David G. Kirsch

20.3k total citations · 4 hit papers
263 papers, 13.3k citations indexed

About

David G. Kirsch is a scholar working on Pulmonary and Respiratory Medicine, Oncology and Molecular Biology. According to data from OpenAlex, David G. Kirsch has authored 263 papers receiving a total of 13.3k indexed citations (citations by other indexed papers that have themselves been cited), including 127 papers in Pulmonary and Respiratory Medicine, 91 papers in Oncology and 67 papers in Molecular Biology. Recurrent topics in David G. Kirsch's work include Sarcoma Diagnosis and Treatment (89 papers), Bone Tumor Diagnosis and Treatments (27 papers) and Vascular Tumors and Angiosarcomas (27 papers). David G. Kirsch is often cited by papers focused on Sarcoma Diagnosis and Treatment (89 papers), Bone Tumor Diagnosis and Treatments (27 papers) and Vascular Tumors and Angiosarcomas (27 papers). David G. Kirsch collaborates with scholars based in United States, Canada and South Korea. David G. Kirsch's co-authors include Michael B. Kastan, Tyler Jacks, J. Marie Hardwick, Everett J. Moding, Ralph Weissleder, Jan Grimm, Rollie J. Clem, Emily H. Cheng, Kazuyoshi Ueno and Andrea Ventura and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

David G. Kirsch

254 papers receiving 13.2k citations

Hit Papers

Restoration of p53 function leads to tumour regression in... 1997 2026 2006 2016 2007 1997 2021 2019 400 800 1.2k

Peers

David G. Kirsch
David G. Kirsch
Citations per year, relative to David G. Kirsch David G. Kirsch (= 1×) peers Kristian Pietras

Countries citing papers authored by David G. Kirsch

Since Specialization
Citations

This map shows the geographic impact of David G. Kirsch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David G. Kirsch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David G. Kirsch more than expected).

Fields of papers citing papers by David G. Kirsch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David G. Kirsch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David G. Kirsch. The network helps show where David G. Kirsch may publish in the future.

