Thomas Bugge

14.4k citations
165 papers · 11.2k indexed · 1 hit paper · h-index 62

Thomas Bugge

164 papers receiving 11.0k citations

Hit Papers

Neutrophil extracellular traps and extracellular histones...902023202620242025255075

Peers

Thomas Bugge
Comparison fields: 5 of 144
  • Immunology and Allergy 1.5k
  • Hematology 2.0k
  • Cancer Research 2.4k
  • Hepatology 668
  • Genetics 850
Replace Jay L. Degen with:
Jay L. Degen United States
Fernando Arenzana‐Seisdedos France
James P. Quigley United States
Steven T. Pals Netherlands
Ramesh A. Shivdasani United States
Charles T. Rauch United States
Alex Y. Strongin United States
Austin Gurney United States
Ofer Lider Israel
Ellson Y. Chen United States
Thomas Bugge relative to Jay L. Degen United States Jay L. Degen's profile →
Citations per field
00.5×1.5×
Jay L. Degen · 1×
Citations per year

Countries citing papers authored by Thomas Bugge

Since Specialization
Citations

This map shows the geographic impact of Thomas Bugge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Bugge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Bugge more than expected).

Fields of papers citing papers by Thomas Bugge

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Bugge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Bugge. The network helps show where Thomas Bugge may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Thomas Bugge, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thomas Bugge Line = papers co-authored together Thomas Bugge links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 202219
2 202210
3 20229
4 201933
5 201813
6 201720
7 201536
8 201429
9 201196
10 201072
11 201026
12 200916
13 200938
14 200816
15 200724
16 200774
17 2002111
18 199952
19 1997274
20 19921

About Thomas Bugge

Thomas Bugge is a scholar working on Immunology and Allergy, Virology and Hematology, having authored 165 papers that have together received 11.2k indexed citations. Recurring topics across this work include Bacillus and Francisella bacterial research (32 papers), Protease and Inhibitor Mechanisms (30 papers), Blood Coagulation and Thrombosis Mechanisms (27 papers), Cell Adhesion Molecules Research (27 papers), Microbial Inactivation Methods (16 papers), Liver physiology and pathology (11 papers), Peptidase Inhibition and Analysis (10 papers) and Toxin Mechanisms and Immunotoxins (9 papers). The work is most often cited by research in Immunology and Allergy (1.5k citations), Hematology (2.0k citations) and Cancer Research (2.4k citations). Thomas Bugge has collaborated with scholars based in United States, Denmark and Germany. Frequent co-authors include Roman Szabo, Jay L. Degen, Karin List, Toni Antalis, Keith W. Kombrinck, J. Silvio Gutkind, Shihui Liu, Qingyu Wu, Stephen H. Leppla and Alfredo Molinolo. Their work appears in journals such as Journal of Biological Chemistry, Blood, Oncogene, Proceedings of the National Academy of Sciences and PLoS ONE.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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