Brian P. Conlon
- Molecular Biology top 5%
- Infectious Diseases top 2%
- Molecular Medicine top 0.5%
- Genetics top 2%
- Microbiology top 1%
- Co-authors
- Sarah E. RoweKim LewisAutumn Brown GandtJoshua AdkinsAustin S. NuxollLauren C. RadlinskiEliza A. ZalisGérémy Clair
- Topics
- Antimicrobial Resistance in Staphylococcus (24 papers)Bacterial biofilms and quorum sensing (20 papers)Antibiotic Resistance in Bacteria (14 papers)
- Partner nations
- United StatesIrelandGermany
In The Last Decade
Brian P. Conlon
36 papers receiving 2.8k citations
Hit Papers
Peers
Comparison fields: 5 of 108
- Molecular Biology 1.7k
- Infectious Diseases 929
- Molecular Medicine 791
- Genetics 732
- Microbiology 446
Countries citing papers authored by Brian P. Conlon
This map shows the geographic impact of Brian P. Conlon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brian P. Conlon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brian P. Conlon more than expected).
Fields of papers citing papers by Brian P. Conlon
This network shows the impact of papers produced by Brian P. Conlon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brian P. Conlon. The network helps show where Brian P. Conlon may publish in the future.
Co-authorship network of co-authors of Brian P. Conlon
This figure shows the co-authorship network connecting the top 25 collaborators of Brian P. Conlon. A scholar is included among the top collaborators of Brian P. Conlon based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brian P. Conlon. Brian P. Conlon is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 1 | |
| 4 | 0 | |
| 5 | 15 | |
| 6 | 6 | |
| 7 | 11 | |
| 8 | 7 | |
| 9 | 7 | |
| 10 | 14 | |
| 11 | 23 | |
| 12 | 25 | |
| 13 | 13 | |
| 14 | 27 | |
| 15 | 4 | |
| 16 | 33 | |
| 17 | ATP-Dependent Persister Formation in Escherichia colibreakdown → | 332 |
| 18 | 135 | |
| 19 | Persister formation in Staphylococcus aureus is associated with ATP depletionbreakdown → | 472 |
| 20 | 120 |
About Brian P. Conlon
Brian P. Conlon is a scholar working on Molecular Medicine, Infectious Diseases and Microbiology, having authored 39 papers that have together received 2.8k indexed citations. Recurring topics across this work include Antimicrobial Resistance in Staphylococcus (24 papers), Bacterial biofilms and quorum sensing (20 papers) and Antibiotic Resistance in Bacteria (14 papers). The work is most often cited by research in Molecular Medicine (791 citations), Microbiology (446 citations) and Endocrinology (362 citations). Brian P. Conlon has collaborated with scholars based in United States, Ireland and Germany. Frequent co-authors include Sarah E. Rowe, Kim Lewis, Autumn Brown Gandt, Joshua Adkins, Kim Lewis, Austin S. Nuxoll, Lauren C. Radlinski, Eliza A. Zalis, Gérémy Clair and James P. O’Gara. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.