Hiroyuki Sorimachi

13.6k total citations · 2 hit papers
162 papers, 11.1k citations indexed

About

Hiroyuki Sorimachi is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Hiroyuki Sorimachi has authored 162 papers receiving a total of 11.1k indexed citations (citations by other indexed papers that have themselves been cited), including 121 papers in Molecular Biology, 109 papers in Cell Biology and 38 papers in Cellular and Molecular Neuroscience. Recurrent topics in Hiroyuki Sorimachi's work include Calpain Protease Function and Regulation (101 papers), Muscle Physiology and Disorders (32 papers) and RNA regulation and disease (29 papers). Hiroyuki Sorimachi is often cited by papers focused on Calpain Protease Function and Regulation (101 papers), Muscle Physiology and Disorders (32 papers) and RNA regulation and disease (29 papers). Hiroyuki Sorimachi collaborates with scholars based in Japan, Germany and United States. Hiroyuki Sorimachi's co-authors include Koichi Suzuki, Yasuko Ono, Shoichi Ishiura, Takaomi C. Saido, Shoji Hata, Siegfried Labeit, Carol C. Gregorio, Yasufumi Emori, Henk Granzier and Hiroshi Kawasaki and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Physical Review Letters and Journal of Biological Chemistry.

In The Last Decade

Hiroyuki Sorimachi

160 papers receiving 10.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Structure and physiological function of calpains

1997 593 citations Hiroyuki Sorimachi, Shoichi Ishiura et al. profile →
Calpain: new perspectives in molecular diversity and physiological‐pathological involvement

1994 579 citations Takaomi C. Saido, Hiroyuki Sorimachi et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Hiroyuki Sorimachi Japan	57	7.8k	6.6k	2.4k	1.6k	1.1k	162	11.1k
Jocelyn Laporte France	56	7.0k 0.9×	4.5k 0.7×	1.8k 0.7×	2.0k 1.2×	874 0.8×	260	9.6k
Athar H. Chishti United States	46	4.1k 0.5×	2.9k 0.4×	1.3k 0.5×	303 0.2×	1.1k 1.0×	136	7.4k
Francesco Zorzato Italy	39	5.4k 0.7×	854 0.1×	1.5k 0.6×	2.2k 1.4×	917 0.8×	127	7.2k
Masaki Inagaki Japan	73	13.1k 1.7×	7.7k 1.2×	2.3k 0.9×	750 0.5×	1.1k 1.0×	223	18.7k
Masatoshi Maki Japan	44	3.9k 0.5×	2.8k 0.4×	609 0.3×	371 0.2×	409 0.4×	147	6.3k
Peter Gierschik Germany	57	6.9k 0.9×	1.6k 0.2×	1.6k 0.7×	881 0.6×	923 0.8×	173	9.7k
John P. Merlie United States	66	9.2k 1.2×	2.5k 0.4×	3.5k 1.5×	795 0.5×	696 0.6×	132	11.7k
Robert Day Canada	52	4.2k 0.5×	1.8k 0.3×	1.3k 0.5×	489 0.3×	673 0.6×	166	8.5k
Enrique Rodríguez-Boulan United States	70	10.1k 1.3×	6.8k 1.0×	1.3k 0.5×	303 0.2×	2.0k 1.9×	161	14.9k
Peter S. McPherson Canada	66	8.5k 1.1×	6.6k 1.0×	2.5k 1.0×	363 0.2×	1.5k 1.3×	159	12.4k

Countries citing papers authored by Hiroyuki Sorimachi

Since Specialization

Citations

This map shows the geographic impact of Hiroyuki Sorimachi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroyuki Sorimachi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroyuki Sorimachi more than expected).

Fields of papers citing papers by Hiroyuki Sorimachi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroyuki Sorimachi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroyuki Sorimachi. The network helps show where Hiroyuki Sorimachi may publish in the future.

Co-authorship network of co-authors of Hiroyuki Sorimachi

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroyuki Sorimachi. A scholar is included among the top collaborators of Hiroyuki Sorimachi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroyuki Sorimachi. Hiroyuki Sorimachi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ishimura, Ryosuke, Daisuke Noshiro, Yasuko Ono, et al.. (2022). The UFM1 system regulates ER-phagy through the ufmylation of CYB5R3. Nature Communications. 13(1). 7857–7857. 50 indexed citations

Sato, Seidai, Akiko Takahashi, Yoshinori Imai, et al.. (2015). Ablation of the p16INK4a tumour suppressor reverses ageing phenotypes of klotho mice. Nature Communications. 6(1). 7035–7035. 60 indexed citations

Ojima, Koichi, Yasuko Ono, Shoji Hata, et al.. (2014). Muscle‐specific calpain‐3 is phosphorylated in its unique insertion region for enrichment in a myofibril fraction. Genes to Cells. 19(11). 830–841. 8 indexed citations

Ono, Yasuko, Koichi Ojima, Naoko Doi, et al.. (2010). Skeletal Muscle-specific Calpain Is an Intracellular Na+-dependent Protease. Journal of Biological Chemistry. 285(30). 22986–22998. 47 indexed citations

