Kazuhiko Watabe

3.4k total citations
116 papers, 2.8k citations indexed

About

Kazuhiko Watabe is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, Kazuhiko Watabe has authored 116 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 48 papers in Cellular and Molecular Neuroscience, 37 papers in Neurology and 33 papers in Molecular Biology. Recurrent topics in Kazuhiko Watabe's work include Nerve injury and regeneration (42 papers), Amyotrophic Lateral Sclerosis Research (22 papers) and Neurogenesis and neuroplasticity mechanisms (16 papers). Kazuhiko Watabe is often cited by papers focused on Nerve injury and regeneration (42 papers), Amyotrophic Lateral Sclerosis Research (22 papers) and Neurogenesis and neuroplasticity mechanisms (16 papers). Kazuhiko Watabe collaborates with scholars based in Japan, Canada and United States. Kazuhiko Watabe's co-authors include Toya Ohashi, Kazunori Sango, Kiyomitsu Oyanagi, Yoshikatsu Eto, Seung Up Kim, Hiroko Yanagisawa, Shizuka Takaku, Emiko Kawakami, Junichi Tanaka and Y. Eto and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Kazuhiko Watabe

116 papers receiving 2.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kazuhiko Watabe Japan 30 1.0k 946 625 577 419 116 2.8k
Kazunori Sango Japan 31 968 1.0× 1.4k 1.5× 1.2k 1.9× 394 0.7× 262 0.6× 115 3.3k
Michela Deleidi Germany 24 909 0.9× 1.6k 1.7× 722 1.2× 723 1.3× 537 1.3× 40 3.1k
Alexandre Leite Rodrigues de Oliveira Brazil 33 1.4k 1.4× 725 0.8× 531 0.8× 360 0.6× 535 1.3× 150 3.7k
Andrew P. Mizisin United States 38 1.2k 1.2× 1.0k 1.1× 1.5k 2.4× 769 1.3× 162 0.4× 97 3.6k
P.C. Barber United Kingdom 27 889 0.9× 714 0.8× 305 0.5× 282 0.5× 348 0.8× 50 2.4k
Ana Maria Blanco Martinez Brazil 31 1.3k 1.3× 754 0.8× 522 0.8× 226 0.4× 393 0.9× 94 2.7k
Pedro Cuevas Spain 32 656 0.6× 1.7k 1.8× 414 0.7× 166 0.3× 252 0.6× 167 4.0k
Mala V. Rao United States 26 817 0.8× 1.0k 1.1× 434 0.7× 721 1.2× 172 0.4× 37 2.7k
W. Marie Campana United States 42 1.5k 1.5× 1.5k 1.6× 1.5k 2.5× 445 0.8× 285 0.7× 80 4.4k

Countries citing papers authored by Kazuhiko Watabe

Since Specialization
Citations

This map shows the geographic impact of Kazuhiko Watabe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kazuhiko Watabe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kazuhiko Watabe more than expected).

Fields of papers citing papers by Kazuhiko Watabe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kazuhiko Watabe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kazuhiko Watabe. The network helps show where Kazuhiko Watabe may publish in the future.

