Matthias Schröder
- Molecular Biology
- Immunology
- Oncology
- Radiology, Nuclear Medicine and Imaging
- Pulmonary and Respiratory Medicine
- Co-authors
- Christopher BachranLee Kim SweeHeinfried H. RadekeRegina ScherließJosef PfeilschifterStefan ZausingerA. BaethmannEdwin Hungerhuber
- Topics
- Immune Cell Function and Interaction (6 papers)Immune cells in cancer (4 papers)Sphingolipid Metabolism and Signaling (4 papers)
- Partner nations
- GermanySwitzerlandUnited States
In The Last Decade
Matthias Schröder
26 papers receiving 318 citations
Peers
Comparison fields: 5 of 73
- Molecular Biology 154
- Immunology 115
- Oncology 57
- Radiology, Nuclear Medicine and Imaging 28
- Pulmonary and Respiratory Medicine 24
Countries citing papers authored by Matthias Schröder
This map shows the geographic impact of Matthias Schröder's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthias Schröder with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthias Schröder more than expected).
Fields of papers citing papers by Matthias Schröder
This network shows the impact of papers produced by Matthias Schröder. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthias Schröder. The network helps show where Matthias Schröder may publish in the future.
Co-authorship network of co-authors of Matthias Schröder
This figure shows the co-authorship network connecting the top 25 collaborators of Matthias Schröder. A scholar is included among the top collaborators of Matthias Schröder based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthias Schröder. Matthias Schröder is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 11 | |
| 3 | 5 | |
| 4 | 24 | |
| 5 | 18 | |
| 6 | 20 | |
| 7 | 11 | |
| 8 | 17 | |
| 9 | 52 | |
| 10 | 14 | |
| 11 | 8 | |
| 12 | 8 | |
| 13 | 8 | |
| 14 | 16 | |
| 15 | 17 | |
| 16 | 1 | |
| 17 | 29 | |
| 18 | 34 | |
| 19 | 4 | |
| 20 | [Studies on the genetics of otosclerosis (author's transl)]. | 1 |
About Matthias Schröder
Matthias Schröder is a scholar working on Immunology, Clinical Biochemistry and Critical Care and Intensive Care Medicine, having authored 28 papers that have together received 325 indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (6 papers), Immune cells in cancer (4 papers) and Sphingolipid Metabolism and Signaling (4 papers). The work is most often cited by research in Immunology (115 citations), Developmental Neuroscience (20 citations) and Pharmaceutical Science (18 citations). Matthias Schröder has collaborated with scholars based in Germany, Switzerland and United States. Frequent co-authors include Christopher Bachran, Lee Kim Swee, Heinfried H. Radeke, Regina Scherließ, Josef Pfeilschifter, Stefan Zausinger, A. Baethmann, Edwin Hungerhuber, Laura Helming and Robert Schmid-Elsaesser. Their work appears in journals such as Circulation, The Journal of Immunology and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.