Lisa Smithson

2.1k total citations
16 papers, 1.4k citations indexed

About

Lisa Smithson is a scholar working on Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Lisa Smithson has authored 16 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Physiology and 5 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Lisa Smithson's work include Alzheimer's disease research and treatments (8 papers), Prion Diseases and Protein Misfolding (5 papers) and Cardiac Fibrosis and Remodeling (4 papers). Lisa Smithson is often cited by papers focused on Alzheimer's disease research and treatments (8 papers), Prion Diseases and Protein Misfolding (5 papers) and Cardiac Fibrosis and Remodeling (4 papers). Lisa Smithson collaborates with scholars based in United States, Netherlands and Italy. Lisa Smithson's co-authors include Todd E. Golde, Robert Price, Ramesh Natarajan, Ramzi Ockaili, Jungsu Kim, Harm Jan Bogaard, Donatas Kraskauskas, Joe M. McCord, Norbert F. Voelkel and Carlin S. Long and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of Neuroscience.

In The Last Decade

Lisa Smithson

16 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lisa Smithson United States 14 553 553 390 363 173 16 1.4k
Libang Yang United States 16 635 1.1× 627 1.1× 71 0.2× 207 0.6× 176 1.0× 31 1.7k
Zhihua Qiu China 18 644 1.2× 214 0.4× 146 0.4× 69 0.2× 72 0.4× 43 1.3k
Zhiming Suo United States 18 464 0.8× 696 1.3× 96 0.2× 102 0.3× 116 0.7× 23 1.5k
Roberto Piñeiro Spain 20 616 1.1× 269 0.5× 238 0.6× 137 0.4× 165 1.0× 44 2.1k
Matthew Wszolek United States 18 723 1.3× 518 0.9× 56 0.1× 293 0.8× 166 1.0× 41 2.0k
Paul Thornhill United Kingdom 8 560 1.0× 311 0.6× 183 0.5× 27 0.1× 132 0.8× 8 1.5k
Margot C. LaPointe United States 31 1.0k 1.8× 341 0.6× 830 2.1× 97 0.3× 389 2.2× 48 2.2k
Huang Guo United States 17 601 1.1× 910 1.6× 90 0.2× 47 0.1× 155 0.9× 19 2.2k
Ian McPhee United Kingdom 22 1.7k 3.0× 207 0.4× 249 0.6× 93 0.3× 694 4.0× 36 2.2k
Michail A. Esterman United States 11 599 1.1× 397 0.7× 76 0.2× 55 0.2× 106 0.6× 16 1.2k

Countries citing papers authored by Lisa Smithson

Since Specialization
Citations

This map shows the geographic impact of Lisa Smithson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lisa Smithson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lisa Smithson more than expected).

Fields of papers citing papers by Lisa Smithson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lisa Smithson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lisa Smithson. The network helps show where Lisa Smithson may publish in the future.