Co-authorship network of co-authors of David G. Kirsch

This figure shows the co-authorship network connecting the top 25 collaborators of David G. Kirsch. A scholar is included among the top collaborators of David G. Kirsch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David G. Kirsch. David G. Kirsch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Su, Chunxia, Collin Kent, Lixia Luo, et al.. (2024). Enhancing Radiotherapy Response via Intratumoral Injection of the TLR9 Agonist CpG to Stimulate CD8 T Cells in an Autochthonous Mouse Model of Sarcoma. International Journal of Radiation Oncology*Biology*Physics. 120(2). e487–e488. 2 indexed citations
2.
Hendrickson, Peter G., et al.. (2024). Response to Central Boost Radiation Therapy in Unresectable Retroperitoneal Sarcoma: A Case Series. Advances in Radiation Oncology. 10(2). 101689–101689. 1 indexed citations
3.
Brighton, Hailey E., Dharshan Sivaraj, Vardhman Kumar, et al.. (2022). Loss of ATRX promotes aggressive features of osteosarcoma with increased NF-κB signaling and integrin binding. JCI Insight. 7(17). 18 indexed citations
4.
Lee, Chang‐Lung, Andrea R. Daniel, Matt Holbrook, et al.. (2021). Characterization of cardiovascular injury in mice following partial-heart irradiation with clinically relevant dose and fractionation. Radiotherapy and Oncology. 157. 155–162. 12 indexed citations
5.
Rao, Sneha, Alexander L. Lazarides, Whitney O. Lane, et al.. (2021). Extent of tumor fibrosis/hyalinization and infarction following neoadjuvant radiation therapy is associated with improved survival in patients with soft‐tissue sarcoma. Cancer Medicine. 11(1). 194–206. 6 indexed citations
6.
Chen, Mark, et al.. (2021). Radiation-Induced Phosphorylation of a Prion-Like Domain Regulates Transformation by FUS-CHOP. Cancer Research. 81(19). 4939–4948. 6 indexed citations
7.
Torok, Jordan A., Katherine D. Castle, Yan Ma, et al.. (2018). Deletion of Atm in Tumor but not Endothelial Cells Improves Radiation Response in a Primary Mouse Model of Lung Adenocarcinoma. Cancer Research. 79(4). 773–782. 25 indexed citations
8.
Cheung, Phyllis F., Sven‐Thorsten Liffers, Kristina Althoff, et al.. (2018). Notch-Induced Myeloid Reprogramming in Spontaneous Pancreatic Ductal Adenocarcinoma by Dual Genetic Targeting. Cancer Research. 78(17). 4997–5010. 8 indexed citations
9.
Peacock, Jacqueline D., Elizabeth A. Tovar, Curt J. Essenburg, et al.. (2018). Genomic Status of MET Potentiates Sensitivity to MET and MEK Inhibition in NF1-Related Malignant Peripheral Nerve Sheath Tumors. Cancer Research. 78(13). 3672–3687. 30 indexed citations
10.
Wisdom, Amy J., Katherine D. Castle, Yvonne M. Mowery, et al.. (2018). Characterizing the Potency and Impact of Carbon Ion Therapy in a Primary Mouse Model of Soft Tissue Sarcoma. Molecular Cancer Therapeutics. 17(4). 858–868. 23 indexed citations
11.
Mater, David Van, Eric S. Xu, Anupama Reddy, et al.. (2018). Injury promotes sarcoma development in a genetically and temporally restricted manner. JCI Insight. 3(20). 10 indexed citations
12.
Dodd, Rebecca D., Chang‐Lung Lee, Wesley Huang, et al.. (2017). NF1+/− Hematopoietic Cells Accelerate Malignant Peripheral Nerve Sheath Tumor Development without Altering Chemotherapy Response. Cancer Research. 77(16). 4486–4497. 21 indexed citations
13.
Coleman, C. Norman, Geoff S. Higgins, J. Martin Brown, et al.. (2016). Improving the Predictive Value of Preclinical Studies in Support of Radiotherapy Clinical Trials. Clinical Cancer Research. 22(13). 3138–3147. 59 indexed citations
14.
Mater, David Van, Jordan M. Blum, Micah T. Webster, et al.. (2014). Acute Tissue Injury Activates Satellite Cells and Promotes Sarcoma Formation via the HGF/c-MET Signaling Pathway. Cancer Research. 75(3). 605–614. 24 indexed citations
15.
Dodd, Rebecca D., Jeffrey K. Mito, William C. Eward, et al.. (2013). NF1 Deletion Generates Multiple Subtypes of Soft-Tissue Sarcoma That Respond to MEK Inhibition. Molecular Cancer Therapeutics. 12(9). 1906–1917. 62 indexed citations
16.
Eisinger‐Mathason, T.S. Karin, Minsi Zhang, Qiong Qiu, et al.. (2013). Hypoxia-Dependent Modification of Collagen Networks Promotes Sarcoma Metastasis. Cancer Discovery. 3(10). 1190–1205. 213 indexed citations
17.
Li, Zhizhong, Yunyu Zhang, Krishnan Ramanujan, et al.. (2013). Oncogenic NRAS, Required for Pathogenesis of Embryonic Rhabdomyosarcoma, Relies upon the HMGA2–IGF2BP2 Pathway. Cancer Research. 73(10). 3041–3050. 47 indexed citations
18.
Kirsch, David G., Scott H. Okuno, John M. Kane, et al.. (2011). RTOG Phase II Trial of Preoperative Image Guided Radiotherapy (IG-RT) For Primary Soft Tissue Sarcoma of the Extremity: Acute Toxicity Report. International Journal of Radiation Oncology*Biology*Physics. 81(2). S117–S117. 8 indexed citations
19.
Kirsch, David G., Philip M. Santiago, Emmanuelle di Tomaso, et al.. (2009). p53 Controls Radiation-Induced Gastrointestinal Syndrome in Mice Independent of Apoptosis. Science. 327(5965). 593–596. 1 indexed citations
20.
Perez, Bradford A., S. M. Johnston, Laura B. Jeffords, et al.. (2009). Dissecting the Mechanism of Tumor Response to Radiation Therapy with Primary Lung Cancers in Mice. International Journal of Radiation Oncology*Biology*Physics. 75(3). S537–S537. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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