Gerald C. Chu

11.3k total citations · 3 hit papers
30 papers, 5.7k citations indexed

About

Gerald C. Chu is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Gerald C. Chu has authored 30 papers receiving a total of 5.7k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 12 papers in Oncology and 5 papers in Cell Biology. Recurrent topics in Gerald C. Chu's work include Pancreatic and Hepatic Oncology Research (7 papers), Epigenetics and DNA Methylation (4 papers) and Renal and related cancers (4 papers). Gerald C. Chu is often cited by papers focused on Pancreatic and Hepatic Oncology Research (7 papers), Epigenetics and DNA Methylation (4 papers) and Renal and related cancers (4 papers). Gerald C. Chu collaborates with scholars based in United States, Australia and United Kingdom. Gerald C. Chu's co-authors include Ronald A. DePinho, John P. Merlie, Alec C. Kimmelman, Aram F. Hezel, Douglas Hanahan, Ann–Hwee Lee, Laurie H. Glimcher, Neal N. Iwakoshi, Joshua R. Sanes and Peter G. Noakes and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Medicine.

In The Last Decade

Gerald C. Chu

29 papers receiving 5.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

p53 and Pten control neural and glioma stem/progenitor cell renewal and differentiation

2008 573 citations Hongwu Zheng, Haoqiang Ying et al. Nature profile →
Smad4 is dispensable for normal pancreas development yet critical in progression and tumor biology of pancreas cancer

2006 506 citations Nabeel Bardeesy, Kuang‐Hung Cheng et al. Genes & Development profile →
Autophagy Is Critical for Pancreatic Tumor Growth and Progression in Tumors with p53 Alterations

2014 384 citations Annan Yang, N.V. Rajeshkumar et al. Cancer Discovery profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Gerald C. Chu United States	26	3.7k	2.2k	1.1k	805	646	30	5.7k
Rosa Marina Melillo Italy	47	3.6k 1.0×	2.6k 1.2×	823 0.7×	427 0.5×	552 0.9×	100	6.8k
Earlene M. Schmitt United States	10	5.0k 1.3×	4.0k 1.8×	1.1k 1.0×	691 0.9×	320 0.5×	13	7.5k
Martin van der Valk Netherlands	33	4.0k 1.1×	3.6k 1.6×	703 0.6×	570 0.7×	418 0.6×	45	6.9k
Marc Billaud France	38	3.2k 0.9×	1.3k 0.6×	812 0.7×	355 0.4×	595 0.9×	84	5.1k
Karen Cichowski United States	35	3.6k 1.0×	1.5k 0.7×	840 0.7×	612 0.8×	319 0.5×	58	6.6k
Oliver Bögler United States	35	3.8k 1.0×	1.0k 0.5×	1.3k 1.2×	631 0.8×	194 0.3×	86	5.5k
Enhua Wang China	37	4.4k 1.2×	1.8k 0.8×	1.3k 1.2×	966 1.2×	368 0.6×	317	6.6k
Eva Y.-H.P. Lee United States	37	6.3k 1.7×	4.7k 2.1×	1.8k 1.5×	878 1.1×	241 0.4×	64	9.4k
Anna Lasorella United States	43	4.3k 1.2×	1.6k 0.7×	1.6k 1.4×	626 0.8×	385 0.6×	92	6.8k
Yosef Yarden Israel	27	3.2k 0.9×	1.6k 0.7×	765 0.7×	374 0.5×	344 0.5×	49	4.9k

Countries citing papers authored by Gerald C. Chu

Since Specialization

Citations

This map shows the geographic impact of Gerald C. Chu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gerald C. Chu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gerald C. Chu more than expected).

Fields of papers citing papers by Gerald C. Chu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gerald C. Chu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gerald C. Chu. The network helps show where Gerald C. Chu may publish in the future.

Co-authorship network of co-authors of Gerald C. Chu

This figure shows the co-authorship network connecting the top 25 collaborators of Gerald C. Chu. A scholar is included among the top collaborators of Gerald C. Chu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gerald C. Chu. Gerald C. Chu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Biancur, Douglas E., João A. Paulo, Beata Małachowska, et al.. (2017). Compensatory metabolic networks in pancreatic cancers upon perturbation of glutamine metabolism. Nature Communications. 8(1). 15965–15965. 229 indexed citations

Boutin, Adam T., Wenting Liao, Soyoon Hwang, et al.. (2017). Oncogenic Kras drives invasion and maintains metastases in colorectal cancer. Genes & Development. 31(4). 370–382. 137 indexed citations

Yang, Annan, N.V. Rajeshkumar, Xiaoxu Wang, et al.. (2014). Autophagy Is Critical for Pancreatic Tumor Growth and Progression in Tumors with p53 Alterations. Cancer Discovery. 4(8). 905–913. 384 indexed citations breakdown →