Co-authorship network of co-authors of Lisa Smithson

This figure shows the co-authorship network connecting the top 25 collaborators of Lisa Smithson. A scholar is included among the top collaborators of Lisa Smithson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lisa Smithson. Lisa Smithson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Park, Hyo‐Jin, Yong Ran, Joo In Jung, et al.. (2015). The stress response neuropeptide CRF increases amyloid‐β production by regulating γ‐secretase activity. The EMBO Journal. 34(12). 1674–1686. 41 indexed citations
2.
Jung, Joo In, Ashleigh Price, Thomas B. Ladd, et al.. (2015). Cholestenoic acid, an endogenous cholesterol metabolite, is a potent γ-secretase modulator. Molecular Neurodegeneration. 10(1). 29–29. 16 indexed citations
3.
Abbate, Antonio, Benjamín W. Van Tassell, Ignacio M. Seropián, et al.. (2010). Interleukin-1β Modulation Using a Genetically Engineered Antibody Prevents Adverse Cardiac Remodelling Following Acute Myocardial Infarction in the Mouse. European Journal of Heart Failure. 12(4). 319–322. 96 indexed citations
4.
Seropián, Ignacio M., Antonio Abbate, Stefano Toldo, et al.. (2010). Pharmacologic Inhibition of Phosphoinositide 3-Kinase Gamma (PI3Kγ) Promotes Infarct Resorption and Prevents Adverse Cardiac Remodeling After Myocardial Infarction in Mice. Journal of Cardiovascular Pharmacology. 56(6). 651–658. 23 indexed citations
5.
Tassell, Benjamín Van, Ignacio M. Seropián, Stefano Toldo, et al.. (2010). Pharmacologic Inhibition of Myeloid Differentiation Factor 88 (MyD88) Prevents Left Ventricular Dilation and Hypertrophy After Experimental Acute Myocardial Infarction in the Mouse. Journal of Cardiovascular Pharmacology. 55(4). 385–390. 53 indexed citations
6.
Tassell, Benjamín Van, Amit Varma, Fadi N. Salloum, et al.. (2010). Interleukin-1 Trap Attenuates Cardiac Remodeling After Experimental Acute Myocardial Infarction in Mice. Journal of Cardiovascular Pharmacology. 55(2). 117–122. 66 indexed citations
7.
Bogaard, Harm Jan, Ramesh Natarajan, Scott C. Henderson, et al.. (2009). Chronic Pulmonary Artery Pressure Elevation Is Insufficient to Explain Right Heart Failure. Circulation. 120(20). 1951–1960. 390 indexed citations
8.
Natarajan, Ramesh, Fadi N. Salloum, Bernard Fisher, et al.. (2009). Prolyl hydroxylase inhibition attenuates post-ischemic cardiac injury via induction of endoplasmic reticulum stress genes. Vascular Pharmacology. 51(2-3). 110–118. 34 indexed citations
9.
Bogaard, Harm Jan, Ramesh Natarajan, Donatas Kraskauskas, Lisa Smithson, & NF Voelkel. (2009). Suppression of Histone Deacetylases Worsens Right Ventricular Hypertrophy and Induces Failure after Pulmonary Artery Banding in Rats.. A4142–A4142. 1 indexed citations
10.
Kim, Jungsu, Virginia M. Miller, Yona Levites, et al.. (2008). BRI2 (ITM2b) Inhibits A  Deposition In Vivo. Journal of Neuroscience. 28(23). 6030–6036. 94 indexed citations
11.
Kim, Jungsu, Luisa Onstead, Suzanne J. Randle, et al.. (2007). Aβ40 Inhibits Amyloid DepositionIn Vivo. Journal of Neuroscience. 27(3). 627–633. 299 indexed citations
12.
Li, Tong, Hongjin Wen, Cory Brayton, et al.. (2007). Epidermal Growth Factor Receptor and Notch Pathways Participate in the Tumor Suppressor Function of γ-Secretase. Journal of Biological Chemistry. 282(44). 32264–32273. 74 indexed citations
13.
Lawlor, P., Ross Bland, Pritam Das, et al.. (2007). Novel rat Alzheimer's disease models based on AAV-mediated gene transfer to selectively increase hippocampal Aβ levels. Molecular Neurodegeneration. 2(1). 11–11. 54 indexed citations
14.
15.
Das, Pritam, Lisa Smithson, Robert Price, et al.. (2006). Interleukin-1 receptor 1 knockout has no effect on amyloid deposition in Tg2576 mice and does not alter efficacy following Abeta immunotherapy.. Journal of Neuroinflammation. 3(1). 17–17. 9 indexed citations
16.
Levites, Yona, Lisa Smithson, Robert Price, et al.. (2006). Insights into the mechanisms of action of anti‐Aβ antibodies in Alzheimer's disease mouse models. The FASEB Journal. 20(14). 2576–2578. 93 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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