Michael Ittmann

83.8k total citations · 3 hit papers
279 papers, 20.8k citations indexed

About

Michael Ittmann is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cancer Research. According to data from OpenAlex, Michael Ittmann has authored 279 papers receiving a total of 20.8k indexed citations (citations by other indexed papers that have themselves been cited), including 190 papers in Molecular Biology, 106 papers in Pulmonary and Respiratory Medicine and 81 papers in Cancer Research. Recurrent topics in Michael Ittmann's work include Prostate Cancer Treatment and Research (94 papers), Fibroblast Growth Factor Research (40 papers) and Epigenetics and DNA Methylation (35 papers). Michael Ittmann is often cited by papers focused on Prostate Cancer Treatment and Research (94 papers), Fibroblast Growth Factor Research (40 papers) and Epigenetics and DNA Methylation (35 papers). Michael Ittmann collaborates with scholars based in United States, Türkiye and China. Michael Ittmann's co-authors include Mustafa Özen, Dipak Kumar Giri, Chad J. Creighton, Ramon Parsons, Jianghua Wang, Bernard Kwabi‐Addo, Katrina Podsypanina, Claudio Basilico, Hanina Hibshoosh and Ben Tycko and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Michael Ittmann

277 papers receiving 20.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

PTEN , a Putative Protein Tyrosine Phosphatase Gene Mutated in Human Brain, Breast, and Prostate Cancer

1997 3.9k citations Michael Ittmann et al. profile →
Heparanase promotes tumor infiltration and antitumor activity of CAR-redirected T lymphocytes

2015 642 citations Ignazio Caruana, Barbara Savoldo et al. profile →
Widespread deregulation of microRNA expression in human prostate cancer

2007 547 citations Mustafa Özen, Chad J. Creighton et al. profile →

Peers

Countries citing papers authored by Michael Ittmann

Since Specialization

Citations

This map shows the geographic impact of Michael Ittmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Ittmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Ittmann more than expected).

Fields of papers citing papers by Michael Ittmann

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Ittmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Ittmann. The network helps show where Michael Ittmann may publish in the future.

