001 2004 2019 2004 2001 400 800 1.2k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nicholas A. Willis United States 18 3.7k 2.3k 998 721 501 37 5.4k
Kenji Hibi Japan 40 2.8k 0.8× 1.9k 0.8× 1.0k 1.0× 897 1.2× 347 0.7× 142 4.6k
Wolfgang Dietmaier Germany 41 2.5k 0.7× 2.3k 1.0× 1.4k 1.4× 989 1.4× 509 1.0× 135 6.2k
Margaret Leversha United States 24 1.9k 0.5× 1.7k 0.7× 1.3k 1.3× 1.3k 1.9× 359 0.7× 37 4.5k
Michael T. Barrett United States 34 1.9k 0.5× 1.4k 0.6× 1.1k 1.1× 840 1.2× 486 1.0× 99 4.2k
Fernanda Milanezi Portugal 36 1.7k 0.5× 1.9k 0.8× 1.2k 1.3× 686 1.0× 332 0.7× 75 4.0k
Mieke Schutte Netherlands 38 5.2k 1.4× 5.3k 2.3× 2.4k 2.4× 523 0.7× 1.3k 2.6× 63 8.7k
Ruth Holm Norway 45 2.8k 0.8× 2.8k 1.2× 1.3k 1.3× 680 0.9× 403 0.8× 187 6.1k
Nina N. Nupponen Finland 35 2.2k 0.6× 999 0.4× 1.1k 1.1× 1.4k 2.0× 610 1.2× 64 4.5k
Katharine Romans United States 18 2.6k 0.7× 2.0k 0.9× 2.5k 2.5× 1.2k 1.6× 334 0.7× 25 5.5k
John F. Lyons United States 32 2.7k 0.7× 1.3k 0.6× 551 0.6× 454 0.6× 308 0.6× 81 4.3k

Countries citing papers authored by Nicholas A. Willis

Since Specialization
Citations

This map shows the geographic impact of Nicholas A. Willis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicholas A. Willis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicholas A. Willis more than expected).

Fields of papers citing papers by Nicholas A. Willis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nicholas A. Willis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicholas A. Willis. The network helps show where Nicholas A. Willis may publish in the future.

Co-authorship network of co-authors of Nicholas A. Willis

This figure shows the co-authorship network connecting the top 25 collaborators of Nicholas A. Willis. A scholar is included among the top collaborators of Nicholas A. Willis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nicholas A. Willis. Nicholas A. Willis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Elango, Rajula, Daniel Nguyen, Emilie Rass, et al.. (2024). Two-ended recombination at a Flp-nickase-broken replication fork. Molecular Cell. 85(1). 78–90.e3. 10 indexed citations
2.
Cvek, Jakub, Charles Kelly, Nick West, et al.. (2024). Timing of development of osteoradionecrosis post head and neck radiotherapy: does a safe time interval exist for dental extraction?. Strahlentherapie und Onkologie. 200(10). 882–894. 2 indexed citations
3.
Cvek, Jakub, C. Kelly, Nick West, et al.. (2023). Extra-mandibular Osteoradionecrosis after the Treatment of Head and Neck Cancer. Clinical Oncology. 35(9). e498–e505. 3 indexed citations
4.
Elango, Rajula, Arvind Panday, Francis P. Lach, et al.. (2022). The structure-specific endonuclease complex SLX4–XPF regulates Tus–Ter-induced homologous recombination. Nature Structural & Molecular Biology. 29(8). 801–812. 14 indexed citations
5.
Panday, Arvind, Nicholas A. Willis, Rajula Elango, et al.. (2021). FANCM regulates repair pathway choice at stalled replication forks. Molecular Cell. 81(11). 2428–2444.e6. 47 indexed citations
6.
Scully, Ralph, Rajula Elango, Arvind Panday, & Nicholas A. Willis. (2021). Recombination and restart at blocked replication forks. Current Opinion in Genetics & Development. 71. 154–162. 16 indexed citations
7.
Kovařík, Josef, et al.. (2021). Osteoradionecrosis after treatment of head and neck cancer: a comprehensive analysis of risk factors with a particular focus on role of dental extractions. British Journal of Oral and Maxillofacial Surgery. 60(2). 168–173. 9 indexed citations
8.
Willis, Nicholas A. & Ralph Scully. (2020). Measurement of Homologous Recombination at Stalled Mammalian Replication Forks. Methods in molecular biology. 2153. 329–353. 5 indexed citations
9.
Smith, Eric A., Boris Gole, Nicholas A. Willis, et al.. (2017). DEK is required for homologous recombination repair of DNA breaks. Scientific Reports. 7(1). 44662–44662. 26 indexed citations
10.
Willis, Nicholas A., Richard L. Frock, Francesca Menghi, et al.. (2017). Mechanism of tandem duplication formation in BRCA1-mutant cells. Nature. 551(7682). 590–595. 95 indexed citations
11.
Willis, Nicholas A., Gurushankar Chandramouly, Bin Huang, et al.. (2014). BRCA1 controls homologous recombination at Tus/Ter-stalled mammalian replication forks. Nature. 510(7506). 556–559. 116 indexed citations
12.
Chandramouly, Gurushankar, Amy Kwok, Bin Huang, et al.. (2013). BRCA1 and CtIP suppress long-tract gene conversion between sister chromatids. Nature Communications. 4(1). 2404–2404. 52 indexed citations
14.
Willis, Nicholas A. & Nick Rhind. (2011). Studying G2 DNA Damage Checkpoints Using the Fission Yeast Schizosaccharomyces pombe. Methods in molecular biology. 782. 1–12. 9 indexed citations
15.
Chandramouly, Gurushankar, Nicholas A. Willis, & Ralph Scully. (2011). A protective role for BRCA2 at stalled replication forks. Breast Cancer Research. 13(5). 314–314. 6 indexed citations
16.
Willis, Nicholas A. & Nick Rhind. (2008). Mus81, Rhp51(Rad51), and Rqh1 Form an Epistatic Pathway Required for the S-Phase DNA Damage Checkpoint. Molecular Biology of the Cell. 20(3). 819–833. 34 indexed citations
17.
Gundersen, Robert E., et al.. (2005). Loss-of-function mutations identified in the Helical domain of the G protein α-subunit, Gα2, of Dictyostelium discoideum. Biochimica et Biophysica Acta (BBA) - General Subjects. 1722(3). 262–270. 4 indexed citations
18.
Olive, Kenneth P., David A. Tuveson, Zachary C. Ruhe, et al.. (2004). Mutant p53 Gain of Function in Two Mouse Models of Li-Fraumeni Syndrome. Cell. 119(6). 847–860. 974 indexed citations breakdown →
19.
Tuveson, David A., Alice T. Shaw, Nicholas A. Willis, et al.. (2004). Endogenous oncogenic K-rasG12D stimulates proliferation and widespread neoplastic and developmental defects. Cancer Cell. 5(4). 375–387. 626 indexed citations breakdown →
20.
Jackson, Erica L., Nicholas A. Willis, Kim L. Mercer, et al.. (2001). Analysis of lung tumor initiation and progression using conditional expression of oncogenic K-ras. Genes & Development. 15(24). 3243–3248. 1392 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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