Hayashi, Chikako, Yasuko Ono, Naoko Doi, et al.. (2008). Multiple Molecular Interactions Implicate the Connectin/Titin N2A Region as a Modulating Scaffold for p94/Calpain 3 Activity in Skeletal Muscle. Journal of Biological Chemistry. 283(21). 14801–14814. 91 indexed citations

Hata, Shoji, Hiroyuki Kawahara, Naoko Doi, et al.. (2006). Stomach-specific Calpain, nCL-2, Localizes in Mucus Cells and Proteolyzes the β-Subunit of Coatomer Complex, β-COP. Journal of Biological Chemistry. 281(16). 11214–11224. 24 indexed citations

Hayashi, Michio, et al.. (2005). Constitutive Activation of the pH-Responsive Rim101 Pathway in Yeast Mutants Defective in Late Steps of the MVB/ESCRT Pathway. Molecular and Cellular Biology. 25(21). 9478–9490. 84 indexed citations

Toyama‐Sorimachi, Noriko, Yoshiki Omatsu, Yusuke Tsujimura, et al.. (2005). Inhibitory NK Receptor Ly49Q Is Expressed on Subsets of Dendritic Cells in a Cellular Maturation- and Cytokine Stimulation-Dependent Manner. The Journal of Immunology. 174(8). 4621–4629. 38 indexed citations

KIMURA, Eiichi, Keiko Abe, Koichi Suzuki, & Hiroyuki Sorimachi. (2003). Heterogeneous Nuclear Ribonucleoprotein K Interacts with and Is Proteolyzed by Calpainin vivo. Bioscience Biotechnology and Biochemistry. 67(8). 1786–1796. 11 indexed citations

10.

Reverter, David, Stefan Strobl, C. Fernandez-Catalan, et al.. (2001). Structural Basis for Possible Calcium-Induced Activation Mechanisms of Calpains. Biological Chemistry. 382(5). 753–66. 36 indexed citations

11.

Hata, Shoji, et al.. (2000). A simple purification and fluorescent assay method of the poliovirus 3C protease searching for specific inhibitors. Journal of Virological Methods. 84(2). 117–126. 5 indexed citations

12.

Lee, Hahn-Jun, et al.. (1998). Molecular Cloning and Characterization of a Novel Tissue-Specific Calpain Predominantly Expressed in the Digestive Tract. Biological Chemistry. 379(2). 175–184. 51 indexed citations

13.

Masumoto, Hiroshi, Toshio Yoshizawa, Hiroyuki Sorimachi, et al.. (1998). Overexpression, Purification, and Characterization of Human m-Calpain and Its Active Site Mutant, m-C105S-Calpain, Using a Baculovirus Expression System. The Journal of Biochemistry. 124(5). 957–961. 27 indexed citations

14.

Henskens, Yvonne, Wim van’t Hof, Enno C.I. Veerman, et al.. (1996). Short Communication. Biological Chemistry Hoppe-Seyler. 377(12). 847–864. 3 indexed citations

15.

Tagawa, Kazuhiko, Kei Maruyama, Hiroyuki Sorimachi, et al.. (1996). Mutation of potential N-linked glycosylation sites in the Alzheimer's disease amyloid precursor protein (APP). Neuroscience Letters. 221(1). 57–60. 34 indexed citations

16.

Kinouchi, Tadatoshi, Hiroyuki Sorimachi, Keiko Mizuno, et al.. (1995). Conventional protein kinase C (PKC)‐α and novel PKCε, but not ‐δ, increase the secretion of an N‐terminal fragment of Alzheimer's disease amyloid precursor protein from PKC cDNA transfected 3Y1 fibroblasts. FEBS Letters. 364(2). 203–206. 62 indexed citations

17.

Kinbara, Kayoko, Hiroshi Kitagaki, Tadatoshi Kinouchi, et al.. (1994). Processing and Secretion of Alzheimer's Disease Amyloid Precursor Protein.. The Tohoku Journal of Experimental Medicine. 174(3). 209–216. 3 indexed citations

18.

Sasagawa, Noboru, Hiroyuki Sorimachi, Kei Maruyama, et al.. (1994). Expression of a novel human myotonin protein kinase (MtPK) cDNA clone which encodes a protein with a thymopoietin‐like domain in COS cells. FEBS Letters. 351(1). 22–26. 18 indexed citations

19.

Saido, Takaomi C., Shunsuke Nagao, Hiroyuki Sorimachi, et al.. (1992). Autolytic Transition of μCalpain upon Activation as Resolved by Antibodies Distinguishing between the Pre- and Post-Autolysis Forms1. The Journal of Biochemistry. 111(1). 81–86. 133 indexed citations

20.

Homma, Yoshimi, Tadaomi Takenawa, Yasufumi Emori, Hiroyuki Sorimachi, & Koichi Suzuki. (1989). Tissue- and cell type-specific expression of mRNAS for four types of inositol phospholipid-specific phospholipase C. Biochemical and Biophysical Research Communications. 164(1). 406–412. 95 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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