Kathryn E. Talmage

2.3k total citations · 1 hit paper
18 papers, 1.8k citations indexed

About

Kathryn E. Talmage is a scholar working on Hematology, Immunology and Allergy and Molecular Biology. According to data from OpenAlex, Kathryn E. Talmage has authored 18 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Hematology, 5 papers in Immunology and Allergy and 4 papers in Molecular Biology. Recurrent topics in Kathryn E. Talmage's work include Platelet Disorders and Treatments (9 papers), Blood Coagulation and Thrombosis Mechanisms (7 papers) and Cell Adhesion Molecules Research (5 papers). Kathryn E. Talmage is often cited by papers focused on Platelet Disorders and Treatments (9 papers), Blood Coagulation and Thrombosis Mechanisms (7 papers) and Cell Adhesion Molecules Research (5 papers). Kathryn E. Talmage collaborates with scholars based in United States, Australia and Canada. Kathryn E. Talmage's co-authors include Joseph S. Palumbo, Jay L. Degen, Matthew J. Flick, Keith W. Kombrinck, Markéta Jiroušková, Kelley A. Barney, David P. Witte, David G. Jackson, Zhiwei Hu and Sherry Thornton and has published in prestigious journals such as Journal of Clinical Investigation, Blood and Cancer Research.

In The Last Decade

Kathryn E. Talmage

18 papers receiving 1.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Platelets and fibrin(ogen) increase metastatic potential by impeding natural killer cell–mediated elimination of tumor cells

2004 779 citations Joseph S. Palumbo, Kathryn E. Talmage et al. Blood profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kathryn E. Talmage United States	11	886	642	429	396	382	18	1.8k
Sebastian F. Mause Germany	10	328 0.4×	416 0.6×	150 0.3×	326 0.8×	917 2.4×	19	1.7k
Évelyne Dupuy France	23	152 0.2×	701 1.1×	292 0.7×	284 0.7×	443 1.2×	62	1.7k
Scott T. Avecilla United States	14	390 0.4×	911 1.4×	123 0.3×	176 0.4×	515 1.3×	36	1.6k
Ryuichi Kamiyama Japan	24	435 0.5×	658 1.0×	193 0.4×	130 0.3×	548 1.4×	75	1.8k
Ingegerd Lecander Sweden	26	165 0.2×	886 1.4×	193 0.4×	970 2.4×	203 0.5×	57	1.7k
Osnat Bairey Israel	27	598 0.7×	338 0.5×	247 0.6×	160 0.4×	542 1.4×	103	2.5k
G Tobelem France	19	168 0.2×	889 1.4×	183 0.4×	167 0.4×	524 1.4×	80	1.8k
Thomas Moehler Germany	25	739 0.8×	1.4k 2.1×	85 0.2×	148 0.4×	1.1k 2.8×	69	2.1k
Carlos Panizo Spain	19	463 0.5×	287 0.4×	172 0.4×	45 0.1×	253 0.7×	93	1.5k
R Hoffman United States	24	374 0.4×	1.4k 2.2×	163 0.4×	40 0.1×	516 1.4×	68	2.0k

Countries citing papers authored by Kathryn E. Talmage

Since Specialization

Citations

This map shows the geographic impact of Kathryn E. Talmage's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kathryn E. Talmage with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kathryn E. Talmage more than expected).

Fields of papers citing papers by Kathryn E. Talmage

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kathryn E. Talmage. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kathryn E. Talmage. The network helps show where Kathryn E. Talmage may publish in the future.

Co-authorship network of co-authors of Kathryn E. Talmage

This figure shows the co-authorship network connecting the top 25 collaborators of Kathryn E. Talmage. A scholar is included among the top collaborators of Kathryn E. Talmage based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kathryn E. Talmage. Kathryn E. Talmage is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

18 of 18 papers shown

Raghu, Harini, et al.. (2011). Plasminogen Mediates Both Positive and Negative Effects on Disease Severity in a Mouse Model of TNFα-Driven Arthritis. Blood. 118(21). 854–854. 1 indexed citations

Flick, Matthew J., Anil K. Chauhan, Kathryn E. Talmage, et al.. (2011). The development of inflammatory joint disease is attenuated in mice expressing the anticoagulant prothrombin mutant W215A/E217A. Blood. 117(23). 6326–6337. 29 indexed citations