Co-authorship network of co-authors of Kazuhiko Watabe

This figure shows the co-authorship network connecting the top 25 collaborators of Kazuhiko Watabe. A scholar is included among the top collaborators of Kazuhiko Watabe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kazuhiko Watabe. Kazuhiko Watabe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Murakami, T., et al.. (2023). Human transthyretin gene expression is markedly increased in repair Schwann cells in an in vitro model of hereditary transthyretin amyloidosis. Neurochemistry International. 164. 105507–105507. 1 indexed citations
2.
Watabe, Kazuhiko. (2020). Praja 1 RING-finger E3 ubiquitin ligase suppresses neuronal cytoplasmic TDP-43 aggregate formation. 1 indexed citations
3.
Niimi, Naoko, Hideji Yako, Shizuka Takaku, et al.. (2017). A spontaneously immortalized Schwann cell line from aldose reductase‐deficient mice as a useful tool for studying polyol pathway and aldehyde metabolism. Journal of Neurochemistry. 144(6). 710–722. 24 indexed citations
4.
Kawakami, Emiko, et al.. (2017). Formation and spreading of TDP-43 aggregates in cultured neuronal and glial cells demonstrated by time-lapse imaging. PLoS ONE. 12(6). e0179375–e0179375. 39 indexed citations
5.
Murakami, T., Kazunori Sango, Kazuhiko Watabe, et al.. (2015). Schwann cells contribute to neurodegeneration in transthyretin amyloidosis. Journal of Neurochemistry. 134(1). 66–74. 19 indexed citations
6.
Fujimaki, Yumi, Setsu Nakatani‐Enomoto, Shiro Matsubara, et al.. (2013). Complex fasciculation potentials and survival in amyotrophic lateral sclerosis. Clinical Neurophysiology. 125(5). 1059–1064. 22 indexed citations
7.
Jin, Haifeng, Nobuo Sanjo, Toshiki Uchihara, et al.. (2010). Presenilin-1 Holoprotein is an Interacting Partner of Sarco Endoplasmic Reticulum Calcium-ATPase and Confers Resistance to Endoplasmic Reticulum Stress. Journal of Alzheimer s Disease. 20(1). 261–273. 27 indexed citations
8.
Wada, Haruka, Takuwa Yasuda, Ikuo Miura, et al.. (2009). Establishment of an Improved Mouse Model for Infantile Neuroaxonal Dystrophy That Shows Early Disease Onset and Bears a Point Mutation in Pla2g6. American Journal Of Pathology. 175(6). 2257–2263. 48 indexed citations
9.
Mori, Yuko, Akihiro Shiotani, Koichiro Saito, et al.. (2007). A Novel Drug Therapy for Recurrent Laryngeal Nerve Injury Using T‐588. The Laryngoscope. 117(7). 1313–1318. 14 indexed citations
10.
Hayashi, Yuichi, Masahiko Satoh, Yoko Uchida, et al.. (2006). Association between metallothionein genes polymorphisms and sporadic amyotrophic lateral sclerosis in a Japanese population. Amyotrophic Lateral Sclerosis. 7(1). 22–26. 19 indexed citations
11.
Ikeda, Ken, Masashi Aoki, Yoko Kawazoe, et al.. (2005). Motoneuron degeneration after facial nerve avulsion is exacerbated in presymptomatic transgenic rats expressing human mutant Cu/Zn superoxide dismutase. Journal of Neuroscience Research. 82(1). 63–70. 10 indexed citations
12.
Shirakura, Masayuki, Makoto Inoue, Satoshi Fujikawa, et al.. (2004). Postischemic administration of Sendai virus vector carrying neurotrophic factor genes prevents delayed neuronal death in gerbils. Gene Therapy. 11(9). 784–790. 47 indexed citations
13.
Sango, Kazunori, Kyoko Ajiki, Masao Horie, et al.. (2003). Synthesis, localization and externalization of galectin‐1 in mature dorsal root ganglion neurons and Schwann cells. European Journal of Neuroscience. 19(1). 55–64. 29 indexed citations
15.
Abiru, Yasuhiro, Kenichi Sakajiri, Kazuhiko Watabe, et al.. (1997). Adenovirus-Mediated Transduction with Human Glial Cell Line-Derived Neurotrophic Factor Gene Prevents 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine-Induced Dopamine Depletion in Striatum of Mouse Brain. Biochemical and Biophysical Research Communications. 238(2). 569–573. 53 indexed citations
16.
Watabe, Kazuhiko, et al.. (1995). Spontaneously immortalized adult mouse Schwann cells secrete autocrine and paracrine growth‐promoting activities. Journal of Neuroscience Research. 41(2). 279–290. 106 indexed citations
17.
Kim, S. U., et al.. (1989). Human fetal schwann cells in culture: Phenotypic expressions and proliferative capabibility. Journal of Neuroscience Research. 22(1). 50–59. 19 indexed citations
18.
Yoshida, Yasuji, Fusahiro Ikuta, Kazuhiko Watabe, & Tomoko Nagata. (1985). Developmental microvascular architecture of the rat cerebellar cortex. Anatomy and Embryology. 171(2). 129–138. 18 indexed citations
19.
Watabe, Kazuhiko, et al.. (1983). Tactile-like corpuscles in neurofibromas: Immunohistochemical demonstration of S-100 protein. Acta Neuropathologica. 61(3-4). 173–177. 26 indexed citations
20.
Ikuta, Fusahiro, Y Yoshida, Eisaku Ohama, et al.. (1983). Revised Pathophysiology on BBB Damage: The Edema as an Ingeniously Provided Condition for Cell Motility and Lesion Repair. PubMed. 8. 103–110. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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