Co-authorship network of co-authors of Michael Ittmann

This figure shows the co-authorship network connecting the top 25 collaborators of Michael Ittmann. A scholar is included among the top collaborators of Michael Ittmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael Ittmann. Michael Ittmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Pan-Cancer Molecular Classes Transcending Tumor Lineage Across 32 Cancer Types, Multiple Data Platforms, and over 10,000 Cases 2018 ·Clinical Cancer Research ·Fengju Chen, Yiqun Zhang, Don L. Gibbons, Benjamin Deneen, David J. Kwiatkowski, Michael Ittmann, Chad J. Creighton	61
2	MNX1 Is Oncogenically Upregulated in African-American Prostate Cancer 2016 ·Cancer Research ·Li Zhang, Jianghua Wang, Yongquan Wang, Yiqun Zhang, Patricia Castro, Longjiang Shao, Arun Sreekumar, Nagireddy Putluri, (unknown), Saligrama Deepak, (unknown), Chad J. Creighton, Michael Ittmann	47
3	Nuclear Receptor Corepressor 1 Expression and Output Declines with Prostate Cancer Progression 2016 ·Clinical Cancer Research ·(unknown), Alexander I. Agoulnik, Manqi Zhang, L. E. Peterson, (unknown), (unknown), Anna Frolov, Rile Li, Kimal Rajapakshe, (unknown), Michael Ittmann, Nancy L. Weigel, Irina U. Agoulnik	21
4	FGFR1–WNT–TGF-β Signaling in Prostate Cancer Mouse Models Recapitulates Human Reactive Stroma 2013 ·Cancer Research ·Julienne L. Carstens, Payam Shahi, (unknown), (unknown), Chad J. Creighton, Yiqun Zhang, (unknown), Mamatha Seethammagari, (unknown), Jonathan M. Levitt, Michael Ittmann, David R. Rowley, David M. Spencer	35
5	Endocrine Fibroblast Growth Factor FGF19 Promotes Prostate Cancer Progression 2013 ·Cancer Research ·Shu Feng, Olga Dakhova, Chad J. Creighton, Michael Ittmann	69
6	Animal Models of Human Prostate Cancer: The Consensus Report of the New York Meeting of the Mouse Models of Human Cancers Consortium Prostate Pathology Committee 2013 ·Cancer Research ·Michael Ittmann, Jiaoti Huang, Enrico Radaelli, Philip Martin, Sabina Signoretti, Ruth Sullivan, Brian W. Simons, Jerrold M. Ward, Brian D. Robinson, Gerald C. Chu, Massimo Loda, George Thomas, Alexander D. Borowsky, Robert D. Cardiff	184
7	ERK and AKT Signaling Drive MED1 Overexpression in Prostate Cancer in Association with Elevated Proliferation and Tumorigenicity 2013 ·Molecular Cancer Research ·Feng Jin, Shazia Irshad, Wei Yü, Madesh Belakavadi, Marina Chekmareva, Michael Ittmann, Cory Abate‐Shen, Joseph D. Fondell	30
8	FGFR1 Is Essential for Prostate Cancer Progression and Metastasis 2013 ·Cancer Research ·Feng Yang, Yongyou Zhang, Steven J. Ressler, Michael Ittmann, Gustavo E. Ayala, Truong D. Dang, Fen Wang, David R. Rowley	82
9	T Lymphocytes Redirected against the Chondroitin Sulfate Proteoglycan-4 Control the Growth of Multiple Solid Tumors both In Vitro and In Vivo 2013 ·Clinical Cancer Research ·Claudia Geldres, Barbara Savoldo, Valentina Hoyos, Ignazio Caruana, Ming Zhang, Eric Yvon, Michele Del Vecchio, Chad J. Creighton, Michael Ittmann, Soldano Ferrone, Gianpietro Dotti	80
10	Targeting Fibroblast Growth Factor Receptor Signaling Inhibits Prostate Cancer Progression 2012 ·Clinical Cancer Research ·Shu Feng, Longjiang Shao, Wendong Yu, Paul R. Gavine, Michael Ittmann	40
11	Highly Specific Targeting of the TMPRSS2/ERG Fusion Gene Using Liposomal Nanovectors 2012 ·Clinical Cancer Research ·Longjiang Shao, İbrahim Tekedereli, Jianghua Wang, Erkan Yuca, (unknown), Anil K. Sood, Gabriel Lopez‐Berestein, Bülent Özpolat, Michael Ittmann	48
12	GLIPR1 Suppresses Prostate Cancer Development through Targeted Oncoprotein Destruction 2011 ·Cancer Research ·Likun Li, Chengzhen Ren, Guang Yang, Elmoataz Abdel Fattah, Alexei A. Goltsov, Soo Mi Kim, Ju‐Seog Lee, Sanghee Park, Francesco J. DeMayo, Michael Ittmann, Patricia Troncoso, Timothy C. Thompson	32
13	Common Structural and Epigenetic Changes in the Genome of Castration-Resistant Prostate Cancer 2011 ·Cancer Research ·Terence W. Friedlander, Ritu Roy, Scott A. Tomlins, Vy Ngo, Yasuko Kobayashi, (unknown), Mark A. Rubin, Kenneth J. Pienta, Arul M. Chinnaiyan, Michael Ittmann, Charles J. Ryan, Pamela L. Paris	88
14	Activation of NF-κB by TMPRSS2/ERG Fusion Isoforms through Toll-Like Receptor-4 2010 ·Cancer Research ·Jianghua Wang, Yi Cai, Longjiang Shao, Javed Siddiqui, Nallasivam Palanisamy, Rile Li, Chengxi Ren, Gustavo Ayala, Michael Ittmann	66
15	Identification of Differentially Methylated Genes in Normal Prostate Tissues from African American and Caucasian Men 2010 ·Clinical Cancer Research ·Bernard Kwabi‐Addo, Songping Wang, Woonbok Chung, Jaroslav Jelı́nek, Steven R. Patierno, Bi‐Dar Wang, Ramez Andrawis, Norman H. Lee, Victor Apprey, Jean‐Pierre J. Issa, Michael Ittmann	98
16	GGAP2/PIKE-A Directly Activates Both the Akt and Nuclear Factor-κB Pathways and Promotes Prostate Cancer Progression 2009 ·Cancer Research ·Yi Cai, Jianghua Wang, Rile Li, Gustavo Ayala, Michael Ittmann, Mingyao Liu	34
17	Global Gene Expression Analysis of Reactive Stroma in Prostate Cancer 2009 ·Clinical Cancer Research ·Olga Dakhova, Mustafa Özen, Chad J. Creighton, Rile Li, Gustavo Ayala, David R. Rowley, Michael Ittmann	125
18	Steroid Receptor Coactivator-3/AIB1 Promotes Cell Migration and Invasiveness through Focal Adhesion Turnover and Matrix Metalloproteinase Expression 2008 ·Cancer Research ·Jun Yan, Halime Erdem, Rile Li, Yi Cai, Gustavo Ayala, Michael Ittmann, Li‐Yuan Yu‐Lee, Sophia Y. Tsai, Ming‐Jer Tsai	90
19	Bortezomib-Mediated Inhibition of Steroid Receptor Coactivator-3 Degradation Leads to Activated Akt 2008 ·Clinical Cancer Research ·Gustavo Ayala, Jun Yan, Rile Li, Yi Ding, Timothy C. Thompson, Martha P. Mims, Teresa G. Hayes, (unknown), (unknown), Anna Frolov, Brian J. Miles, Thomas M. Wheeler, J. Wade Harper, Ming‐Jer Tsai, Michael Ittmann, Dov Kadmon	24
20	Androgens Modulate Expression of Transcription Intermediary Factor 2, an Androgen Receptor Coactivator whose Expression Level Correlates with Early Biochemical Recurrence in Prostate Cancer 2006 ·Cancer Research ·Irina U. Agoulnik, Ajula Vaid, Manjula Nakka, (unknown), William E. Bingman, Halime Erdem, Anna Frolov, C. Smith, Gustavo E. Ayala, Michael Ittmann, Nancy L. Weigel	139

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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