Horowitz, Netanel A., Kathryn E. Talmage, Eric S. Mullins, et al.. (2011). Thrombomodulin is a determinant of metastasis through a mechanism linked to the thrombin binding domain but not the lectin-like domain. Blood. 118(10). 2889–2895. 71 indexed citations

Horowitz, Netanel A., Kathryn E. Talmage, Eric S. Mullins, et al.. (2010). Thrombin-Thrombomodulin Interactions Are An Important Determinant of Metastatic Potential. Blood. 116(21). 822–822. 1 indexed citations

Steinbrecher, Kris A., Netanel A. Horowitz, Kelley A. Barney, et al.. (2010). Colitis-Associated Cancer Is Dependent on the Interplay between the Hemostatic and Inflammatory Systems and Supported by Integrin αMβ2 Engagement of Fibrinogen. Cancer Research. 70(7). 2634–2643. 136 indexed citations

Palumbo, Joseph S., Kelley A. Barney, Allan Mishra, et al.. (2008). Factor XIII transglutaminase supports hematogenous tumor cell metastasis through a mechanism dependent on natural killer cell function. Journal of Thrombosis and Haemostasis. 6(5). 812–819. 77 indexed citations

Mullins, Eric S., Keith W. Kombrinck, Kathryn E. Talmage, et al.. (2008). Genetic elimination of prothrombin in adult mice is not compatible with survival and results in spontaneous hemorrhagic events in both heart and brain. Blood. 113(3). 696–704. 49 indexed citations

Mullins, Eric S., Keith W. Kombrinck, Kathryn E. Talmage, et al.. (2008). Genetic Elimination of Prothrombin in Adult Mice Results in Fatal Spontaneous Hemorrhage in the Heart and Central Nervous System. Blood. 112(11). 395–395. 1 indexed citations

Flick, Matthew J., Kathryn E. Talmage, David P. Witte, et al.. (2007). Fibrin(ogen) exacerbates inflammatory joint disease through a mechanism linked to the integrin αMβ2 binding motif. Journal of Clinical Investigation. 117(11). 3224–3235. 132 indexed citations

10.

Palumbo, Joseph S., Kathryn E. Talmage, Matthew J. Flick, et al.. (2007). Tumor cell–associated tissue factor and circulating hemostatic factors cooperate to increase metastatic potential through natural killer cell–dependent and–independent mechanisms. Blood. 110(1). 133–141. 237 indexed citations

11.

Palumbo, Joseph S., Kelley A. Barney, Matthew J. Flick, et al.. (2006). Tumor Cell-Associated Tissue Factor Supports Metastatic Potential through Both NK Cell-Dependent and -Independent Mechanisms.. Blood. 108(11). 66–66. 1 indexed citations

12.

Flick, Matthew J., Kathryn E. Talmage, David P. Witte, et al.. (2006). Fibrin(ogen) Exacerbates Inflammatory Joint Disease Via a Mechanism Linked to Its αMβ2 Binading Motif.. Blood. 108(11). 64–64. 1 indexed citations

13.

Palumbo, Joseph S., et al.. (2005). Interplay between Tumor Cell-Associated and Circulating Coagulation Factors in Establishing Metastatic Potential.. Blood. 106(11). 686–686. 1 indexed citations

14.

Palumbo, Joseph S., Mark Zogg, Kathryn E. Talmage, et al.. (2004). Role of fibrinogen‐ and platelet‐mediated hemostasis in mouse embryogenesis and reproduction. Journal of Thrombosis and Haemostasis. 2(8). 1368–1379. 31 indexed citations

15.

Palumbo, Joseph S., et al.. (2004). Hemostatic Factors Contribute to Tumor Cell Metastatic Potential by a Mechanism Linked to Natural Killer Cell Function.. Blood. 104(11). 690–690. 4 indexed citations

16.

Palumbo, Joseph S., Kathryn E. Talmage, Matthew J. Flick, et al.. (2004). Platelets and fibrin(ogen) increase metastatic potential by impeding natural killer cell–mediated elimination of tumor cells. Blood. 105(1). 178–185. 779 indexed citations breakdown →

17.

Palumbo, Joseph S., et al.. (2003). Plasminogen supports tumor growth through a fibrinogen-dependent mechanism linked to vascular patency. Blood. 102(8). 2819–2827. 59 indexed citations

18.

Palumbo, Joseph S., et al.. (2002). Spontaneous hematogenous and lymphatic metastasis, but not primary tumor growth or angiogenesis, is diminished in fibrinogen-deficient mice.. PubMed. 62(23). 6966–72. 215 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact