Kayoko Kinbara

1.1k total citations
10 papers, 947 citations indexed

About

Kayoko Kinbara is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Kayoko Kinbara has authored 10 papers receiving a total of 947 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 8 papers in Cell Biology and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Kayoko Kinbara's work include Calpain Protease Function and Regulation (7 papers), Muscle Physiology and Disorders (3 papers) and Cell death mechanisms and regulation (2 papers). Kayoko Kinbara is often cited by papers focused on Calpain Protease Function and Regulation (7 papers), Muscle Physiology and Disorders (3 papers) and Cell death mechanisms and regulation (2 papers). Kayoko Kinbara collaborates with scholars based in Japan, United States and Germany. Kayoko Kinbara's co-authors include Shoichi Ishiura, Hiroyuki Sorimachi, Koichi Suzuki, Mark H. Ginsberg, Lawrence E. Goldfinger, Fan-Li Chou, Malene Bredahl Hansen, Toshio Yoshizawa, Sumiko Kimura and Kazuhiko Tagawa and has published in prestigious journals such as Journal of Biological Chemistry, Nature Reviews Molecular Cell Biology and Circulation Research.

In The Last Decade

Kayoko Kinbara

10 papers receiving 930 citations

Peers

Kayoko Kinbara
Sheela G. Bhartur United States
Kang Rao United States
Yanfeng Kong United States
Sheree Lynn Rybak United States
Diego Gravotta United States
Karin Paiha Austria
Kayoko Kinbara
Citations per year, relative to Kayoko Kinbara Kayoko Kinbara (= 1×) peers L. Brenguier

Countries citing papers authored by Kayoko Kinbara

Since Specialization
Citations

This map shows the geographic impact of Kayoko Kinbara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kayoko Kinbara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kayoko Kinbara more than expected).

Fields of papers citing papers by Kayoko Kinbara

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kayoko Kinbara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kayoko Kinbara. The network helps show where Kayoko Kinbara may publish in the future.

Co-authorship network of co-authors of Kayoko Kinbara

This figure shows the co-authorship network connecting the top 25 collaborators of Kayoko Kinbara. A scholar is included among the top collaborators of Kayoko Kinbara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kayoko Kinbara. Kayoko Kinbara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Goldfinger, Lawrence E., Ellie Tzima, Rebecca Stockton, et al.. (2008). Localized α4 Integrin Phosphorylation Directs Shear Stress–Induced Endothelial Cell Alignment. Circulation Research. 103(2). 177–185. 40 indexed citations
2.
Kinbara, Kayoko, Lawrence E. Goldfinger, Malene Bredahl Hansen, Fan-Li Chou, & Mark H. Ginsberg. (2003). Ras GTPases: integrins' friends or foes?. Nature Reviews Molecular Cell Biology. 4(10). 767–777. 180 indexed citations
3.
Lee, Hahn-Jun, Shigeo Tomioka, Kayoko Kinbara, et al.. (1999). Characterization of a Human Digestive Tract-Specific Calpain, nCL-4, Expressed in the Baculovirus System. Archives of Biochemistry and Biophysics. 362(1). 22–31. 35 indexed citations
4.
Kinbara, Kayoko, Shoichi Ishiura, Shigeo Tomioka, et al.. (1998). Purification of native p94, a muscle-specific calpain, and characterization of its autolysis. Biochemical Journal. 335(3). 589–596. 99 indexed citations
5.
Kinbara, Kayoko, et al.. (1998). Discussion. Biochemical Pharmacology. 56(4). 415–420. 38 indexed citations
6.
Kinbara, Kayoko, Hiroyuki Sorimachi, Shoichi Ishiura, & Koichi Suzuki. (1997). Muscle-Specific Calpain, p94, Interacts with the Extreme C-Terminal Region of Connectin, a Unique Region Flanked by Two Immunoglobulin C2 Motifs. Archives of Biochemistry and Biophysics. 342(1). 99–107. 101 indexed citations
7.
Sorimachi, Hiroyuki, Kayoko Kinbara, Sumiko Kimura, et al.. (1995). Muscle-specific Calpain, p94, Responsible for Limb Girdle Muscular Dystrophy Type 2A, Associates with Connectin through IS2, a p94-specific Sequence. Journal of Biological Chemistry. 270(52). 31158–31162. 277 indexed citations
8.
Suzuki, Koichi, Hiroyuki Sorimachi, Toshio Yoshizawa, Kayoko Kinbara, & Shoichi Ishiura. (1995). Review. Biological Chemistry Hoppe-Seyler. 376(9). 523–530. 167 indexed citations
9.
Kinbara, Kayoko, Hiroshi Kitagaki, Tadatoshi Kinouchi, et al.. (1994). Processing and Secretion of Alzheimer's Disease Amyloid Precursor Protein.. The Tohoku Journal of Experimental Medicine. 174(3). 209–216. 3 indexed citations
10.
Sakamoto, Kanta, Hiroyuki Sorimachi, Kayoko Kinbara, et al.. (1994). <b>QUANTIFICATION OF CALPAIN-RELATED MOLECULES BY SPECIFIC PCR AMPLIFICATION AND ITS APPLICATION TO HUMAN MUSCULAR </b><b>DYSTROPHY </b>. Biomedical Research. 15(5). 337–346. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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