Ittmann, Michael, Jiaoti Huang, Enrico Radaelli, et al.. (2013). Animal Models of Human Prostate Cancer: The Consensus Report of the New York Meeting of the Mouse Models of Human Cancers Consortium Prostate Pathology Committee. Cancer Research. 73(9). 2718–2736. 184 indexed citations

Wang, Yufang, Sérgia Velho, Efsevia Vakiani, et al.. (2012). Mutant N-RAS Protects Colorectal Cancer Cells from Stress-Induced Apoptosis and Contributes to Cancer Development and Progression. Cancer Discovery. 3(3). 294–307. 42 indexed citations

Kwong, Lawrence N., James C. Costello, Huiyun Liu, et al.. (2012). Oncogenic NRAS signaling differentially regulates survival and proliferation in melanoma. Nature Medicine. 18(10). 1503–1510. 274 indexed citations

Moskowitz, Ivan P., Jun Wang, Michael Peterson, et al.. (2011). Transcription factor genes Smad4 and Gata4 cooperatively regulate cardiac valve development. Proceedings of the National Academy of Sciences. 108(10). 4006–4011. 77 indexed citations

Friedlander, Sharon, Gerald C. Chu, Eric L. Snyder, et al.. (2009). Context-Dependent Transformation of Adult Pancreatic Cells by Oncogenic K-Ras. Europe PMC (PubMed Central). 249 indexed citations

Nolan-Stevaux, Olivier, Janet Lau, Morgan Truitt, et al.. (2009). GLI1 is regulated through Smoothened-independent mechanisms in neoplastic pancreatic ducts and mediates PDAC cell survival and transformation. Genes & Development. 23(1). 24–36. 313 indexed citations

10.

Wiedemeyer, Ruprecht, Cameron Brennan, Timothy P. Heffernan, et al.. (2008). Feedback Circuit among INK4 Tumor Suppressors Constrains Human Glioblastoma Development. Cancer Cell. 13(4). 355–364. 93 indexed citations

11.

Zheng, Hongwu, Haoqiang Ying, Hai Yan, et al.. (2008). Pten and p53 Converge on c-Myc to Control Differentiation, Self-renewal, and Transformation of Normal and Neoplastic Stem Cells in Glioblastoma. Cold Spring Harbor Symposia on Quantitative Biology. 73(0). 427–437. 103 indexed citations

12.

Zheng, Hongwu, Haoqiang Ying, Haiyan Yan, et al.. (2008). p53 and Pten control neural and glioma stem/progenitor cell renewal and differentiation. Nature. 455(7216). 1129–1133. 573 indexed citations breakdown →

13.

Chu, Gerald C., Alec C. Kimmelman, Aram F. Hezel, & Ronald A. DePinho. (2007). Stromal biology of pancreatic cancer. Journal of Cellular Biochemistry. 101(4). 887–907. 258 indexed citations

14.

Bardeesy, Nabeel, Kuang‐Hung Cheng, Justin H. Berger, et al.. (2006). Smad4 is dispensable for normal pancreas development yet critical in progression and tumor biology of pancreas cancer. Genes & Development. 20(22). 3130–3146. 506 indexed citations breakdown →

15.

Bardeesy, Nabeel, Andrew J. Aguirre, Gerald C. Chu, et al.. (2006). Both p16 ^Ink4a and the p19 ^Arf -p53 pathway constrain progression of pancreatic adenocarcinoma in the mouse. Proceedings of the National Academy of Sciences. 103(15). 5947–5952. 420 indexed citations

16.

Lee, Ann–Hwee, Gerald C. Chu, Neal N. Iwakoshi, & Laurie H. Glimcher. (2005). XBP‐1 is required for biogenesis of cellular secretory machinery of exocrine glands. The EMBO Journal. 24(24). 4368–4380. 366 indexed citations

17.

Chu, Gerald C., N. Ray Dunn, Dorian C. Anderson, Leif Oxburgh, & Elizabeth J. Robertson. (2004). Differential requirements for Smad4 in TGFβ-dependent patterning of the early mouse embryo. Development. 131(15). 3501–3512. 191 indexed citations

18.

Chu, Gerald C., et al.. (1996). Maturation of the Acetylcholine Receptor in Skeletal Muscle: Regulation of the AChR γ-to-ϵ Switch. Developmental Biology. 179(1). 223–238. 166 indexed citations

19.

Moscoso, Lisa M., Gerald C. Chu, Medha Gautam, et al.. (1995). Synapse-Associated Expression of an Acetylcholine Receptor-Inducing Protein, ARIA/Heregulin, and Its Putative Receptors, ErbB2 and ErbB3, in Developing Mammalian Muscle. Developmental Biology. 172(1). 158–169. 160 indexed citations

20.

Gautam, Medha, Peter G. Noakes, Jacqueline L. Mudd, et al.. (1995). Failure of postsynaptic specialization to develop at neuromuscular junctions of rapsyn-deficient mice. Nature. 377(6546). 232–236. 450 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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