Matthew C. Kostek
- Molecular Biology top 10%
- Physiology top 5%
- Cell Biology top 5%
- Orthopedics and Sports Medicine top 2%
- Genetics top 10%
- Co-authors
- Matthew J. DelmonicoJames A. CarsonBradley S. GordonShuichi SatoJames P. WhiteJohn BaynesLydia E. MatesicStephen M. Roth
- Topics
- Muscle Physiology and Disorders (22 papers)Exercise and Physiological Responses (15 papers)Genetics and Physical Performance (10 papers)
- Partner nations
- United StatesColombiaIreland
In The Last Decade
Matthew C. Kostek
39 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 698
- Physiology 697
- Cell Biology 322
- Orthopedics and Sports Medicine 302
- Genetics 298
Countries citing papers authored by Matthew C. Kostek
This map shows the geographic impact of Matthew C. Kostek's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew C. Kostek with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew C. Kostek more than expected).
Fields of papers citing papers by Matthew C. Kostek
This network shows the impact of papers produced by Matthew C. Kostek. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew C. Kostek. The network helps show where Matthew C. Kostek may publish in the future.
Co-authorship network of co-authors of Matthew C. Kostek
This figure shows the co-authorship network connecting the top 25 collaborators of Matthew C. Kostek. A scholar is included among the top collaborators of Matthew C. Kostek based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew C. Kostek. Matthew C. Kostek is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 19 | |
| 2 | 6 | |
| 3 | 110 | |
| 4 | 16 | |
| 5 | 9 | |
| 6 | 2 | |
| 7 | Exercise Increases Utrophin Protein Expression in the MDX Mouse Model of Duchenne Muscular Dystrophy | 0 |
| 8 | 180 | |
| 9 | 9 | |
| 10 | 29 | |
| 11 | 210 | |
| 12 | 37 | |
| 13 | 60 | |
| 14 | 125 | |
| 15 | 5 | |
| 16 | 39 | |
| 17 | 23 | |
| 18 | 88 | |
| 19 | 1 | |
| 20 | 121 |
About Matthew C. Kostek
Matthew C. Kostek is a scholar working on Rehabilitation, Geriatrics and Gerontology and Complementary and alternative medicine, having authored 42 papers that have together received 1.5k indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (22 papers), Exercise and Physiological Responses (15 papers) and Genetics and Physical Performance (10 papers). The work is most often cited by research in Orthopedics and Sports Medicine (302 citations), Rehabilitation (227 citations) and Physiology (697 citations). Matthew C. Kostek has collaborated with scholars based in United States, Colombia and Ireland. Frequent co-authors include Matthew J. Delmonico, James A. Carson, Bradley S. Gordon, Shuichi Sato, James P. White, John Baynes, Lydia E. Matesic, Stephen M. Roth, Ben F. Hurley and Joan M. Conway. Their work appears in journals such as PLoS ONE, Scientific Reports and The FASEB Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.