Mark T. Keating
- Cardiology and Cardiovascular Medicine top 0.02%
- Cardiac electrophysiology and arrhythmias 49
- Cardiomyopathy and Myosin Studies 15
- Developmental Neuroscience top 0.1%
- Williams Syndrome Research 13
- Molecular Biology top 0.05%
- Ion channel regulation and function 44
- Congenital heart defects research 10
- Developmental Biology and Gene Regulation 9
- Receptor Mechanisms and Signaling 7
- Ion Transport and Channel Regulation 6
- Cellular and Molecular Neuroscience top 0.1%
- Cell Biology top 0.5%
- Co-authors
- Michael C. SanguinettiMark CurranIgor SplawskiKatherine W. TimothyKenneth D. PossDonald L. AtkinsonPeter J. SchwartzJeffrey A. Towbin
- Partner nations
- United StatesItalyPoland
In The Last Decade
Mark T. Keating
117 papers receiving 30.8k citations
Hit Papers
Peers
Comparison fields: 5 of 165
- Cardiology and Cardiovascular Medicine 19.7k
- Developmental Neuroscience 1.7k
- Molecular Biology 24.3k
- Cellular and Molecular Neuroscience 5.1k
- Cell Biology 1.9k
Countries citing papers authored by Mark T. Keating
This map shows the geographic impact of Mark T. Keating's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark T. Keating with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark T. Keating more than expected).
Fields of papers citing papers by Mark T. Keating
This network shows the impact of papers produced by Mark T. Keating. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark T. Keating. The network helps show where Mark T. Keating may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Mark T. Keating, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2022 | 19 | |
| 2 | 2021 | 11 | |
| 3 | 2007 | 124 | |
| 4 | 2006 | 290 | |
| 5 | Abstract 2439: Clinical Profile and Risk of Sudden Death in Children with Timothy Syndrome | 2006 | 1 |
| 6 | 2006 | 93 | |
| 7 | 2006 | 182 | |
| 8 | 2005 | 456 | |
| 9 | 2004 | 134 | |
| 10 | 2003 | 56 | |
| 11 | 2001 | 177 | |
| 12 | 2000 | 129 | |
| 13 | 2000 | 268 | |
| 14 | 1999 | 3 | |
| 15 | 1999 | 185 | |
| 16 | 1998 | 262 | |
| 17 | 1998 | 188 | |
| 18 | Mutations in the hminK gene cause long QT syndrome and suppress lKs functionbreakdown → | 1997 | 581 |
| 19 | 1996 | 138 | |
| 20 | A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndromebreakdown → | 1995 | 1746 |
About Mark T. Keating
Mark T. Keating is a scholar working on Developmental Neuroscience, Cardiology and Cardiovascular Medicine, Molecular Biology, Cell Biology and Immunology and Allergy, having authored 117 papers that have together received 31.6k indexed citations. Recurring topics across this work include Cardiac electrophysiology and arrhythmias (49 papers), Ion channel regulation and function (44 papers), Cardiomyopathy and Myosin Studies (15 papers), Williams Syndrome Research (13 papers), Congenital heart defects research (10 papers), Developmental Biology and Gene Regulation (9 papers), Receptor Mechanisms and Signaling (7 papers) and Ion Transport and Channel Regulation (6 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (19.7k citations), Developmental Neuroscience (1.7k citations), Molecular Biology (24.3k citations), Cellular and Molecular Neuroscience (5.1k citations) and Cell Biology (1.9k citations). Mark T. Keating has collaborated with scholars based in United States, Italy and Poland. Frequent co-authors include Michael C. Sanguinetti, Mark Curran, Igor Splawski, Katherine W. Timothy, Kenneth D. Poss, Donald L. Atkinson, Peter J. Schwartz, Jeffrey A. Towbin, Michael H. Lehmann and Jiaxiang Shen. Their work appears in journals such as Circulation, Journal of Biological Chemistry, Cell, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.